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one form in which body fuel is stored; stored primarily in the liver and broken down into glucose when needed by the body (同)animal_starch

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Geniposide Attenuates the Phosphorylation of Tau Protein in Cellular and Insulin-deficient APP/PS1 Transgenic Mouse Model of Alzheimer's Disease.

Zhang Y1, Yin F1,2, Liu J1,2, Liu Z2.
Chemical biology & drug design.Chem Biol Drug Des.2016 Mar;87(3):409-18. doi: 10.1111/cbdd.12673. Epub 2015 Nov 12.
Our previous studies have shown that geniposide plays an essential role in glucose-stimulated insulin secretion from pancreatic β cells and also regulates the metabolism of Aβ and its deposition in neurons. In this study, we reported that insulin deficiency induced significant increase of tau phos
PMID 26475430

Liu ZC1,2, Chu J1, Lin L1,3, Song J1, Ning LN1, Luo HB1,4, Yang SS1,3, Shi Y1, Wang Q1, Qu N1, Zhang Q1, Wang JZ5, Tian Q6.
Molecular neurobiology.Mol Neurobiol.2016 Mar;53(2):983-94. doi: 10.1007/s12035-014-9039-4. Epub 2015 Jan 10.
Endoplasmic reticulum (ER) stress has been indicated in the early stage of Alzheimer's disease (AD), in which tau hyperphosphorylation is one major pathological alteration. The elevation of binding immunoglobulin protein (Bip), an important ER chaperon, was reported in AD brain. It is important to s
PMID 25575678

Protective effects of butyrate-based compounds on a mouse model for spinal muscular atrophy.

Butchbach ME1, Lumpkin CJ2, Harris AW3, Saieva L4, Edwards JD5, Workman E5, Simard LR6, Pellizzoni L4, Burghes AH7.
Experimental neurology.Exp Neurol.2016 Feb 15;279:13-26. doi: 10.1016/j.expneurol.2016.02.009. [Epub ahead of print]
Proximal spinal muscular atrophy (SMA) is a childhood-onset degenerative disease resulting from the selective loss of motor neurons in the spinal cord. SMA is caused by the loss of SMN1 (survival motor neuron 1) but retention of SMN2. The number of copies of SMN2 modifies disease severity in SMA pat
PMID 26892876

仲佐輝子,沖中靖,松本博
ビタミン 53(11), 447-454, 1979-11-25
… 生後3週齢のWistar系雄ラットを用い36日間飼育して, 肝臓中の解糖系, 糖新生系及び脂肪酸合成系の酵素に対する食餌性B_6欠乏の影響について検討した.解糖系酵素ではB_6欠乏群において対照群に比べglycogen phosphorylase, pyruvate kinase及びpyruvate dehydrogenation complex活性は低値を示したが, hexokinase及びfructosediphosphate aldolase活性は高値を示した.Glyceraldehyde-3-phosphate dehydrogenase活性は両群の間に有意差がなかった.血糖値は差がなかった …
NAID 110002845190