PrepTutorEJDIC
- general practitioner一般開業医
English Journal
- [Correlation of Plasma Co-stimulatory Molecules B7-H2 and B7-H3 with Platelet Auto-antibodies in Patients with Immune Thrombocytopenic Purpura].
- Zuo B1, Zhao YX2, Yang JF1, He Y1,3.
- Zhongguo shi yan xue ye xue za zhi / Zhongguo bing li sheng li xue hui = Journal of experimental hematology / Chinese Association of Pathophysiology.Zhongguo Shi Yan Xue Ye Xue Za Zhi.2015 Aug;23(4):1075-80. doi: 10.7534/j.issn.1009-2137.2015.04.033.
- OBJECTIVE: To investigate whether the plasma level of platelet auto- antibodies in ITP patients is related to that of co-stimulatory molecules sB7-H2 and sB7-H3.METHODS: A total of 61 ITP patients and 25 healthy controls from the First Affiliated Hospital of Soochow University from June 2012 to Augu
- PMID 26314449
- An improved flow cytometric immunobead array to detect autoantibodies in plasma from patients with immune thrombocytopenic purpura.
- Zhao Y1, Zhu M1, Jiang M1, Zuo B1, Wu Q1, Ruan C1, He Y2.
- Clinica chimica acta; international journal of clinical chemistry.Clin Chim Acta.2015 Jan 1;438:396-400. doi: 10.1016/j.cca.2014.09.018. Epub 2014 Sep 28.
- BACKGROUND: Autoantibodies against platelet glycoproteins (GPs) play an important role in immune thrombocytopenic purpura (ITP). This study was to develop an improved flow cytometric immunobead array (FCIA) assay to detect platelet autoantibodies in ITP patient plasma.METHODS: Plasma samples were is
- PMID 25269089
- Spectrum of the mutations in Bernard-Soulier syndrome.
- Savoia A1, Kunishima S, De Rocco D, Zieger B, Rand ML, Pujol-Moix N, Caliskan U, Tokgoz H, Pecci A, Noris P, Srivastava A, Ward C, Morel-Kopp MC, Alessi MC, Bellucci S, Beurrier P, de Maistre E, Favier R, Hézard N, Hurtaud-Roux MF, Latger-Cannard V, Lavenu-Bombled C, Proulle V, Meunier S, Négrier C, Nurden A, Randrianaivo H, Fabris F, Platokouki H, Rosenberg N, HadjKacem B, Heller PG, Karimi M, Balduini CL, Pastore A, Lanza F.
- Human mutation.Hum Mutat.2014 Sep;35(9):1033-45. doi: 10.1002/humu.22607. Epub 2014 Jul 15.
- Bernard-Soulier syndrome (BSS) is a rare autosomal recessive bleeding disorder characterized by defects of the GPIb-IX-V complex, a platelet receptor for von Willebrand factor (VWF). Most of the mutations identified in the genes encoding for the GP1BA (GPIbα), GP1BB (GPIbβ), and GP9 (GPIX) subunit
- PMID 24934643
Japanese Journal
- 新生児期から持続する血小板減少症から診断されたBernard-Soulier症候群の1例
- 日本小児血液・がん学会雑誌 52(5), 426-429, 2015
- NAID 130005125284
- Platelets with a W127X mutation in GPIX express sufficient residual amounts of GPIbα to support adhesion to von Willebrand factor and collagen
- International journal of hematology 96(6), 733-742, 2012-12-01
- NAID 10031144612
- Bernard-Soulier Syndrome due to GPIX W127X Mutation in Japan Is Frequently Misdiagnosed as Idiopathic Thrombocytopenic Purpura
- International journal of hematology 83(4), 366-367, 2006-05-15
- NAID 10017475827
Related Links
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Related Pictures
★リンクテーブル★
| リンク元 | 「CD42」「GpIX」 |
| 関連記事 | 「GPI」「GP」 |
「CD42」
- 関
- CD
発現細胞
分子量
- a:23, B:135&23, c:22, d:85
機能
- binds von Willebrand factor, thrombin; essential for platelet adhesion at sites of injury
別名
ファミリー
- leucine-rich repeat
「GpIX」
臨床関連
「GPI」
「GP」