ナトリウム利尿の、ナトリウム排泄増加の

関: natriuresis

WordNet

of or relating to natriuresis
the presence of abnormally large amounts of sodium in the urine

Wikipedia preview

出典(authority):フリー百科事典『ウィキペディア（Wikipedia）』「2016/08/08 20:49:44」(JST)

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Natriuresis is the process of sodium excretion in the urine through the action of the kidneys.^[1] It is promoted by ventricular and atrial natriuretic peptides as well as calcitonin,^[2] and inhibited by chemicals such as aldosterone. Natriuresis lowers the concentration of sodium in the blood and also tends to lower blood volume because osmotic forces drag water out of the body's blood circulation and into the urine along with the sodium. Many diuretic drugs take advantage of this mechanism to treat medical conditions like hypernatremia and hypertension, which involve excess blood volume.

Excess natriuresis can be caused by:

Medullary cystic disease
Bartter syndrome
Diuretic phase of acute tubular necrosis
Some diuretics
Primary renal diseases
Congenital adrenal hyperplasia
Syndrome of inappropriate antidiuretic hormone hypersecretion

Endogenous natriuretic hormones include:

Atrial natriuretic peptide
Brain natriuretic peptide
C-type natriuretic peptide ^[3]

References

^ "Natriuresis". Merriam-Webster. Retrieved 23 December 2013.
^ Boron, Walter F. and Boulpaep, Emile L. "Medical Physiology". Saunders, 2012, p.1108.
^ Levin ER, Gardner DG, Samson WK (July 1998). "Natriuretic peptides". The New England Journal of Medicine. 339 (5): 321–8. doi:10.1056/NEJM199807303390507. PMID 9682046.

Further reading

Granger, JP; Alexander, BT; Llinas, M (2002). "Mechanisms of pressure natriuresis". Current hypertension reports. 4 (2): 152–9. doi:10.1007/s11906-002-0040-3. PMID 11884271.
Hall, J. E.; Mizelle, H. L.; Hildebrandt, D. A.; Brands, M. W. (1990). "Abnormal pressure natriuresis. A cause or a consequence of hypertension?". Hypertension. 15 (6_Pt_1): 547–59. doi:10.1161/01.HYP.15.6.547. PMID 1971810.

UpToDate Contents

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1. 心不全におけるナトリウム利尿ペプチド測定 natriuretic peptide measurement in heart failure
2. Blood biomarkers for stroke
3. Pregnancy and valve disease
4. Deep venous thrombosis: Long-term anticoagulation (three to six months)
5. 非心不全下におけるナトリウム利尿ペプチド測定 natriuretic peptide measurement in non heart failure settings

English Journal

Oral Levosimendan Increases Cerebral Blood Flow Velocities in Patients with a History of Stroke or Transient Ischemic Attack: A Pilot Safety Study.

Kivikko M1, Kuoppamäki M2, Soinne L3, Sundberg S4, Pohjanjousi P2, Ellmen J2, Roine RO5.
Current therapeutic research, clinical and experimental.Curr Ther Res Clin Exp.2015 Jan 29;77:46-51. doi: 10.1016/j.curtheres.2015.01.001. eCollection 2015.
BACKGROUND: Intravenous levosimendan is indicated for acute heart failure. The compound has shown promising beneficial effects in ischemic stroke models.OBJECTIVE: We evaluated the efficacy and safety of oral levosimendan in patients with a history of cerebral ischemia.METHODS: In a randomized, doub
PMID 26082815

Application of the United States acute heart failure risk prediction model in Japanese patients; analysis from a contemporary multicenter registry.

Makino J1, Kohsaka S2, Shiraishi Y1, Inohara T1, Goda A3, Izumi Y4, Yagawa M4, Mizuno A5, Fukuda K1, Yoshikawa T4.
International journal of cardiology.Int J Cardiol.2015 Sep 15;195:323-5. doi: 10.1016/j.ijcard.2015.05.164. Epub 2015 Jun 2.
PMID 26056965

Targeted delivery of interleukin-10 to chronic cardiac allograft rejection using a human antibody specific to the extra domain A of fibronectin.

Franz M1, Doll F2, Grün K3, Richter P4, Köse N4, Ziffels B3, Schubert H5, Figulla HR3, Jung C3, Gummert J6, Renner A6, Neri D2, Berndt A4.
International journal of cardiology.Int J Cardiol.2015 Sep 15;195:311-22. doi: 10.1016/j.ijcard.2015.05.144. Epub 2015 May 29.
BACKGROUND AND AIMS: Management of chronic rejection is challenging since there are not sufficient preventive or therapeutic strategies. The rejection process leads to overexpression of ED-A(+) fibronectin (ED-A(+) Fn). The human antibody F8, specific to ED-A(+) Fn, may serve as a vehicle for target
PMID 26056964

Japanese Journal

Noninvasive Surrogate Markers to Predict Thrombogenesis in Patients with Nonvalvular Atrial Fibrillation

日大医学雑誌 = Journal of Nihon University Medical Association 75(2), 88-91, 2016-04
NAID 40020845083

Plasma atrial natriuretic peptide and N-terminal pro B-type natriuretic peptide concentrations in dogs with right-sided congestive heart failure

The journal of veterinary medical science 78(4), 535-542, 2016-04
NAID 40020843320

急性腎障害における低用量心房性ナトリウム利尿ペプチドの腎保護効果 (特集急性腎障害 : ブレークスルーを求めて)

細胞 48(3), 112-115, 2016-03
NAID 40020848018

「ナトリウム排泄増加」

　　[★]

英: natriuresis、natriuretic
関: ナトリウム利尿、ナトリウム排泄量

「natriuresis」

　　[★]

ナトリウム利尿、ナトリウム排泄増加

関: natriuretic

「atrial natriuretic peptide receptor」

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心房性ナトリウム利尿ペプチド受容体、心房性ナトリウム利尿ペプチドレセプター

関: ANP receptor

「atrial natriuretic factor」

　　[★]

同: 心房性ナトリウム利尿因子、心房性ナトリウム利尿ホルモン、ANF

「b-type natriuretic peptide」

　　[★] 脳性ナトリウム利尿ペプチド BNP

「brain natriuretic peptide」