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出典(authority):フリー百科事典『ウィキペディア（Wikipedia）』「2016/07/27 23:56:08」(JST)

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1. 無症候性頸動脈アテローム性動脈硬化症のマネージメント management of asymptomatic carotid atherosclerotic disease
2. 無症候性頸動脈狭窄のスクリーニング screening for asymptomatic carotid artery stenosis
3. 頚動脈内膜剥離術 carotid endarterectomy
4. 頸動脈硬化の症状の病態生理 pathophysiology of symptoms from carotid atherosclerosis
5. 頭蓋内大血管の動脈硬化 intracranial large artery atherosclerosis

"True" Accessory Anterior Cerebral Artery: A Newly Reported Anterior Cerebral Arterial Anomaly and a Proposal for Its Classification.

Shibata Y1, Mashiko R2.
Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association.J Stroke Cerebrovasc Dis.2016 May 20. pii: S1052-3057(16)30024-6. doi: 10.1016/j.jstrokecerebrovasdis.2016.04.015. [Epub ahead of print]
If we use the same naming policy of middle cerebral arterial anomalies, then accessory anterior cerebral arteries (ACAs) should be applied for ACA anomalies originating from the ACA. We experienced such a rare ACA anomaly, which we referred to as true accessory ACA.Copyright © 2016 National Stroke
PMID 27216379

A pseudouridylation switch in rRNA is implicated in ribosome function during the life cycle of Trypanosoma brucei.

Chikne V1, Doniger T1, Rajan KS1, Bartok O2, Eliaz D1, Cohen-Chalamish S1, Tschudi C3, Unger R1, Hashem Y4, Kadener S2, Michaeli S1.
Scientific reports.Sci Rep.2016 May 4;6:25296. doi: 10.1038/srep25296.
The protozoan parasite Trypanosoma brucei, which causes devastating diseases in humans and animals in sub-Saharan Africa, undergoes a complex life cycle between the mammalian host and the blood-feeding tsetse fly vector. However, little is known about how the parasite performs most molecular functio
PMID 27142987

Co-inhibition of EGFR and IGF1R synergistically impacts therapeutically on adrenocortical carcinoma.

Xu L1, Qi Y2, Xu Y1,3, Lian J1, Wang X1, Ning G2, Wang W2, Zhu Y1.
Oncotarget.Oncotarget.2016 Apr 18. doi: 10.18632/oncotarget.8827. [Epub ahead of print]
PURPOSE: Adrenocortical carcinoma (ACC) is a rare tumor with very poor prognosis and no effective treatment. The aim of this study was to explore a novel therapy co-targeting EGFR and IGF1R in vitro and vivo.METHODS: The expression of EGFR and IGF1R were evaluated in a series of adrenocortical tumor
PMID 27105537