WordNet

the 3rd letter of the Roman alphabet (同)c
(music) the keynote of the scale of C major
a general-purpose programing language closely associated with the UNIX operating system

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Inhibition of endogenous heat shock protein 70 attenuates inducible nitric oxide synthase induction via disruption of heat shock protein 70/Na(+) /H(+) exchanger 1-Ca(2+) -calcium-calmodulin-dependent protein kinase II/transforming growth factor β-activated kinase 1-nuclear factor-κB signals in BV-2 microglia.

Huang C1, Lu X1, Wang J1, Tong L1, Jiang B1, Zhang W1.
Journal of neuroscience research.J Neurosci Res.2015 Aug;93(8):1192-202. doi: 10.1002/jnr.23571. Epub 2015 Feb 17.
Inducible nitric oxide synthase (iNOS) critically contributes to inflammation and host defense. The inhibition of heat shock protein 70 (Hsp70) prevents iNOS induction in lipopolysaccharide (LPS)-stimulated macrophages. However, the role and mechanism of endogenous Hsp70 in iNOS induction in microgl
PMID 25691123

Ca2+ channel subunit α 1D promotes proliferation and migration of endometrial cancer cells mediated by 17β-estradiol via the G protein-coupled estrogen receptor.

Hao J1, Bao X1, Jin B1, Wang X1, Mao Z1, Li X1, Wei L1, Shen D1, Wang JL2.
FASEB journal : official publication of the Federation of American Societies for Experimental Biology.FASEB J.2015 Jul;29(7):2883-93. doi: 10.1096/fj.14-265603. Epub 2015 Mar 24.
Calcium and calcium channels are closely related to the estrogen-induced nongenomic effect of endometrial carcinoma, but the specific role of calcium channels is unknown. This study aimed to explore the expression and the biologic effect of the L-type calcium channel in endometrial carcinoma cells a
PMID 25805831

Spontaneous Vesicle Release Is Not Tightly Coupled to Voltage-Gated Calcium Channel-Mediated Ca2+ Influx and Is Triggered by a Ca2+ Sensor Other Than Synaptotagmin-2 at the Juvenile Mice Calyx of Held Synapses.

Dai J1, Chen P2, Tian H1, Sun J3.
The Journal of neuroscience : the official journal of the Society for Neuroscience.J Neurosci.2015 Jul 1;35(26):9632-7. doi: 10.1523/JNEUROSCI.0457-15.2015.
It is well known that voltage-gated calcium channels (VGCCs)-mediated Ca(2+) influx triggers evoked synaptic vesicle release. However, the mechanisms of Ca(2+) regulation of spontaneous miniature vesicle release (mini) remain poorly understood. Here we show that blocking VGCCs at the juvenile mice (
PMID 26134646