関: epidermolysis bullosa acquisita

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locate (a moving entity) by means of a tracking system such as radar
win something through ones efforts; "I acquired a passing knowledge of Chinese"; "Gain an understanding of international finance" (同)win, gain
gotten through environmental forces; "acquired characteristics (such as a suntan or a broken nose) cannot be passed on"

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出典(authority):フリー百科事典『ウィキペディア（Wikipedia）』「2014/09/29 01:22:24」(JST)

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Epidermolysis bullosa acquisita is a chronic subepidermal blistering disease associated with autoimmunity to type VII collagen^[1] within anchoring fibril structures that are located at the dermoepidermal junction.^[2]^:609

References

^ Helen Chapel; Mansel Haeney; Siraj Misbah (2006). Essentials of clinical immunology. Wiley-Blackwell. pp. 207–. ISBN 978-1-4051-2761-5. Retrieved 25 June 2010.
^ Freedberg, et al. (2003). Fitzpatrick's Dermatology in General Medicine. (6th ed.). McGraw-Hill. ISBN 978-0-07-138076-8.

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1. 後天性表皮水疱症 epidermolysis bullosa acquisita
2. 表皮水疱症の疫学、病因、分類、および臨床的特徴 epidemiology pathogenesis classification and clinical features of epidermolysis bullosa
3. 表皮水疱症の診断 diagnosis of epidermolysis bullosa
4. 皮膚剥脱症候群 peeling skin syndrome
5. 遺伝性皮膚症 the genodermatoses

English Journal

Anti-p200 pemphigoid.

Goletz S1, Hashimoto T2, Zillikens D1, Schmidt E3.
Journal of the American Academy of Dermatology.J Am Acad Dermatol.2014 Jul;71(1):185-191. doi: 10.1016/j.jaad.2014.02.036. Epub 2014 Apr 24.
Anti-p200 pemphigoid is a rare subepidermal blistering skin disease. Patients' autoantibodies label the dermal side of 1 mol/L NaCl-split human skin by indirect immunofluorescence microscopy and recognize a 200-kd protein by immunoblotting of human dermal extract. Clinically, anti-p200 pemphigoid is
PMID 24767733

Esophageal involvement in epidermolysis bullosa acquisita.

Ishii N1, Furumura M, Hamada T, Mori O, Ohzono A, Ueda A, Karashima T, Nakama T, Tsuruta D, Takedatsu H, Fujita H, Hashimoto T.
The British journal of dermatology.Br J Dermatol.2014 Jun 28. doi: 10.1111/bjd.13224. [Epub ahead of print]
Epidermolysis bullosa acquisita (EBA) is an acquired autoimmune bullous disease of skin and mucous membranes characterized by IgG autoantibodies to type VII collagen, major component of the anchoring fibrils in basement membrane zone (BMZ).1 Type VII collagen is also expressed on esophageal mucosa.2
PMID 24975454

Peristomal Epidermolysis Bullosa Acquisita in a Patient with Crohn's Disease.

Ormaechea-Pérez N1, Tuneu-Valls A, Borja-Consigliere HA, Alcazar-Viladomiu ED, Jaka-Moreno A.
Acta dermato-venereologica.Acta Derm Venereol.2014 Jun 24;94(4):489-490. doi: 10.2340/00015555-1769.
Abstract is missing (Short).
PMID 24322876

Japanese Journal

Childhood epidermolysis bullosa acquisita with autoantibodies against the noncollagenous 1 and 2 domains of type VII collagen : case report and review of the literature

Mayuzumi M.,Akiyama M.,Nishie W.,Ukae S.,Abe M.,Sawamura D.,Hashimoto T.,Shimizu H.
British Journal of Dermatology 155(5), 1048-1052, 2006-11-00
… Epidermolysis bullosa acquisita (EBA) is an acquired subepidermal bullous disease characterized by IgG autoantibodies to type VII collagen, a major component of anchoring fibrils. …
NAID 120000955851