WordNet

be a contributing factor; "make things factor into a companys profitability"
any of the numbers (or symbols) that form a product when multiplied together
an independent variable in statistics
anything that contributes causally to a result; "a number of factors determined the outcome"
consider as relevant when making a decision; "You must factor in the recent developments" (同)factor in, factor out
resolve into factors; "a quantum computer can factor the number 15" (同)factor in, factor out
an event known to have happened or something known to have existed; "your fears have no basis in fact"; "how much of the story is fact and how much fiction is hard to tell"
a concept whose truth can be proved; "scientific hypotheses are not facts"
a piece of information about circumstances that exist or events that have occurred; "first you must collect all the facts of the case"
a statement or assertion of verified information about something that is the case or has happened; "he supported his argument with an impressive array of facts"
(genetics) the organic process whereby the DNA sequence in a gene is copied into mRNA; the process whereby a base sequence of messenger RNA is synthesized on a template of complementary DNA
a sound or television recording (e.g., from a broadcast to a tape recording)
something written, especially copied from one medium to another, as a typewritten version of dictation (同)written text
the 20th letter of the Roman alphabet (同)t

PrepTutorEJDIC

(…の)『要因』,(…を生み出す)要素《+『in』+『名』(do『ing』)》 / 囲数,約数 / 代理人,《おもに英》仲買人 / =factorize
〈C〉『事実』,実際にある(あった)事 / 〈U〉真相,真実(truth) / 《the~》(法律用語で)犯行
〈U〉(…を)書き写す(書き直す)こと,(…の)転写,謄写《+of+名》 / 〈C〉写し(書き直し)[たもの] / 〈U〉〈C〉編曲[されたもの] / 〈U〉〈C〉(ラジオ・テレビの)録音(録画)
tritiumの化学記号
Teachers College 教員養成大学

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1. 胃癌の病理および分子学的病因 pathology and molecular pathogenesis of gastric cancer
2. 大腸癌の分子遺伝学 molecular genetics of colorectal cancer
3. 特発性肺線維症の病因 pathogenesis of idiopathic pulmonary fibrosis
4. デスモイド腫瘍：疫学、危険因子、分子学的病因、臨床症状、診断、および局所療法 desmoid tumors epidemiology risk factors molecular pathogenesis clinical presentation diagnosis and local therapy
5. 骨格の正常な発達および骨形成ならびに骨吸収の調節 normal skeletal development and regulation of bone formation and resorption

GPR48, a poor prognostic factor, promotes tumor metastasis and activates β-catenin/TCF signaling in colorectal cancer.

Wu J, Xie N, Xie K, Zeng J, Cheng L, Lei Y, Liu Y, Song L, Dong D, Chen Y, Zeng R, Nice EC, Huang C, Wei Y.SourceThe State Key Laboratory of Biotherapy, West China Hospital, Sichuan University, 610041, P. R. China.
Carcinogenesis.Carcinogenesis.2013 Jun 26. [Epub ahead of print]
G protein-coupled receptor 48 (GPR48) is an orphan receptor belonging to the G protein-coupled receptors (GPCRs) family, which plays an important role in the development of various organs and cancer development and progression such as gastric cancer and colorectal cancer. However, the prognostic val
PMID 23803691

Androgens Up-regulate Transcription of the Notch Inhibitor Numb in C2C12 Myoblasts via Wnt/β-Catenin Signaling to T Cell Factor Elements in the Numb Promoter.

Liu XH, Wu Y, Yao S, Levine AC, Kirschenbaum A, Collier L, Bauman WA, Cardozo CP.SourceFrom the National Center of Excellence for the Medical Consequences of Spinal Cord Injury, James J. Peter Veterans Affairs Medical Center, Bronx, New York 10468 and.
The Journal of biological chemistry.J Biol Chem.2013 Jun 21;288(25):17990-8. doi: 10.1074/jbc.M113.478487. Epub 2013 May 6.
Androgen signaling via the androgen receptor is a key pathway that contributes to development, cell fate decisions, and differentiation, including that of myogenic progenitors. Androgens and synthetic steroids have well established anabolic actions on skeletal muscle. Wnt and Notch signaling pathway
PMID 23649620

SNAI1-Mediated Epithelial-Mesenchymal Transition Confers Chemoresistance and Cellular Plasticity by Regulating Genes Involved in Cell Death and Stem Cell Maintenance.

Lim S, Becker A, Zimmer A, Lu J, Buettner R, Kirfel J.SourceInstitute of Pathology, University of Cologne, Cologne, Germany.
PloS one.PLoS One.2013 Jun 17;8(6):e66558. Print 2013.
Tumor cells at the tumor margin lose epithelial properties and acquire features of mesenchymal cells, a process called epithelial-to-mesenchymal transition (EMT). Recently, features of EMT were shown to be linked to cells with tumor-founding capability, so-called cancer stem cells (CSCs). Inducers o
PMID 23799116

High Expression of Pitx-2 in the ICAT-deficient Metanephros Leads to Developmental Arrest

Hasegawa Yoshimi,Iizuka-Kogo Akiko,Akiyama Tetsu,Senda Takao
ACTA HISTOCHEMICA ET CYTOCHEMICA advpub(0), 1004020079, 2010
… ICAT (Inhibitor of β-catenin and T cell factor) inhibits the interaction between β-catenin and TCF/LEF transcription factor and serves as a negative regulator of Wnt signaling. …
NAID 130000248642