WordNet
- a disorder of eating seen among young women who go on eating binges and then feel guilt and depression and self-condemnation (同)binge-eating syndrome
- pathologically insatiable hunger (especially when caused by brain lesions)
- suffering from bulimia
- a person suffering from bulimia
- the quantity contained in a bin (同)binful
- store in bins
- a container; usually has a lid
- the act of consuming food (同)feeding
PrepTutorEJDIC
- (穀物・石炭などを入れる)ふたつきの大箱 / (れんがなどで仕切った)貯蔵所
- (飲み食いしての)浮かれ騒き,とんちゃん騒ぎ,(何かを)思いきりすること
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出典(authority):フリー百科事典『ウィキペディア（Wikipedia）』「2013/05/26 10:25:49」(JST)
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- 1. 成人における気晴らし食い-摂食障害：治療の概要 binge eating disorder in adults overview of treatment
- 2. 気晴らし食い症候群：認知行動療法（CBT） binge eating disorder cognitive behavioral therapy cbt
- 3. 摂食障害：疫学の概要、病因、および経過 eating disorders overview of epidemiology diagnosis and course of illness
- 4. 成人における神経性大食症症および気晴らし食い症候群：内科的合併症およびそのマネージメント bulimia nervosa and binge eating disorder in adults medical complications and their management
- 5. 摂食障害：治療の概要 eating disorders overview of treatment
English Journal
- Mu opioid receptor antagonism in the nucleus accumbens shell blocks consumption of a preferred sucrose solution in an anticipatory contrast paradigm.
- Katsuura Y1, Taha SA2.Author information 1Department of Neurobiology and Anatomy, University of Utah, Salt Lake City, UT, USA.2Department of Neurobiology and Anatomy, University of Utah, Salt Lake City, UT, USA. Electronic address: s.taha@utah.edu.AbstractBinge eating, a central feature of multiple eating disorders, is characterized by excessive consumption occurring during discrete, often brief, intervals. Highly palatable foods play an important role in these binge episodes - foods chosen during bingeing are typically higher in fat or sugar than those normally consumed. Multiple lines of evidence suggest a central role for signaling by endogenous opioids in promoting palatability-driven eating. This role extends to binge-like feeding studied in animal models, which is reduced by administration of opioid antagonists. However, the neural circuits and specific opioid receptors mediating these effects are not fully understood. In the present experiments, we tested the hypothesis that endogenous opioid signaling in the nucleus accumbens promotes consumption in a model of binge eating. We used an anticipatory contrast paradigm in which separate groups of rats were presented sequentially with 4% sucrose and then either 20% or 0% sucrose solutions. In rats presented with 4% and then 20% sucrose, daily training in this paradigm produced robust intake of 20% sucrose, preceded by learned hypophagia during access to 4% sucrose. We tested the effects of site-specific infusions of naltrexone (a nonspecific opioid receptor antagonist: 0, 1, 10, and 50μg/side in the nucleus accumbens core and shell), naltrindole (a delta opioid receptor antagonist: 0, 0.5, 5, and 10μg/side in the nucleus accumbens shell) and beta-funaltrexamine (a mu opioid receptor antagonist: 0 and 2.5μg/side in the nucleus accumbens shell) on consumption in this contrast paradigm. Our results show that signaling through the mu opioid receptor in the nucleus accumbens shell is dynamically modulated during formation of learned food preferences, and promotes binge-like consumption of palatable foods based on these learned preferences.
- Neuroscience.Neuroscience.2014 Mar 7;261:144-52. doi: 10.1016/j.neuroscience.2013.12.004. Epub 2013 Dec 14.
- Binge eating, a central feature of multiple eating disorders, is characterized by excessive consumption occurring during discrete, often brief, intervals. Highly palatable foods play an important role in these binge episodes - foods chosen during bingeing are typically higher in fat or sugar than th
- PMID 24342569
- Anticipation of a psychosocial stressor differentially influences ghrelin, cortisol and food intake among emotional and non-emotional eaters.
