- 関
- mdx mice、mdx mouse
WordNet
- produced by inbreeding
- the syllable naming the third (mediant) note of any major scale in solmization
PrepTutorEJDIC
- 生まれつきの(inborn) / 近親交配による
- ミ(全音階の第3音)
- mouseの複数形
UpToDate Contents
全文を閲覧するには購読必要です。 To read the full text you will need to subscribe.
- 1. 遺伝学およびゲノミクスのためのツール：研究用に育種された遺伝子改変マウス tools for genetics and genomics specially bred and genetically engineered mice
- 2. レプチンの生理学 physiology of leptin
- 3. 肥満の病因 pathogenesis of obesity
- 4. 慢性骨髄性白血病の細胞生物学および分子生物学 cellular and molecular biology of chronic myeloid leukemia
- 5. シャルコーマリートゥース病などの一次性の遺伝性運動感覚性ニューロパチー hereditary primary motor sensory neuropathies including charcot marie tooth disease
English Journal
- Opposing roles of miR-21 and miR-29 in the progression of fibrosis in Duchenne muscular dystrophy.
- Zanotti S1, Gibertini S1, Curcio M1, Savadori P1, Pasanisi B1, Morandi L1, Cornelio F1, Mantegazza R1, Mora M2.
- Biochimica et biophysica acta.Biochim Biophys Acta.2015 Jul;1852(7):1451-64. doi: 10.1016/j.bbadis.2015.04.013. Epub 2015 Apr 17.
- Excessive extracellular matrix deposition progressively replacing muscle fibres is the endpoint of most severe muscle diseases. Recent data indicate major involvement of microRNAs in regulating pro- and anti-fibrotic genes. To investigate the roles of miR-21 and miR-29 in muscle fibrosis in Duchenne
- PMID 25892183
- Altered ROS production, NF-κB activation and interleukin-6 gene expression induced by electrical stimulation in dystrophic mdx skeletal muscle cells.
- Henríquez-Olguín C1, Altamirano F2, Valladares D3, López JR4, Allen PD4, Jaimovich E5.
- Biochimica et biophysica acta.Biochim Biophys Acta.2015 Jul;1852(7):1410-9. doi: 10.1016/j.bbadis.2015.03.012. Epub 2015 Apr 7.
- Duchenne muscular dystrophy is a fatal X-linked genetic disease, caused by mutations in the dystrophin gene, which cause functional loss of this protein. This pathology is associated with an increased production of reactive oxygen (ROS) and nitrogen species. The aim of this work was to study the alt
- PMID 25857619
- Metformin increases peroxisome proliferator-activated receptor γ Co-activator-1α and utrophin a expression in dystrophic skeletal muscle.
- Ljubicic V1, Jasmin BJ1.
- Muscle & nerve.Muscle Nerve.2015 Jul;52(1):139-42. doi: 10.1002/mus.24692. Epub 2015 May 14.
- INTRODUCTION: Metformin (MET) stimulates skeletal muscle AMP-activated protein kinase (AMPK), a key phenotype remodeling protein with emerging therapeutic relevance for Duchenne muscular dystrophy (DMD). Our aim was to identify the mechanism of impact of MET on dystrophic muscle.METHODS: We investig
- PMID 25908446
Related Links
- Mice, Inbred Mdx. On-line free medical diagnosis assistant. Ranked list of possible diseases from either several symptoms or a full patient history. A similarity measure between symptoms and diseases is provided. ... Category:Inbred mdx mice ...
- A strain of mice arising from a spontaneous MUTATION (mdx) in inbred C57BL mice. This mutation is X chromosome-linked and produces viable homozygous animals that lack the muscle protein DYSTROPHIN, have high serum ...
Related Pictures
★リンクテーブル★
| リンク元 | 「純系mdxマウス」「mdx mice」「近交系mdxマウス」 |
| 関連記事 | 「inbred」「mi」「mdx mice」 |
「純系mdxマウス」
「mdx mice」
- 関
- inbred mdx mice、mdx mouse
「近交系mdxマウス」
「inbred」
- 近交系の、純系の、同系繁殖の、同系交配の
「mi」
- mi-, mio-
- conb form
- より小さい、より少ない。劣った
- ex.
- ？
- MI
「mdx mice」
- 関
- inbred mdx mice、mdx mouse