- 同
- glucuronosyl transferase
WordNet
- the 21st letter of the Roman alphabet (同)u
PrepTutorEJDIC
- 〈U〉〈C〉(…の)(量・額などの)不足,欠乏《+『of』(『in』)+『名』》 / 〈C〉不足分,不足量,不足額 / 〈C〉(精神・肉体などの)欠陥
- uraniumの化学記号
- 上流の,上流階級にふさわしい / (映画が)児童観覧用の
UpToDate Contents
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English Journal
- A new case of UDP-galactose transporter deficiency (SLC35A2-CDG): molecular basis, clinical phenotype, and therapeutic approach.
- Dörre K1, Olczak M, Wada Y, Sosicka P, Grüneberg M, Reunert J, Kurlemann G, Fiedler B, Biskup S, Hörtnagel K, Rust S, Marquardt T.
- Journal of inherited metabolic disease.J Inherit Metab Dis.2015 Mar 17. [Epub ahead of print]
- Congenital disorders of glycosylation (CDG) are a group of hereditary metabolic diseases characterized by abnormal glycosylation of proteins and lipids. Often, multisystem disorders with central nervous system involvement and a large variety of clinical symptoms occur. The main characteristics are d
- PMID 25778940
- Spectrum of UGT1A1 Variations in Chinese Patients with Crigler-Najjar Syndrome Type II.
- Li L1, Deng G2, Tang Y1, Mao Q2.
- PloS one.PLoS One.2015 May 20;10(5):e0126263. doi: 10.1371/journal.pone.0126263. eCollection 2015.
- Crigler-Najjar Syndrome type II (CNS-II) is an autosomal recessive hereditary condition of unconjugated hyperbilirubinemia without hemolysis, with bilirubin levels ranging from 102.6 μmol/L to 342 μmol/L. CNS-II is caused by a deficiency of UDP-glucuronyl transferase (UGT), which is encoded by the
- PMID 25993113
- [Effect of Corynebacterium non diphtheriae on functional activity and apoptosis of macrophages].
- Kharseeva GG, Voronina NA, Tiukavkina SIu.
- Zhurnal mikrobiologii, epidemiologii, i immunobiologii.Zh Mikrobiol Epidemiol Immunobiol.2014 Nov-Dec;(6):96-100.
- AIM: Determine the ability of Corynebacterium non diphtheriae to induce phagocytosis and apoptosis of macrophages and evaluate regulatory effect of nuetrophilokines (NPK) induced by Corynebacterium non diphtheriae on these processes.MATERIALS AND METHODS: The ability of Corynebacterium non diphtheri
- PMID 25816522
Japanese Journal
- 千葉 寛,森本 かおり
- YAKUGAKU ZASSHI 131(2), 247-253, 2011
- … In addition to SLCO1B1, recent studies suggested that variants of genes encoding transporters (ABCG2 and ABCB1) and metabolic enzymes (CYP2C8 and UGT1A3) involved in the disposition of statins, and those involved in the metabolic muscle disease (glycogen storage disorders, carnitine palmitoyl-2 deficiency and myoadenylate deaminase deficiency) are also risk factors of statin-induced myopathy. …
- NAID 130000451459
- Effects of Selenium Deficiency on Aldehyde Oxidase 1 in Rats(Molecular and Cell Biology)
- Itoh Kunio,Adachi Mayuko,Sato Jun [他],SHOUJI Kanako,FUKIYA Kensuke,FUJII Keiko,TANAKA Yorihisa
- Biological & pharmaceutical bulletin 32(2), 190-194, 2009-02-01
- … Selenium deficiency has been reported to result in an extraordinary decrease of glutathione peroxidase (GSH-Px) and, reversely, an increase of detoxifying enzymes such as glutathione-S-transferase (GST), uridine-5'-diphosphate glucuronosyltransferase (UGT), nicotinamide-dependent quinine oxidoreductase (NQO1; … The aim of this study is to clarify the effects of selenium deficiency on AOX1 in rats. …
- NAID 110007042164
- EFFECT OF TROGLITAZONE ON THE LIVER OF A GUNN RAT MODEL OF GENETIC ENZYME POLYMORPHISM
- Watanabe Toshiyuki,Furukawa Tadashi,Sharyo Satoru [他],OHASHI Yoshihiko,YASUDA Mitsuya,TAKAOKA Masaya,MANABE Sunao
- Journal of toxicological sciences 25(5), 423-431, 2000-12-25
- … this study, Gunn rats, which are hereditarily deficient in the UGT1 family of UGT isozymes, and Wistar rats, the parent strain of Gunn rats, were treated with troglitazone for 3 months at dose levels of 0, 100 or 400 mg/kg to investigate two possibilities:first, whether the genetic deficiency in UGT1s induces an alteration of the metabolic profile of troglitazone followed by liver dysfunction, and second, wheter the dosing of troglitazone to Gunn rats which sho …
- NAID 110001806880
Related Links
- Crigler-Najjar syndrome ( Bilirubin uridinediphosphate glucuronosyltransferase deficiency Bilirubin-UGT deficiency Hereditary unconjugated hyperbilirubinemia UGT deficiency ) Orphanumber: 205 It's a disease Autosomal recessive ...
- Flavonoids have poor bioavailabilities largely because of metabolism via UDP-glucuronosyltransferases (UGTs). This study aims to further understand the functions of UGT in metabolizing genistein and apigenin, two compounds ...
Related Pictures
★リンクテーブル★
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- 不足、欠乏、欠失、欠如、欠損、不十分。栄養不足、栄養素欠乏、欠乏症。(遺伝子)(染色体内の)遺伝子欠失
- 欠けているもの、不足している物。不足分。不完全なもの、欠点のあるもの
- 関
- absence, agenesis, dearth, defect, defective, deficient, deficit, delete, deletion, deletional, depletion, deprivation, deprive, lack, miss, missing, morphological defect, paucity, scarce, scarcity, starve
[★]
UDP-グルクロノシルトランスフェラーゼ
- 関
- Uridine diphosphate-glucurono- syltransferases
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