- Raspopow K1, Abizaid A2, Matheson K3, Anisman H4.Author information 1Department of Neuroscience, LSRB 333, Carleton University, 1125 Colonel By Drive, Ottawa, ON K1S 5B6, Canada. Electronic address: kraspopo@connect.carleton.ca.2Department of Neuroscience, LSRB 333, Carleton University, 1125 Colonel By Drive, Ottawa, ON K1S 5B6, Canada. Electronic address: alfonso.abizaid@carleton.ca.3Department of Neuroscience, LSRB 333, Carleton University, 1125 Colonel By Drive, Ottawa, ON K1S 5B6, Canada; Department of Psychology, Carleton University, 1125 Colonel By Drive, Ottawa, ON K1S 5B6, Canada. Electronic address: kim.matheson@carleton.ca.4Department of Neuroscience, LSRB 333, Carleton University, 1125 Colonel By Drive, Ottawa, ON K1S 5B6, Canada. Electronic address: hymie.anisman@carleton.ca.AbstractNegative emotions trigger eating in some individuals (emotional eaters) possibly by influencing stress hormones that contribute to eating regulation (e.g., cortisol), or eating-related peptides (e.g., ghrelin) signaling food initiation. The present study assessed whether stressor-elicited cortisol and ghrelin changes would differ between emotional and non-emotional eaters, and whether eating would influence these neuroendocrine responses. Undergraduate women (N=103) who completed measures of emotional eating, were assigned to anticipate either a stressful (public speaking) or non-stressful event. During this period, participants were or were not offered food. Blood samples were taken continuously over a 40-min period to assess changes of cortisol and ghrelin levels, and mood was assessed after the anticipation period. Baseline ghrelin levels were lower in emotional than non-emotional eaters, and this relation was mediated by percent body fat. Ghrelin levels were elevated among women anticipating a stressor, compared to those in the control condition. Additionally, the normal decline of ghrelin following food consumption was not apparent among emotional eaters. Although food intake was not tied to hormone responses, reported hunger was associated with greater food intake for women in the stressor condition. It was suggested that emotional eating coupled with subjective feelings of hunger, might contribute to eating in response to an acute stressor. Additionally, feedback mechanisms controlling the normalization of ghrelin levels might be disturbed in emotional eaters. The similarity of the ghrelin profile of emotional eaters to that of binge eaters and obese individuals, raises the possibility that disturbed ghrelin response might be a risk factor for such conditions.
- Appetite.Appetite.2014 Mar;74:35-43. doi: 10.1016/j.appet.2013.11.018. Epub 2013 Dec 1.
- Negative emotions trigger eating in some individuals (emotional eaters) possibly by influencing stress hormones that contribute to eating regulation (e.g., cortisol), or eating-related peptides (e.g., ghrelin) signaling food initiation. The present study assessed whether stressor-elicited cortisol a
- PMID 24295926
- A new biomarker of hedonic eating? A preliminary investigation of cortisol and nausea responses to acute opioid blockade.
- Daubenmier J1, Lustig RH2, Hecht FM3, Kristeller J4, Woolley J5, Adam T6, Dallman M7, Epel E5.Author information 1Osher Center for Integrative Medicine, Department of Medicine, University of California, San Francisco, United States. Electronic address: Jennifer.daubenmier@ucsf.edu.2Department of Pediatrics, University of California, San Francisco, United States.3Osher Center for Integrative Medicine, Department of Medicine, University of California, San Francisco, United States.4Department of Psychology, Indiana State University, United States.5Department of Psychiatry, University of California, San Francisco, United States.6Department of Human Biology, Maastricht University, The Netherlands.7Department of Physiology, University of California, San Francisco, United States.AbstractOverweight and obese individuals differ in their degree of hedonic eating. This may reflect adaptations in reward-related neural circuits, regulated in part by opioidergic activity. We examined an indirect, functional measure of central opioidergic activity by assessing cortisol and nausea responses to acute opioid blockade using the opioid antagonist naltrexone in overweight/obese women (mean BMI=31.1±4.8) prior to the start of a mindfulness-based intervention to reduce stress eating. In addition, we assessed indices of hedonic-related eating, including eating behaviors (binge eating, emotional eating, external eating, restraint) and intake of sweets/desserts and carbohydrates (Block Food Frequency); interoceptive awareness (which is associated with dysregulated eating behavior); and level of adiposity at baseline. Naltrexone-induced increases in cortisol were associated with greater emotional and restrained eating and lower interoceptive awareness. Naltrexone-induced nausea was associated with binge eating and higher adiposity. Furthermore, in a small exploratory analysis, naltrexone-induced nausea predicted treatment response to the mindfulness intervention, as participants with more severe nausea at baseline maintained weight whereas those with little or no nausea responses tended to gain weight. These preliminary data suggest that naltrexone-induced cortisol release and nausea may help identify individuals who have greater underlying food reward dependence, which leads to an excessive drive to eat. Future research is needed to confirm this finding and to test if these markers of opioidergic tone might help predict success in certain types of weight management programs.
