WordNet

a disorder (usually of unknown origin) of the heart muscle (myocardium) (同)myocardiopathy

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1. 成人におけるショックの定義、症状、病因、および病態生理 definition classification etiology and pathophysiology of shock in adults
2. 心不全および心筋症における心室性不整脈の病因 pathogenesis of ventricular arrhythmias in heart failure and cardiomyopathy
3. 小児におけるQRS幅の広い頻脈の原因 causes of wide qrs complex tachycardia in children
4. 孤立性左室心筋緻密化障害 isolated left ventricular noncompaction
5. 小児における突然の心停止および突然死 sudden cardiac arrest and death in children

Global metabolomic analysis of heart tissue in a hamster model for dilated cardiomyopathy.

Maekawa K, Hirayama A, Iwata Y, Tajima Y, Nishimaki-Mogami T, Sugawara S, Ueno N, Abe H, Ishikawa M, Murayama M, Matsuzawa Y, Nakanishi H, Ikeda K, Arita M, Taguchi R, Minamino N, Wakabayashi S, Soga T, Saito Y.SourceProject Team for Disease Metabolomics, National Institute of Health Sciences, Tokyo 158-8501, Japan.
Journal of molecular and cellular cardiology.J Mol Cell Cardiol.2013 Jun;59:76-85. doi: 10.1016/j.yjmcc.2013.02.008. Epub 2013 Feb 20.
Dilated cardiomyopathy (DCM), a common cause of heart failure, is characterized by cardiac dilation and reduced left ventricular ejection fraction, but the underlying mechanisms remain unclear. To investigate the mechanistic basis, we performed global metabolomic analysis of myocardial tissues from
PMID 23454301

Ahuja P, Wanagat J, Wang Z, Wang Y, Liem DA, Ping P, Antoshechkin IA, Margulies KB, Maclellan WR.SourceDepartment of Anesthesiology, Division of Molecular Medicine, David Geffen School of Medicine at UCLA, BH-569 CHS, BOX 957115, Los Angeles, CA 90095. Pahuja@mednet.ucla.edu.
Circulation.Circulation.2013 May 14;127(19):1957-67. doi: 10.1161/CIRCULATIONAHA.112.001219. Epub 2013 Apr 15.
BACKGROUND: Mitochondria are key players in the development and progression of heart failure (HF). Mitochondrial (mt) dysfunction leads to diminished energy production and increased cell death contributing to the progression of left ventricular failure. The fundamental mechanisms that underlie mt dy
PMID 23589024

Activated apoptotic and anti-survival effects on rat hearts with fructose induced metabolic syndrome.

Cheng SM, Cheng YJ, Wu LY, Kuo CH, Lee YS, Wu MC, Huang CY, Ting H, Lee SD.SourceDepartment of Psychology, Asia University, Taichung, Taiwan.
Cell biochemistry and function.Cell Biochem Funct.2013 May 9. doi: 10.1002/cbf.2982. [Epub ahead of print]
Consumption of fructose has been linked to the development of metabolic syndrome, whereas the cardiomyopathic changes and cardiac apoptosis of dietary high-fructose intake have not yet been clarified. The purpose of this study was to evaluate the effects of high-fructose on cardiac apoptotic and sur
PMID 23657904

21世紀に向けた第二世代のアフェレシス療法について : Molecular and Cellular Surgery(<特集>腎疾患とアフェレシス-最近の知見-)

能勢之彦,太田和秀,高場準二,岡久稔也,宮本弘志,杉田洋一,本村禎
日本アフェレシス学会雑誌 30(2), 147-155, 2011-05-31
… Several examples of treating malignant tumors by apheresis together with treatments of end-stage cardiomyopathic patients (BIC MAC) and diabetic renal complication patients (CRARA) were described in this paper. …
NAID 110008661255

Miyamoto Hiroshi,Nosé Yukihiko
ANTI-AGING MEDICINE 7(9), 100-106, 2010
… The new technologies included in this paper were the cryoaggregate filtration to remove pathological globulin molecules for the treatment of cardiomyopathic diseases, cryoreactive albumin removal apheresis (CRARA) for the treatment and for the prevention of diabetic complications of diabetic patients by removing pathological albumin and bioincompatible apheresis system for cancer treatment. …
NAID 130000428309

Beneficial Effects of Growth Hormone-Releasing Peptide on Myocardial Oxidative Stress and Left Ventricular Dysfunction in Dilated Cardiomyopathic Hamsters

KATO Yosuke,IWASE Mitsunori,ICHIHARA Sahoko,KANAZAWA Hiroaki,HASHIMOTO Katsunori,NODA Akiko,NAGATA Kohzo,KOIKE Yasuo,YOKOTA Mitsuhiro
Circulation journal : official journal of the Japanese Circulation Society 74(1), 163-170, 2009-12-20
… Growth hormone-releasing peptide (GHRP) may act directly on the myocardium and improve left ventricular (LV) function, suggesting a potential new approach to the treatment of cardiomyopathic hearts. … Dilated cardiomyopathic TO-2 hamsters were injected with GHRP-2 (1 mg/kg) or saline from 6 to 12 weeks of age. … GHRP-2 can suppress the increase in the level of myocardial oxidative stress, leading to attenuation of progressive LV remodeling and dysfunction in dilated cardiomyopathic hamsters. …
NAID 10025941641