WordNet

medicine that prevents or retards the clotting of blood (同)anticoagulant medication, decoagulant
any of a large group of nitrogenous organic compounds that are essential constituents of living cells; consist of polymers of amino acids; essential in the diet of animals for growth and for repair of tissues; can be obtained from meat and eggs and milk and legumes; "a diet high in protein"
the 9th letter of the Roman alphabet (同)i
pertaining to or having or occurring by means of a placenta; "all mammals except monotremes and marsupials are placental mammals"
mammals having a placenta; all mammals except monotremes and marsupials (同)placental mammal, eutherian, eutherian mammal
that part of the ovary of a flowering plant where the ovules form
the vascular structure in the uterus of most mammals providing oxygen and nutrients for and transferring wastes from the developing fetus

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1. 静脈血栓塞栓症の患者における理論的根拠と無期限の抗凝固の適応 rationale and indications for indefinite anticoagulation in patients with venous thromboembolism
2. ワルファリンに関係した脳出血における抗凝固療法の反転 reversal of anticoagulation in warfarin associated intracerebral hemorrhage
3. 直接トロンビン阻害剤および直接Xa因子阻害剤による抗凝固療法 anticoagulation with direct thrombin inhibitors and direct factor xa inhibitors
4. 胎盤の発達および生理学 placental development and physiology
5. 下肢深部静脈血栓症：抗凝固療法の開始（開始後10日～3ヶ月） lower extremity deep venous thrombosis long term anticoagulation 10 days to three months

Quantitative chemical proteomics for investigating the biomarkers of dioscin against liver fibrosis caused by CCl4 in rats.

Zhang X1, Xu L, Yin L, Qi Y, Xu Y, Han X, Peng J.
Chemical communications (Cambridge, England).Chem Commun (Camb).2015 Jun 25;51(55):11064-7. doi: 10.1039/c4cc09160d.
In the present work, the effect of dioscin against liver fibrosis in rats caused by carbon tetrachloride (CCl4) was confirmed. Then, the differentially expressed proteins from rat liver were identified using two-dimensional differential in-gel electrophoresis technology. Ten new biomarkers including
PMID 26069897

ANXA3/JNK Signaling Promotes Self-Renewal and Tumor Growth, and Its Blockade Provides a Therapeutic Target for Hepatocellular Carcinoma.

Tong M1, Fung TM1, Luk ST1, Ng KY1, Lee TK2, Lin CH3, Yam JW2, Chan KW4, Ng F5, Zheng BJ5, Yuan YF6, Xie D6, Lo CM7, Man K7, Guan XY8, Ma S9.
Stem cell reports.Stem Cell Reports.2015 Jun 16. pii: S2213-6711(15)00156-3. doi: 10.1016/j.stemcr.2015.05.013. [Epub ahead of print]
Frequent tumor relapse in hepatocellular carcinoma (HCC) has been commonly attributed to the presence of residual cancer stem cells (CSCs) after conventional treatments. We have previously identified and characterized CD133 to mark a specific CSC subset in HCC. In the present study, we found endogen
PMID 26095609

Tumour antigen expression in hepatocellular carcinoma in a low-endemic western area.

Sideras K1, Bots SJ1, Biermann K2, Sprengers D1, Polak WG3, IJzermans JN3, de Man RA1, Pan Q1, Sleijfer S4, Bruno MJ1, Kwekkeboom J1.
British journal of cancer.Br J Cancer.2015 Jun 9;112(12):1911-20. doi: 10.1038/bjc.2015.92.
BACKGROUND: Identification of tumour antigens is crucial for the development of vaccination strategies against hepatocellular carcinoma (HCC). Most studies come from eastern-Asia, where hepatitis-B is the main cause of HCC. However, tumour antigen expression is poorly studied in low-endemic, western
PMID 26057582