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the 14th letter of the Roman alphabet (同)n

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nitrogenの化学記号
neodymiumの化学記号

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1. 緩和ケア：末期肝不全palliative care end stage liver disease [show details]
…illness and approach the end of life. The hope of a life-saving transplant can lead patients (and their providers) to avoid discussions of significant issues, including ACP and end of life issues, even as …
2. 末期関節リウマチの評価および内科的マネージメントevaluation and medical management of end stage rheumatoid arthritis [show details]
…of patients with end-stage RA. The medical management of patients with active disease and the surgical management of those with end-stage RA are discussed in detail separately. End-stage rheumatoid …
3. 末期腎不全の保存療法conservative care of end stage renal disease [show details]
…emotion such as worries and fears of end of life. We recommend exploring these patients concerns and readdressing goals of care. For seriously ill patients who are near the end of life, this approach may also …
4. 終末期の栄養補給および水分補給の停止stopping nutrition and hydration at the end of life [show details]
…patients at the end of life, we suggest not providing artificial nutrition . For most patients at the end of life, we suggest not providing artificial hydration, particularly for those with end-stage cancer …
5. 末期腎疾患患者の肺高血圧症pulmonary hypertension in patients with end stage renal disease [show details]
… Patients with end-stage renal disease (ESRD), particularly those on hemodialysis, are at risk for development of or worsening of preexisting pulmonary hypertension (PH). Although multifactorial, the increased …

Japanese Journal

HP-030-1 ヒストンH3K4メチルトランスフェラーゼであるSMYD3はN末側cleavageによりメチル化酵素活性を増強する(大腸(発癌・悪性度),ハイブリッドポスター,第109回日本外科学会定期学術集会)

國崎真己,荒木政人,飛永修一,田中研次,福岡秀敏,竹下浩明,日高重和,七島篤志,澤井照光,安武亨,永安武
日本外科学会雑誌 110(臨時増刊号_2), 445, 2009-02-25
NAID 110007166719

肺癌抑制因子CADM1接着分子のshedding：N末側断片と分泌型アイソフォームとの分子的・機能的相違

萩山満,伊藤彰彦,松本真明,長柄雄介,石浦章一,横崎宏
日本分子腫瘍マーカー研究会誌 23, 31-32, 2008
NAID 130004436410

Ｄｏｍｉｎａｎｔ‐ｎｅｇａｔｉｖｅ　ＥＢＮＡ１によるＥＢウイルス腫ようの抑制

今井章介,黒田正幸,山下竜右,石浦嘉人
ウイルス 55(2), 239-249, 2005
… ゲノム（episome）が感染細胞内で複製・維持されるために必須な唯一のウイルス蛋白である．したがってEBNA1は，格好のEBV陽性腫瘍の治療標的分子になりうると考えられる．我々は，野生型（wild-type, wt）EBNA1のN末側ドメインの大半を欠失するEBNA1変異体を独自に作製，これがwtEBNA1の機能を阻害することで，latency type，cell typeを問わず高率に感染細胞からEBV episome脱落を促進するdominant-negative（dn）EBNA1であることを明らかに …
NAID 130004470889