同: IIRF

WordNet

be a contributing factor; "make things factor into a companys profitability"
any of the numbers (or symbols) that form a product when multiplied together
an independent variable in statistics
anything that contributes causally to a result; "a number of factors determined the outcome"
consider as relevant when making a decision; "You must factor in the recent developments" (同)factor in, factor out
resolve into factors; "a quantum computer can factor the number 15" (同)factor in, factor out
an event known to have happened or something known to have existed; "your fears have no basis in fact"; "how much of the story is fact and how much fiction is hard to tell"
a concept whose truth can be proved; "scientific hypotheses are not facts"
a piece of information about circumstances that exist or events that have occurred; "first you must collect all the facts of the case"
a statement or assertion of verified information about something that is the case or has happened; "he supported his argument with an impressive array of facts"
the act of keeping something within specified bounds (by force if necessary); "the restriction of the infection to a focal area" (同)confinement
a principle that limits the extent of something; "I am willing to accept certain restrictions on my movements" (同)limitation
having the same evolutionary origin but not necessarily the same function; "the wing of a bat and the arm of a man are homologous"
corresponding or similar in position or structure or function or characteristics; especially derived from an organism of the same species; "a homologous tissue graft"

PrepTutorEJDIC

(…の)『要因』,(…を生み出す)要素《+『in』+『名』(do『ing』)》 / 囲数,約数 / 代理人,《おもに英》仲買人 / =factorize
〈C〉『事実』,実際にある(あった)事 / 〈U〉真相,真実(truth) / 《the~》(法律用語で)犯行
(…を)『制限』(『限定』)『すること』《+『of』+『名』》,制限(限定)されていること / 〈C〉(…に対する)制限事項,制約《+『on』(『against』)+『名』》
(位置・性質・構造などが)一致する,相応する;(生物の器官が)相同の

UpToDate Contents

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1. 活性化プロテインＣ抵抗性および第V因子ライデン activated protein c resistance and factor v leiden
2. 発作性夜間血色素尿症の病因：細胞蛋白の欠損 pathogenesis of paroxysmal nocturnal hemoglobinuria missing cell proteins
3. インスリン様成長因子 Iの生理学 physiology of insulin like growth factor i
4. 遺伝子組換え第VIIa凝固因子の治療使用 therapeutic uses of recombinant coagulation factor viia
5. 蛋白質摂取制限と慢性腎臓病の進行 protein restriction and progression of chronic kidney disease

English Journal

Epigenetic Inactivation of BRCA1 Through Promoter Hypermethylation and Its Clinical Importance in Triple-Negative Breast Cancer.

Yamashita N1, Tokunaga E2, Kitao H3, Hitchins M4, Inoue Y1, Tanaka K1, Hisamatsu Y1, Taketani K1, Akiyoshi S1, Okada S1, Oda Y5, Saeki H1, Oki E1, Maehara Y1.
Clinical breast cancer.Clin Breast Cancer.2015 Jun 18. pii: S1526-8209(15)00125-1. doi: 10.1016/j.clbc.2015.06.009. [Epub ahead of print]
BACKGROUND: Triple-negative breast cancer (TNBC) has many similarities with basal-like breast cancer. Additionally, TNBCs are associated with Breast cancer susceptibility gene I (BRCA1) functional loss, which leads to impaired homologous recombination-mediated DNA repair. Although somatic mutations
PMID 26195437

Congenital CD59 Deficiency.

Höchsmann B1, Schrezenmeier H2.
Hematology/oncology clinics of North America.Hematol Oncol Clin North Am.2015 Jun;29(3):495-507. doi: 10.1016/j.hoc.2015.01.006. Epub 2015 Apr 4.
The severe clinical symptoms of inherited CD59 deficiency confirm the importance of CD59 as essential complement regulatory protein for protection of cells against complement attack, in particular protection of hematopoietic cells and human neuronal tissue. Targeted complement inhibition might becom
PMID 26043388

Polo-like kinase 3 regulates CtIP during DNA double-strand break repair in G1.

Barton O1, Naumann SC1, Diemer-Biehs R1, Künzel J1, Steinlage M1, Conrad S1, Makharashvili N2, Wang J3, Feng L3, Lopez BS4, Paull TT2, Chen J3, Jeggo PA5, Löbrich M6.
The Journal of cell biology.J Cell Biol.2014 Sep 29;206(7):877-94. doi: 10.1083/jcb.201401146.
DNA double-strand breaks (DSBs) are repaired by nonhomologous end joining (NHEJ) or homologous recombination (HR). The C terminal binding protein-interacting protein (CtIP) is phosphorylated in G2 by cyclin-dependent kinases to initiate resection and promote HR. CtIP also exerts functions during NHE
PMID 25267294

Japanese Journal

エイズウイルスタンパク質の分子機能に関する研究

藤田美歌子,フジタミカコ,Fujita Mikako
藥學雑誌 133(10), 1103-1111, 2013-10
… Described herein are viral protein X (Vpx), uniquely found in HIV-2,and its homologous protein Vpr found both in HIV-1 and -2. … In 2011, the protein SAMHD1 was identified as the host restriction factor counteracted by Vpx, by foreign researchers. …
NAID 120005531235

エイズウイルスタンパク質の分子機能に関する研究

藤田美歌子
薬学雑誌. 乙号 133(10), 1103-1111, 2013
… Described herein are viral protein X (Vpx), uniquely found in HIV-2, and its homologous protein Vpr found both in HIV-1 and -2. … In 2011, the protein SAMHD1 was identified as the host restriction factor counteracted by Vpx, by foreign reserchers. …
NAID 130004427988

脊髄損傷の神経修復

川口三郎
Otology Japan 8(3), 135-145, 1998
… Current endeavors to repair spinal cord injury can be classified into four categories: 1) employment of a peripheral nerve segment or Schwann cells as conduit of CNS axons, 2) blockade of neurite growth inhibitory factor by antibody (IN-1) and enhancement of axonal outgrowth by neurotrophin-3, 3) transplantation of embryonic CNS structures to supplement neurons and also to provide positional and guiding cues for axons that are contained in glia and extracellular matrix, and 4) transplantation of stem cells derived from the adult brain. …
NAID 130001788082