関: trimethoprim-sulfamethoxazole

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1. トリメトプリムスルファメトキサゾール：概要 trimethoprim sulfamethoxazole an overview
2. フルオロキノロン類、ドキシサイクリン、およびトリメトプリムスルファメトキサゾールに対する肺炎レンサ球菌の耐性 resistance of streptococcus pneumoniae to the fluoroquinolones doxycycline and trimethoprim sulfamethoxazole
3. 非HIV感染患者におけるニューモシスチス肺炎の治療および予防 treatment and prevention of pneumocystis pneumonia in non hiv infected patients
4. Drug-induced thrombotic microangiopathy
5. 女性における急性単純性膀胱炎および腎盂腎炎 acute uncomplicated cystitis and pyelonephritis in women

English Journal

Combination of micelle collapse and field-amplified sample stacking in capillary electrophoresis for determination of trimethoprim and sulfamethoxazole in animal-originated foodstuffs.

Liu L1, Wan Q2, Xu X2, Duan S3, Yang C4.
Food chemistry.Food Chem.2017 Mar 15;219:7-12. doi: 10.1016/j.foodchem.2016.09.118. Epub 2016 Sep 19.
An on-line preconcentration method combining micelle to solvent stacking (MSS) with field-amplified sample stacking (FASS) was employed for the analysis of trimethoprim (TMP) and sulfamethoxazole (SMZ) by capillary zone electrophoresis (CZE). The optimized experimental conditions were as followings:
PMID 27765261

Disseminated nocardiosis caused by Nocardia otitidiscaviarum in an immunocompetent host: A case report and literature review.

Jiang Y1, Huang A1, Fang Q1.
Experimental and therapeutic medicine.Exp Ther Med.2016 Nov;12(5):3339-3346. Epub 2016 Sep 28.
The aim of the present study was to summarize the clinical characteristics of nocardiosis caused by Nocardia otitidiscaviarum in order to improve the knowledge of nocardiosis. A case of dissemination nocardiosis caused by N. otitidiscaviarum in an immunocompetent host is reported and the associated
PMID 27882160

Bioactivation of Trimethoprim to Protein-Reactive Metabolites in Human Liver Microsomes.

Goldman JL1, Koen YM2, Rogers SA2, Li K2, Leeder JS2, Hanzlik RP2.
Drug metabolism and disposition: the biological fate of chemicals.Drug Metab Dispos.2016 Oct;44(10):1603-7. doi: 10.1124/dmd.116.072041. Epub 2016 Jul 25.
The formation of drug-protein adducts via metabolic activation and covalent binding may stimulate an immune response or may result in direct cell toxicity. Protein covalent binding is a potentially pivotal step in the development of idiosyncratic adverse drug reactions (IADRs). Trimethoprim (TMP)-su
PMID 27457783

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