癌原遺伝子産物c-Abl
- 関
- c-Abl protein
WordNet
- the 3rd letter of the Roman alphabet (同)c
- (music) the keynote of the scale of C major
- a general-purpose programing language closely associated with the UNIX operating system
- in favor of a proposition, opinion, etc.
- an argument in favor of a proposal
- in favor of (an action or proposal etc.); "a pro vote"
- any of a large group of nitrogenous organic compounds that are essential constituents of living cells; consist of polymers of amino acids; essential in the diet of animals for growth and for repair of tissues; can be obtained from meat and eggs and milk and legumes; "a diet high in protein"
- the 1st letter of the Roman alphabet (同)a
- the blood group whose red cells carry the A antigen (同)type_A, group A
- indicating the first or earliest or original; "`proto is a combining form in a word like `protolanguage that refers to the hypothetical ancestor of another language or group of languages"
PrepTutorEJDIC
- carbonの化学記号
- =professional
- 賛成論;賛成票(者) / 賛成して
- 蛋白(たんばく)質
- answer / ampere
UpToDate Contents
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English Journal
- Functional interaction between nonreceptor tyrosine kinase c-Abl and SR-Rich protein RBM39.
- Mai S1, Qu X2, Li P1, Ma Q1, Liu X3, Cao C4.
- Biochemical and biophysical research communications.Biochem Biophys Res Commun.2016 Apr 22;473(1):355-60. doi: 10.1016/j.bbrc.2016.03.108. Epub 2016 Mar 24.
- RBM39, also known as splicing factor HCC1.4, acts as a transcriptional coactivator for the steroid nuclear receptors JUN/AP-1, ESR1/ER-α and ESR2/ER-β. RBM39 is involved in the regulation of the transcriptional responses of these steroid nuclear receptors and promotes transcriptional initiation. I
- PMID 27018250
- Structural basis of how stress-induced MDMX phosphorylation activates p53.
- Chen X1,2, Gohain N1, Zhan C1, Lu WY2, Pazgier M1, Lu W1,3.
- Oncogene.Oncogene.2016 Apr 14;35(15):1919-25. doi: 10.1038/onc.2015.255. Epub 2015 Jul 6.
- The tumor-suppressor protein p53 is tightly controlled in normal cells by its two negative regulators--the E3 ubiquitin ligase MDM2 and its homolog MDMX. Under stressed conditions such as DNA damage, p53 escapes MDM2- and MDMX-mediated functional inhibition and degradation, acting to prevent damaged
- PMID 26148237
- Transcription factors that interact with p53 and Mdm2.
- Inoue K1, Fry EA1, Frazier DP1.
- International journal of cancer.Int J Cancer.2016 Apr 1;138(7):1577-85. doi: 10.1002/ijc.29663. Epub 2015 Jul 14.
- The tumor suppressor p53 is activated upon cellular stresses such as DNA damage, oncogene activation, hypoxia, which transactivates sets of genes that induce DNA repair, cell cycle arrest, apoptosis, or autophagy, playing crucial roles in the prevention of tumor formation. The central regulator of t
- PMID 26132471
Related Links
- ... tyrosine-protein kinase 1, bcr/c-abl oncogene protein, c-abl oncogene 1, receptor tyrosine kinase, proto-oncogene c-Abl, proto-oncogene tyrosine-protein kinase ABL1, tyrosine-protein kinase ABL1, v-abl Abelson murine ...
- Proto-Oncogene C-Abl 3 4 EC 2.7.10.2 4 63 P150 3 4 JTK7 3 4 ABL 3 4 Abelson Murine Leukemia Viral Oncogene Homolog 1 ...
★リンクテーブル★
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- 英
- proto-oncogene protein c-Abl
- 関
- c-Ablタンパク質
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c-Ablタンパク質
- 関
- proto-oncogene protein c-Abl
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- 関
- oncogene product、oncoprotein
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