WordNet

a small vascular growth on the surface of a mucous membrane (同)polypus
one of two forms that coelenterates take (e.g. a hydra or coral): usually sedentary with a hollow cylindrical body usually with a ring of tentacles around the mouth; "in some species of coelenterate, polyps are a phase in the life cycle that alternates with a medusoid phase"
of or relating to or characteristic of or appropriate for children or young people; "juvenile diabetes"; "juvenile fashions"
a young person, not fully developed (同)juvenile person

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The gastrointestinal manifestation of constitutional mismatch repair deficiency syndrome (CMMRD): from a single adenoma to polyposis-like phenotype and early onset cancer.

Levi Z1, Kariv R, Barnes-Kedar I, Goldberg Y, Half E, Morgentern S, Eli B, Baris HN, Vilkin A, Belfer RG, Niv Y, Elhasid R, Dvir R, Freha NA, Cohen S.
Clinical genetics.Clin Genet.2014 Oct 13. doi: 10.1111/cge.12518. [Epub ahead of print]
Data on the clinical presentation of constitutional mismatch repair deficiency syndrome (CMMRD) is accumulating. However, as the extra-intestinal manifestations are often fatal and occur at early age, data on the systematic evaluation of the gastrointestinal tract is scarce. Here we describe eleven
PMID 25307252

Identification of Coding Exon 3 Duplication in the BMPR1A Gene in a Patient with Juvenile Polyposis Syndrome.

Yamaguchi J1, Nagayama S2, Chino A3, Sakata A4, Yamamoto N4, Sato Y1, Ashihara Y1, Kita M1, Nomura S5, Ishikawa Y4, Igarashi M3, Ueno M2, Arai M6.
Japanese journal of clinical oncology.Jpn J Clin Oncol.2014 Aug 16. pii: hyu111. [Epub ahead of print]
Juvenile polyposis syndrome is an autosomal dominant inherited disorder characterized by multiple juvenile polyps arising in the gastrointestinal tract and an increased risk of gastrointestinal cancers, specifically colon cancer. BMPR1A and SMAD4 germline mutations have been found in patients with j
PMID 25129392

Appreciating the broad clinical features of SMAD4 mutation carriers: a multicenter chart review.

Wain KE1, Ellingson MS2, McDonald J3, Gammon A4, Roberts M5, Pichurin P6, Winship I7, Riegert-Johnson DL8, Weitzel JN9, Lindor NM10.
Genetics in medicine : official journal of the American College of Medical Genetics.Genet Med.2014 Aug;16(8):588-93. doi: 10.1038/gim.2014.5. Epub 2014 Feb 13.
Heterozygous loss-of-function SMAD4 mutations are associated with juvenile polyposis syndrome and hereditary hemorrhagic telangiectasia. Some carriers exhibit symptoms of both conditions, leading to juvenile polyposis-hereditary hemorrhagic telangiectasia syndrome. Three families have been reported
PMID 24525918

Han Jian,Kubota Shin,Li Guoxiang,Yao Xiaoyong,Yang Xiaoguang,Shu Degan,Li Yong,Kinoshita Shunichi,Sasaki Osamu,Komiya Tsuyoshi,Yan Gang
PloS one 8(8), 2013-08-12
… The well-developed endodermic lamellae and gastric pockets developed in the late embryonic stages of these cubozoan fossils are comparable with extant pelagic juvenile cubomedusae rather than sessile cubopolyps, whcih indicates a direct development in these fossil taxa, lacking characteristic stages of a typical cnidarian metagenesis such as planktonic planula and sessile polyps. …
NAID 120005323006

高山哲治,高岡遠,青木秀俊,岡本耕一
日本消化器内視鏡学会雑誌 55(6), 1787-1795, 2013
大腸癌の前病変としては，まず腺腫（adenoma）があげられ，腺腫はadenoma-carcinoma sequenceにより癌に進展する．次いで，traditional serrated adenoma（TSA）やsessile serrated adenoma/polyp（SSA/P）などの鋸歯状ポリープがあげられ，これらの病変はserrated pathwayにより癌に進展する．腺腫，TSA …
NAID 130003375308

大久保洋平,小出直彦,関野康,石曽根聡,丸山雅史,宮川眞一
Nippon Shokakibyo Gakkai Zasshi 110(6), 1022-1029, 2013
抗リン脂質抗体症候群に合併した胃限局性若年性ポリポーシスの1例を報告する．症例は42歳の女性．6年前に抗リン脂質抗体症候群と診断され，アスピリンの内服を行っていた．2年前に消化管内視鏡検査で数個の胃ポリープを指摘され，ポリープの数と大きさの増大を認め，生検にて胃限局性若年性ポリポーシスと診断された．胃ポリポーシスから出血を繰り返し，貧血および低アルブミン血症の増悪を認め，胃全摘術を施行した．
NAID 130003364382