- adj.
- 門脈の
WordNet
- a grand and imposing entrance (often extended metaphorically); "the portals of the cathedral"; "the portals of heaven"; "the portals of success"
- turn or go to the port or left side, of a ship; "The big ship was slowly porting"
- an opening (in a wall or ship or armored vehicle) for firing through (同)embrasure, porthole
- sweet dark-red dessert wine originally from Portugal (同)port wine
- a place (seaport or airport) where people and merchandise can enter or leave a country
- located on the left side of a ship or aircraft (同)larboard
- bring to port; "the captain ported the ship at night"
- carry or hold with both hands diagonally across the body, especially of weapons; "port a rifle"
- carry, bear, convey, or bring; "The small canoe could be ported easily"
- drink port; "We were porting all in the club after dinner"
- land at or reach a port; "The ship finally ported"
- modify (software) for use on a different machine or platform
- put or turn on the left side, of a ship; "port the helm"
PrepTutorEJDIC
- 〈C〉(堂々とした)門,入り口 / 《複数形》《文》(大きな建物の)正門,表玄関 / 《複数形》《文》(…の)発端《+『of』+『名』》
- 〈C〉〈U〉『港』 / 〈C〉『港町』,『港市』
- (船の)左舷(さげん);(飛行機の)(機首に向かって)左側 / 左舷の,左側の / 〈かじなど〉‘を'左舷に向ける
- =porthole / (船の)荷役口 / (装甲車・城壁などの)銃眼,砲門
- ポートワイン(ポルトガル原産の甘口のぶどう酒で深紅色)
- 控え銃(つつ)の姿勢(銃をからだの正面に斜めに保持し先端が左肩の近くにくる) / 〈銃など〉‘を'控え銃にする
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出典(authority):フリー百科事典『ウィキペディア（Wikipedia）』「2013/03/31 14:26:44」(JST)
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- 1. 成人における門脈血栓症の疫学および病因 epidemiology and pathogenesis of portal vein thrombosis in adults
- 2. 成人における慢性門脈血栓症：臨床症状、診断、および管理 chronic portal vein thrombosis in adults clinical manifestations diagnosis and management
- 3. 成人における門脈圧亢進症 portal hypertension in adults
- 4. 肝細胞癌に対する治療法の概要 overview of treatment approaches for hepatocellular carcinoma
- 5. 非肝硬変性門脈圧亢進症 noncirrhotic portal hypertension
English Journal
- Pulmonary delivery of nanosized alendronate for decorporation of inhaled heavy metals: formulation development, characterization and gamma scintigraphic evaluation.
- Sultana S1, Bhatnagar A, Rawat H, Nishad DK, Talegaonkar S, Ahmad FJ, Mittal G.Author information 1Department of Pharmaceutics, Faculty of Pharmacy, Hamdard University , New Delhi , India and.AbstractCONTEXT: Medical management of heavy metal toxicity including radioactive ones is the cause of concern because of their increased use in energy production, healthcare and mining. As inhalation is one of the primary routes for internalization, a formulation is needed to trap metal(s) at the portal of entry itself.
- Pharmaceutical development and technology.Pharm Dev Technol.2014 Aug;19(5):623-33. doi: 10.3109/10837450.2013.819013. Epub 2013 Jul 25.
- CONTEXT: Medical management of heavy metal toxicity including radioactive ones is the cause of concern because of their increased use in energy production, healthcare and mining. As inhalation is one of the primary routes for internalization, a formulation is needed to trap metal(s) at the portal of
- PMID 23883097
- The importance of liver microcirculation in promoting autoimmune hepatitis via maintaining an inflammatory cytokine milieu - A mathematical model study.
- Lettmann KA1, Hardtke-Wolenski M2.Author information 1Kirchplatz 3, 27305 Bruchhausen-Vilsen, Germany; Institute for Chemistry and Biology of the Marine Environment, Carl von Ossietzky University, Oldenburg, Germany. Electronic address: karsten.lettmann@uni-oldenburg.de.2Hannover Medical School, Department of Gastroenterology, Hepatology and Endocrinology, Germany.AbstractIn autoimmune diseases, inflammatory cytokine concentrations are important for initiating and maintaining the status of autoimmunity. Autoimmune hepatitis (AIH) is an inflammatory liver disease characterized by a loss of immune tolerance against specific antigens located in hepatocytes. During the progression of the disease, antigen-presenting cells and different classes of T-helper cells secrete specific cytokines important for maintaining the disease. As these cytokines are secreted into the local liver environment, the blood flow in liver sinusoids might influence the local cytokine concentration. Considering the liver tissue as a porous medium, based on Darcy׳s law, the microcirculation within a liver lobule was modelled. Using realistic physiological pressure differences and tissue permeabilities, the blood velocity inside the sinusoids could be calculated and validated with blood velocity data obtained via Orthogonal Polarization Spectral Imaging (OPSI). Furthermore, oxygen consumption is modelled to obtain Rappaport׳s acinus model. Finally, steady state spatial distributions of secreted cytokines within the liver lobule could be estimated for specified realistic production rates of T-helper cells. It could be demonstrated that the characteristics of the liver microcirculation have an important impact on establishing inflammatory cytokine levels within the portal fields and the vascular septa promoting the occurrence of interface hepatitis.
- Journal of theoretical biology.J Theor Biol.2014 May 7;348:33-46. doi: 10.1016/j.jtbi.2014.01.016. Epub 2014 Jan 28.
