WordNet

a light soft silver-white metallic element of the alkali metal group; oxidizes rapidly in air and reacts violently with water; is abundant in nature in combined forms occurring in sea water and in carnallite and kainite and sylvite (同)K, atomic number 19
a nonmetallic univalent element that is normally a colorless and odorless highly flammable diatomic gas; the simplest and lightest and most abundant element in the universe (同)H, atomic number 1

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1. Chapter 12B：腎臓のカリウム排泄 chapter 12b renal potassium excretion
2. Chapter 11B：腎臓の水排泄調節 chapter 11b regulation of renal hydrogen excretion
3. 腎尿細管性アシドーシスの病態生理およびカリウムバランスへの影響 pathophysiology of renal tubular acidosis and the effect on potassium balance
4. Chapter 11A：腎臓での水排泄 chapter 11a renal hydrogen excretion
5. 赤血球水分量の制御 control of red blood cell hydration

Hydrogen sulfide enhances salt tolerance through nitric oxide-mediated maintenance of ion homeostasis in barley seedling roots.

Chen J1, Wang WH2, Wu FH3, He EM2, Liu X4, Shangguan ZP5, Zheng HL4.
Scientific reports.Sci Rep.2015 Jul 27;5:12516. doi: 10.1038/srep12516.
Hydrogen sulfide (H2S) and nitric oxide (NO) are emerging as messenger molecules involved in the modulation of plant physiological processes. Here, we investigated a signalling network involving H2S and NO in salt tolerance pathway of barley. NaHS, a donor of H2S, at a low concentration of either 50
PMID 26213372

Furosemide-induced urinary acidification is caused by pronounced H+ secretion in the thick ascending limb.

de Bruijn PI, Larsen CK, Frische S, Himmerkus N, Praetorius HA, Bleich M, Leipziger J.
American journal of physiology. Renal physiology.Am J Physiol Renal Physiol.2015 Jul 15;309(2):F146-53.
The loop diuretic furosemide inhibits NaCl reabsorption in the thick ascending limb (TAL). In addition, furosemide acidifies the urine, which is traditionally explained by increased Na+ loading to the distal tubule causing an activation of H+ secretion via H+-ATPase in α-intercalated cells. The ina
PMID 25995110

Tubuloglomerular and connecting tubuloglomerular feedback during inhibition of various Na transporters in the nephron.

Wang H1, D'Ambrosio MA1, Ren Y1, Monu SR1, Leung P1, Kutskill K1, Garvin JL2, Janic B1, Peterson EL3, Carretero OA4.
American journal of physiology. Renal physiology.Am J Physiol Renal Physiol.2015 May 1;308(9):F1026-31. doi: 10.1152/ajprenal.00605.2014. Epub 2015 Feb 25.
Afferent (Af-Art) and efferent arterioles resistance regulate glomerular capillary pressure. The nephron regulates Af-Art resistance via: 1) vasoconstrictor tubuloglomerular feedback (TGF), initiated in the macula densa via Na-K-2Cl cotransporters (NKCC2) and 2) vasodilator connecting tubuloglomerul
PMID 25715987