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1. 負荷時の放射性核種心筋血流イメージングの概要 overview of stress radionuclide myocardial perfusion imaging
2. アデノシンデアミナーゼ欠損症：治療 adenosine deaminase deficiency treatment
3. アデノシンデアミナーゼ欠損症：病因、臨床症状、および診断 adenosine deaminase deficiency pathogenesis clinical manifestations and diagnosis
4. 拡張能障害の細胞学的機序 cellular mechanisms of diastolic dysfunction
5. 心筋における興奮収縮連関 excitation contraction coupling in myocardium

English Journal

MiR-429 increases the metastatic capability of HCC via regulating classic Wnt pathway rather than epithelial-mesenchymal transition.

Tang J1, Li L1, Huang W2, Sui C3, Yang Y4, Lin X4, Hou G4, Chen X4, Fu J4, Yuan S3, Li S5, Wen W4, Tang S4, Cao D4, Wu M1, Chen L6, Wang H7.
Cancer letters.Cancer Lett.2015 Aug 1;364(1):33-43. doi: 10.1016/j.canlet.2015.04.023. Epub 2015 Apr 27.
Epigenetic modification of miR-429 can manipulate liver T-ICs via targeting the RBBP4/E2F1/Oct4 axis, which might be crucial for hepatocarcinogenesis. However, whether miR-429 plays a role in regulating metastasis of hepatocellular carcinoma is still unclear. Using quantitative methylation analysis
PMID 25931210

Teimourian S1, Moghanloo E2.
Molecular immunology.Mol Immunol.2015 Aug;66(2):319-24. doi: 10.1016/j.molimm.2015.03.251. Epub 2015 Apr 26.
NETosis has been associated with a particular mode of cell death although it is still controversial as to what extent autophagy is involved in NETosis. Class I/AKT/mTOR pathway is a key regulator of autophagy. PTEN tumor suppressor gene encodes a dual specificity phosphatase that antagonizes the pho
PMID 25913476

Molecular profiling of ETS and non-ETS aberrations in prostate cancer patients from northern India.

Ateeq B1, Kunju LP2,3,4, Carskadon SL2,3, Pandey SK1, Singh G5, Pradeep I5, Tandon V6, Singhai A7, Goel A8, Amit S9, Agarwal A9, Dinda AK5, Seth A10, Tsodikov A11, Chinnaiyan AM2,3,4,12, Palanisamy N2,13.
The Prostate.Prostate.2015 Jul 1;75(10):1051-62. doi: 10.1002/pros.22989. Epub 2015 Mar 23.
BACKGROUND: Molecular stratification of prostate cancer (PCa) based on genetic aberrations including ETS or RAF gene-rearrangements, PTEN deletion, and SPINK1 over-expression show clear prognostic and diagnostic utility. Gene rearrangements involving ETS transcription factors are frequent pathogenet
PMID 25809148