- In autoimmune diseases, inflammatory cytokine concentrations are important for initiating and maintaining the status of autoimmunity. Autoimmune hepatitis (AIH) is an inflammatory liver disease characterized by a loss of immune tolerance against specific antigens located in hepatocytes. During the p
- PMID 24486232
- The anti-fibrotic effects of CCN1/CYR61 in primary portal myofibroblasts are mediated through induction of reactive oxygen species resulting in cellular senescence, apoptosis and attenuated TGF-β signaling.
- Borkham-Kamphorst E1, Schaffrath C2, Van de Leur E2, Haas U2, Tihaa L2, Meurer SK2, Nevzorova YA3, Liedtke C3, Weiskirchen R4.Author information 1Institute of Clinical Chemistry and Pathobiochemistry, RWTH Aachen University Hospital, Germany. Electronic address: ekamphorst@ukaachen.de.2Institute of Clinical Chemistry and Pathobiochemistry, RWTH Aachen University Hospital, Germany.3Department of Internal Medicine III, RWTH Aachen University Hospital, Germany.4Institute of Clinical Chemistry and Pathobiochemistry, RWTH Aachen University Hospital, Germany. Electronic address: rweiskirchen@ukaachen.de.AbstractCysteine-rich protein 61 (CCN1/CYR61) is a CCN (CYR61, CTGF (connective tissue growth factor), and NOV (Nephroblastoma overexpressed gene)) family matricellular protein comprising six secreted CCN proteins in mammals. CCN1/CYR61 expression is associated with inflammation and injury repair. Recent studies show that CCN1/CYR61 limits fibrosis in models of cutaneous wound healing by inducing cellular senescence in myofibroblasts of the granulation tissue which thereby transforms into an extracellular matrix-degrading phenotype. We here investigate CCN1/CYR61 expression in primary profibrogenic liver cells (i.e., hepatic stellate cells and periportal myofibroblasts) and found an increase of CCN1/CYR61 expression during early activation of hepatic stellate cells that declines in fully transdifferentiated myofibroblasts. By contrast, CCN1/CYR61 levels found in primary parenchymal liver cells (i.e., hepatocytes) were relatively low compared to the levels exhibited in hepatic stellate cells and portal myofibroblasts. In models of ongoing liver fibrogenesis, elevated levels of CCN1/CYR61 were particularly noticed during early periods of insult, while expression declined during prolonged phases of fibrogenesis. We generated an adenovirus type 5 encoding CCN1/CYR61 (i.e., Ad5-CMV-CCN1/CYR61) and overexpressed CCN1/CYR61 in primary portal myofibroblasts. Interestingly, overexpressed CCN1/CYR61 significantly inhibited production of collagen type I at both mRNA and protein levels as evidenced by quantitative real-time polymerase chain reaction, Western blot and immunocytochemistry. CCN1/CYR61 further induces production of reactive oxygen species (ROS) leading to dose-dependent cellular senescence and apoptosis. Additionally, we demonstrate that CCN1/CYR61 attenuates TGF-β signaling by scavenging TGF-β thereby mitigating in vivo liver fibrogenesis in a bile duct ligation model. Conclusion: In line with dermal fibrosis and scar formation, CCN1/CYR61 is involved in liver injury repair and tissue remodeling. CCN1/CYR61 gene transfer into extracellular matrix-producing liver cells is therefore potentially beneficial in liver fibrotic therapy.
- Biochimica et biophysica acta.Biochim Biophys Acta.2014 May;1843(5):902-14. doi: 10.1016/j.bbamcr.2014.01.023. Epub 2014 Jan 31.
- Cysteine-rich protein 61 (CCN1/CYR61) is a CCN (CYR61, CTGF (connective tissue growth factor), and NOV (Nephroblastoma overexpressed gene)) family matricellular protein comprising six secreted CCN proteins in mammals. CCN1/CYR61 expression is associated with inflammation and injury repair. Recent st
- PMID 24487063
- Portal hypertension.
- Herrera JL.
- Clinics in liver disease.Clin Liver Dis.2014 May;18(2):xiii-xiv. doi: 10.1016/j.cld.2014.02.001.
- PMID 24679508
Japanese Journal
- Chylothorax and Bilious Pleural Effusion
- Yokoe Norihito,Yamaguchi Etsuro
- Internal Medicine, 2020
- NAID 130007677021
- A Case of Polyarteritis Nodosa with Marked Eosinophilia, Associated with Severe Gastrointestinal Tract Involvement and Recurrent Venous Thrombosis
- Oiwa Hiroshi,Taniguchi Kohei,Miyoshi Natsuki,Sasaki Keiko,Ichimura Kouichi,Kubota Tetsushi,Sato Daisuke
- Internal Medicine, 2020
- … Histopathology showed subserous medium-sized arteritis with abundant eosinophil infiltrates and thrombosis in the portal vein branches. …
- NAID 130007668768
- 全学的学習支援システムの連携とそれに基づく安否確認システムの開発
- 中野 裕司
- 情報処理学会論文誌教育とコンピュータ（TCE） 5(2), 12-19, 2019-06-28
- 2000年前後から大学の各種手続きや学習環境のオンライン化が始まり，その数が増えるにつれて，各々のシステムでのログイン，同じような情報の多重入力，システムごとの情報の差異等が問題になってきた．そのため，シングルサインオン，学務情報データの共有，大学ポータルの導入，標準化等により，学務情報システム，学習管理システム，シラバス，時間割，学修成果可視化等様々な学習支援システムの全学的な連携を行ってきた． …
- NAID 170000150393
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