WordNet

the 2nd letter of the Roman alphabet (同)b
the blood group whose red cells carry the B antigen (同)type_B, group B

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出典(authority):フリー百科事典『ウィキペディア（Wikipedia）』「2013/03/21 17:22:46」(JST)

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This article is about the bank. For the computer game, see BnB.

"BNB" redirects here. For the Congolese airport with this IATA airport code, see Boende Airport.

The historical building of BNB today (now occupied by Office of Foreign economic affairs of Bulgarian Ministry of Economy, Energy and Tourism^[1]

The Bulgarian National Bank (Bulgarian: Българска народна банка, Balgarska narodna banka, IPA: [bəːlɡarskɐ narodnɐ banka]) is the central bank of the Republic of Bulgaria with its headquarters in Sofia. The BNB was established on 25 January 1879.

It is an independent institution responsible for issuing all banknotes and coins in the country, overseeing and regulating the banking sector and keeping the government's currency reserves. The BNB is also the sole owner of the Bulgarian Mint. The governor is Ivan Iskrov.

Headquarters

The Bulgarian National Bank's headquarters in Sofia are located on the central Battenberg Square. The current edifice was commissioned to the noted architects Ivan Vasilyov and Dimitar Tsolov and built between 1934 and 1939 in the strict and non-decorative Neoclassical style of the time. It spreads over an area of 3,700 m² and has four overground and three underground storeys. The inner decoration is the work of Ivan Penkov and Dechko Uzunov.

Notes

^ (Bulgarian) МИEТ, Европейска интеграция

References

Georgieva, Silviya (12 April 2006). "Сградата на БНБ респектира със строга красота" (in Bulgarian). Sega. http://www.segabg.com/online/article.asp?issueid=2211&sectionid=17&id=0002201. Retrieved 30 October 2006.

External links

(Bulgarian) (English) Bulgarian National Bank official site
Gallery of Bulgarian paper money

Coordinates: 42°41′47″N 23°19′31″E / 42.69639°N 23.32528°E / 42.69639; 23.32528

English Journal

Biology of the blood-nerve barrier and its alteration in immune mediated neuropathies.

Kanda T.SourceDepartment of Neurology and Clinical Neuroscience, Yamaguchi University Graduate School of Medicine, 1-1-1 Minamikogushi, Ube, Yamaguchi 7558505, Japan; tkanda@yamaguchi-u.ac.jp.
Journal of neurology, neurosurgery, and psychiatry.J Neurol Neurosurg Psychiatry.2013 Feb;84(2):208-12. doi: 10.1136/jnnp-2012-302312. Epub 2012 Dec 13.
The blood-nerve barrier (BNB) is a dynamic and competent interface between the endoneurial microenvironment and the surrounding extracellular space or blood. It is localised at the innermost layer of the multilayered ensheathing perineurium and endoneurial microvessels, and is the key structure that
PMID 23243216

Tumor Necrosis Factor-α Enhances CCL2 and ICAM-1 Expression in Peripheral Nerve Microvascular Endoneurial Endothelial Cells.

Langert KA, Von Zee CL, Stubbs Jr EB.AbstractRecruitment and trafficking of autoreactive leukocytes across the blood-nerve barrier is an early pathological insult in Guillain-Barré syndrome (GBS), an aggressive autoimmune disorder of the PNS. Whereas the etiology and pathogenesis of GBS remain unclear, pro-inflammatory cytokines, including tumor necrosis factor-α (TNF-α), are reported to be elevated early in the course of GBS and may initiate nerve injury by activating the blood-nerve barrier (BNB). Previously, we reported that disrupting leukocyte trafficking in vivo therapeutically attenuates the course of an established animal model of GBS. Here, peripheral nerve microvascular endoneurial endothelial cells (PNMECs) that form the BNB were harvested from rat sciatic nerves, immortalized by SV40 large T antigen transduction, and subsequently challenged with TNF-α. Relative changes in chemokine (C-C motif) ligand 2 (CCL2) and intercellular adhesion molecule (ICAM)-1 expression were determined. We report that TNF-α elicits marked dose- and time-dependent increases in CCL2 and ICAM-1 mRNA and protein content and promotes secretion of functional CCL2 from immortalized and primary PNMEC cultures. TNF-α mediated secretion of CCL2 promotes, in vitro, the transendothelial migration of CCR2-expressing THP-1 monocytes. Increased CCL2 and ICAM-1 expression in response to TNF-α may facilitate recruitment and trafficking of autoreactive leukocytes across the blood-nerve barrier in autoimmune disorders, including GBS.
ASN neuro.ASN Neuro.2013 Jan 7. [Epub ahead of print]
Recruitment and trafficking of autoreactive leukocytes across the blood-nerve barrier is an early pathological insult in Guillain-Barré syndrome (GBS), an aggressive autoimmune disorder of the PNS. Whereas the etiology and pathogenesis of GBS remain unclear, pro-inflammatory cytokines, including tu
PMID 23293927

Japanese Journal

The role of sulfoglucuronosyl glycosphingolipids in the pathogenesis of monoclonal IgM paraproteinemia and peripheral neuropathy

ARIGA Toshio
Proceedings of the Japan Academy, Series B 87(7), 386-404, 2011
… In an in vitro study, destruction or malfunction of the blood-nerve barrier (BNB) was found, resulting in the leakage of circulating antibodies into the PNS parenchyma, which may be considered as the initial key step for development of disease.<BR><BR>(Communicated by Kunihiko SUZUKI, M.J.A.) …
NAID 130001066426

ギラン・バレー症候群の臨床経過と回復パターン : 電気生理学的検討

川上治,安藤哲朗,杉浦真,加藤博子,土方靖浩,坪井崇
末梢神経 = Peripheral nerve 19(2), 346-349, 2008-12-01
NAID 10025625570

Related Pictures

★リンクテーブル★

リンク元	「血液神経関門」
関連記事	「B」「BN」

「血液神経関門」

Bulgarian National Bank
	Bulgarian National Bank; Българска народна банка (Bulgarian); Balgarska narodna banka
Bulgarian National Bank (BNB) headquarters in Sofia	Coat of Arms of the Bulgarian National Bank
Headquarters	Sofia
Established	25 January 1879
Governor	Ivan Iskrov
Central bank of	Bulgaria
Currency	Bulgarian lev
ISO 4217 Code	BGN
Website	bnb.bg