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- 1. 川崎病：疫学および病因 kawasaki disease epidemiology and etiology
- 2. 分娩の生理学 physiology of parturition
- 3. T細胞受容体シグナル伝達 t cell receptor signaling
- 4. 血小板生物学 platelet biology
English Journal
- Ca(2+) Signalling in Endothelial Progenitor Cells: Friend or Foe?
- Moccia F1, Guerra G2.
- Journal of cellular physiology.J Cell Physiol.2016 Feb;231(2):314-27. doi: 10.1002/jcp.25126.
- Endothelial progenitor cells (EPCs) are mobilized either from the bone marrow and/or the arterial to replace dysfunctional endothelial cells and rescue blood perfusion in ischemic tissues. In addition, they may contribute to the angiogenic switch, thereby sustaining tumour growth and metastatization
- PMID 26247172
- Regulation of insulin signaling in skeletal muscle by PIP3 phosphatase, SKIP, and endoplasmic reticulum molecular chaperone glucose-regulated protein 78.
- Ijuin T1, Hatano N2, Hosooka T3, Takenawa T4.
- Biochimica et biophysica acta.Biochim Biophys Acta.2015 Dec;1853(12):3192-201. doi: 10.1016/j.bbamcr.2015.09.009. Epub 2015 Sep 14.
- Insulin resistance is characterized as a pathogenic factor in type 2 diabetes. Despite skeletal muscle being primarily responsible for systemic glucose disposal, the mechanisms underlying the induction of insulin resistance in skeletal muscle have not been fully elucidated. A number of studies have
- PMID 26376412
- Involvement of PRIP (Phospholipase C-Related But Catalytically Inactive Protein) in BMP-Induced Smad Signaling in Osteoblast Differentiation.
- Kotani M1,2, Matsuda M1, Murakami A1, Takahashi I2, Katagiri T3, Hirata M1.
- Journal of cellular biochemistry.J Cell Biochem.2015 Dec;116(12):2814-23. doi: 10.1002/jcb.25228.
- Phospholipase C-related but catalytically inactive protein (PRIP) was first isolated as an inositol 1,4,5-trisphosphate binding protein. We generated PRIP gene-deficient mice which exhibited the increased bone mineral density and trabecular bone volume, indicating that PRIP is implicated in the regu
- PMID 25981537
Japanese Journal
- Role of Inositol-1,4,5-Trisphosphate Receptor in the Regulation of Calcium Transients in Neonatal Rat Ventricular Myocytes
- Zeng Zheng,Zhang Heping,Lin Na [他]
- Journal of pharmacological sciences 126(1), 37-46, 2014-09
- NAID 40020200052
- Role of Inositol-1,4,5-Trisphosphate Receptor in the Regulation of Calcium Transients in Neonatal Rat Ventricular Myocytes
- Zeng Zheng,Zhang Heping,Lin Na,Kang Man,Zheng Yuanyuan,Li Chen,Xu Pingxiang,Wu Yongquan,Luo Dali
- Journal of Pharmacological Sciences 126(1), 37-46, 2014
- … This study determined the regulatory effect of inositol 1,4,5-trisphosphate receptors (IP3Rs) on the basal Ca2+ transients in cardiomyocytes. …
- NAID 130004677803
- Current Perspective : Regulatory Mechanisms and Pathophysiological Significance of IP₃ Receptors and Ryanodine Receptors in Drug Dependence
- Mizuno Koji,Kurokawa Kazuhiro,Ohkuma Seitaro
- Journal of pharmacological sciences 123(4), 306-311, 2013-12
- NAID 40019923730
Related Links
- Phosphatidylinositol (3,4,5)-triphosphate (PtdIns(3,4,5)P3), abbreviated PIP3, is the product of the class I phosphoinositide 3-kinases (PI 3-kinases) phosphorylation on phosphatidylinositol (4,5)-bisphosphate (PIP2).
Related Pictures
★リンクテーブル★
| リンク元 | 「triphosphate」「トリスリン酸」 |
| 拡張検索 | 「inositol 1,4,5-trisphosphate receptor」「myo-inositol 1,4,5-trisphosphate」 |
「triphosphate」
「トリスリン酸」
- 英
- trisphosphate
- 同
- 2,3-ジブロモプロピルリン酸
「inositol 1,4,5-trisphosphate receptor」
[★] イノシトール1,4,5-三リン酸受容体、イノシトール三リン酸受容体、イノシトール三リン酸レセプター、イノシトール3リン酸受容体、イノシトール3リン酸レセプター、IP3 receptor、IP3R
「myo-inositol 1,4,5-trisphosphate」
ミオイノシトール1