WordNet

second in order of importance; "the candidate, considered a beta male, was perceived to be unable to lead his party to victory"
the 2nd letter of the Greek alphabet
preliminary or testing stage of a software or hardware product; "a beta version"; "beta software"
the 1st letter of the Roman alphabet (同)a
the blood group whose red cells carry the A antigen (同)type_A, group A

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Sporadic inclusion-body myositis: A degenerative muscle disease associated with aging, impaired muscle protein homeostasis and abnormal mitophagy.

Askanas V1, Engel WK2, Nogalska A2.
Biochimica et biophysica acta.Biochim Biophys Acta.2015 Apr;1852(4):633-43. doi: 10.1016/j.bbadis.2014.09.005. Epub 2014 Sep 18.
Sporadic inclusion-body myositis (s-IBM) is the most common degenerative muscle disease in which aging appears to be a key risk factor. In this review we focus on several cellular molecular mechanisms responsible for multiprotein aggregation and accumulations within s-IBM muscle fibers, and their po
PMID 25241263

Differential Effects of Palmitoylethanolamide against Amyloid-β Induced Toxicity in Cortical Neuronal and Astrocytic Primary Cultures from Wild-Type and 3xTg-AD Mice.

Tomasini MC1, Borelli AC2, Beggiato S1, Ferraro L3, Cassano T4, Tanganelli S5, Antonelli T5.
Journal of Alzheimer's disease : JAD.J Alzheimers Dis.2015 Mar 12. [Epub ahead of print]
BACKGROUND: Considering the heterogeneity of pathological changes occurring in Alzheimer's disease (AD), a therapeutic approach aimed both to neuroprotection and to neuroinflammation reduction may prove effective. Palmitoylethanolamide (PEA) has attracted attention for its anti-inflammatory/neuropro
PMID 25765918

Neuronal amyloid-β accumulation within cholinergic basal forebrain in ageing and Alzheimer's disease.

Baker-Nigh A1, Vahedi S1, Davis EG1, Weintraub S1, Bigio EH1, Klein WL2, Geula C3.
Brain : a journal of neurology.Brain.2015 Mar 1. pii: awv024. [Epub ahead of print]
The mechanisms that contribute to selective vulnerability of the magnocellular basal forebrain cholinergic neurons in neurodegenerative diseases, such as Alzheimer's disease, are not fully understood. Because age is the primary risk factor for Alzheimer's disease, mechanisms of interest must include
PMID 25732182

Amyloid Beta42 Fibril Mimic Peptides Inhibit the Amyloid Beta42 Fibrillation by Binding to the Prefibrillar Soluble Oligomers

TANAKA Koichi,NISHIMURA Masaaki,TAKIGUCHI Sho,YAMAGUCHI Makoto,KITAMOTO Sho,HASHIGUCHI Shuhei,ITO Yuji,SUGIMURA Kazuhisa
Peptide science : proceedings of the ... Japanese Peptide Symposium 2008, 67-70, 2009-03-01
NAID 10027165961

Amyloid Beta42 Fibril Mimic Peptides Inhibit the Amyloid Beta42 Fibrillation by Binding to Prefibrillar Oligomers

TANAKA Koichi,YOSHIHARA Tomoki,TAKIGUCHI Sho,KUBA Sayaka,HASHIGUCHI Shuhei,ITO Yuji,SUGIMURA Kazuhisa
Peptide science : proceedings of the ... Japanese Peptide Symposium 2007, 83-86, 2008-03-01
NAID 10021906650