- Appetite.Appetite.2014 Mar;74:92-100. doi: 10.1016/j.appet.2013.11.014. Epub 2013 Nov 27.
- Overweight and obese individuals differ in their degree of hedonic eating. This may reflect adaptations in reward-related neural circuits, regulated in part by opioidergic activity. We examined an indirect, functional measure of central opioidergic activity by assessing cortisol and nausea responses
- PMID 24291355
Japanese Journal
- 大学生女子のむちゃ食い行動中の気分の認知の変化
- 島谷 まき子/椿 徳子
- 昭和女子大学生活心理研究所紀要 15, 21-31, 2013-03-31
- … before,during,and after binge eating were investigated. … First, a pilot study was conducted with female university students (N=153) and a questionnaire was developed that consisted of items measuring binge eating disorder (BED) tendencies, eating behavior addiction, and changes in cognition about the mood; …
- NAID 110009560367
- 2型糖尿病の心身医療 (特集 内分泌・代謝疾患の心身医療)
- 野崎 剛弘,須藤 信行
- 心身医学 53(1), 20-28, 2013-00-00
- NAID 40019528774
- ロールシャッハ・テスト包括システムによる摂食障害の心理的特徴の検討:第2報 : 神経性食欲不振症・「制限型群」と比較した「むちゃ食い/排出型群」の心理的特徴
- 塚野 佳世子,秋庭 篤代,津久井 要 [他],伊藤 牧子,江花 昭一,山本 晴義
- 心身医学 52(7), 646-653, 2012-07-01
- 摂食障害の病態は多様であり,病態によってその心理的特徴も異なることが指摘されている.本研究では,入院中の摂食障害患者にロールシャッハ・テストを施行し,DSM-IVにおける神経性無食欲症制限型(ANR:23名)とむちゃ食い/排出型(ANBP:10名)に分けて比較し,それぞれの心理的特徴を検討した.その結果ANBP群はANR群に比べて,警戒心が強く,外界からの刺激に影響を受けやすく,周囲に合わせる傾向 …
- NAID 110009470831
Related Links
- Binge eating is a pattern of disordered eating which consists of episodes of uncontrollable eating. It is sometimes a symptom of binge eating disorder or compulsive overeating disorder. During such binges, a person rapidly consumes an ...
- Binge eating disorder (BED) is the most common eating disorder in the United States affecting 3.5% of females and 2% of males and is prevalent in up to 30% of those seeking weight loss treatment. Although it is not yet classified as a separate ...
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★リンクテーブル★
| リンク元 | 「過食」「bulimia」「bulimic」「polyphagia」「increased appetite」 |
| 関連記事 | 「eating」「binge」 |
「過食」
- 英
- bulimia、hyperphagia、binge eating、overfeeding、overeating、increased appetite、polyphagia、bulimic
- 関
- 栄養過剰、過食症、食欲亢進、食欲過剰、摂食亢進、多食症、気晴らし食い、大食、大食症
「bulimia」
- n.
- 関
- binge eating、binge eating disorder、bulimic、hyperphagia、increased appetite、overeating、overfeeding、polyphagia
「bulimic」
- adj.
- 過食症の、過食の
- 関
- binge eating、binge eating disorder、bulimia、hyperphagia、increased appetite、overeating、overfeeding、polyphagia
「polyphagia」
- n.
- (医)多食、多食症、過食、過食症
- 関
- binge eating, binge eating disorder, bulimia, bulimic, hyperphagia, increased appetite, overeating, overfeeding
「increased appetite」
「eating」
- n.
「binge」
- n.