WordNet

the 18th letter of the Roman alphabet (同)r
a tributary of the Mississippi River that flows eastward from Texas along the southern boundary of Oklahoma and through Louisiana (同)Red River

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1. 遺伝性のびまん性胃癌 hereditary diffuse gastric cancer
2. 転移性乳癌における予後および予測因子 prognostic and predictive factors in metastatic breast cancer
3. 頭頸部の扁平上皮癌化：分子学的および遺伝学的変化 head and neck squamous cell carcinogenesis molecular and genetic alterations
4. 前立腺癌の分子生物学 molecular biology of prostate cancer
5. 胃癌の病理および分子学的病因 pathology and molecular pathogenesis of gastric cancer

Autophagy induction impairs migration and invasion by reversing EMT in glioblastoma cells.

Catalano M1, D'Alessandro G1, Lepore F2, Corazzari M3, Caldarola S2, Valacca C4, Faienza F2, Esposito V5, Limatola C1, Cecconi F6, Di Bartolomeo S7.
Molecular oncology.Mol Oncol.2015 Oct;9(8):1612-25. doi: 10.1016/j.molonc.2015.04.016. Epub 2015 May 13.
Cell migration and invasion are highly regulated processes involved in both physiological and pathological conditions. Here we show that autophagy modulation regulates the migration and invasion capabilities of glioblastoma (GBM) cells. We observed that during autophagy occurrence, obtained by nutri
PMID 26022108

Epithelial morphological reversion drives Profilin-1-induced elevation of p27(kip1) in mesenchymal triple-negative human breast cancer cells through AMP-activated protein kinase activation.

Jiang C1, Veon W1, Li H2, Hallows KR2,3, Roy P1,4,5.
Cell cycle (Georgetown, Tex.).Cell Cycle.2015 Sep 17;14(18):2914-23. doi: 10.1080/15384101.2015.1069929. Epub 2015 Jul 15.
Profilin-1 (Pfn1) is an important regulator of actin polymerization that is downregulated in human breast cancer. Previous studies have shown Pfn1 has a tumor-suppressive effect on mesenchymal-like triple-negative breast cancer cells, and Pfn1-induced growth suppression is partly mediated by upregul
PMID 26176334

Appolloni I1, Barilari M1, Caviglia S2, Gambini E2, Reisoli E1, Malatesta P3.
Oncogene.Oncogene.2015 Apr 9;34(15):1991-2002. doi: 10.1038/onc.2014.122. Epub 2014 May 26.
Although the infiltrative behavior of malignant gliomas is one of their most critical aspects, the mechanisms underlying it have not yet been elucidated. To migrate in the brain parenchyma, malignant glioma cells need to bypass the cell-cell contact inhibitory signals. Here we propose that the blind
PMID 24858041

平岡智之
杏林医学会雑誌 38(4), 93-101, 2007
… 網膜再生能を有するアカハライモリ<I>Cynops pyrrhogaster</I>を用い神経発生に重要な役割を果たすneural cell adhesion molecule（NCAM）およびN-，R-cadherinの網膜再生過程での眼内発現様式を免疫組織化学的手法で調べた。 … 再生眼では術後3週で網膜辺縁部のN-cadherinの発現が観察されるのみで他の分子は未発現だった。 …
NAID 130000135251

Shibuya Yasuyuki,Takeuchi Junichiro,Kobayashi Masako,Murata Maho,Munemoto Sachiko,Yokoo Satoshi,Umeda Masahiro,Komori Takahide
The Kobe journal of the medical sciences 51(3/4), 35-40, 2005-08
NAID 120000941682

Shibuya Yasuyuki,Takeuchi Junichiro,Kobayashi Masaki,Murata Maho,Munemoto Sachiko,Yokoo Satoshi,Umeda Masahiro,Komori Takahide
The Kobe journal of the medical sciences 51(3/4), 35-40, 2005
… Localizations of N- and R-cadherins in dorsal root ganglia and lumbar spinal cord of the chick were immunocytochemically examined. … As a result, N- and R-cadherin appeared to be co-localized on plasmalemma of neurons in dorsal root ganglia that were in contact with satellite cells, whereas there was no expression in neurons of the ventral horn. …
NAID 110004091713