- 関
- interleukin-17 receptor
WordNet
- the 9th letter of the Roman alphabet (同)i
- a cellular structure that is postulated to exist in order to mediate between a chemical agent that acts on nervous tissue and the physiological response
PrepTutorEJDIC
- 『私は』私が
- iodineの化学記号
- =sense organ / 受信装置
- Illinois
UpToDate Contents
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English Journal
- Hypoxia and hypoxia-inducible factor-1α provoke toll-like receptor signalling-induced inflammation in rheumatoid arthritis.
- Hu F1, Mu R, Zhu J, Shi L, Li Y, Liu X, Shao W, Li G, Li M, Su Y, Cohen PL, Qiu X, Li Z.Author information 1Department of Rheumatology and Immunology, Peking University People's Hospital, , Beijing, China.AbstractOBJECTIVES: Hyperplasia of synovial fibroblasts, infiltration with lymphocytes and tissue hypoxia are major characteristics of rheumatoid arthritis (RA). Extensive data support a key role for toll-like receptors (TLRs) in RA. Little is known regarding the impact of hypoxia on TLR-induced inflammation in RA. The aim of this study was to reveal the effects of hypoxia and its regulator, hypoxia-inducible factor-1α (HIF-1α), on the inflammatory response of RA synovial fibroblasts (RASF) to TLR ligands.
- Annals of the rheumatic diseases.Ann Rheum Dis.2014 May 1;73(5):928-36. doi: 10.1136/annrheumdis-2012-202444. Epub 2013 May 3.
- OBJECTIVES: Hyperplasia of synovial fibroblasts, infiltration with lymphocytes and tissue hypoxia are major characteristics of rheumatoid arthritis (RA). Extensive data support a key role for toll-like receptors (TLRs) in RA. Little is known regarding the impact of hypoxia on TLR-induced inflammatio
- PMID 23644550
- The microbiome and regulation of mucosal immunity.
- McDermott AJ1, Huffnagle GB.Author information 1Division of Pulmonary and Critical Care Medicine, Department of Internal Medicine, University of Michigan Medical School, Ann Arbor, MI, USA; Department of Microbiology and Immunology, University of Michigan Medical School, Ann Arbor, MI, USA.AbstractThe gastrointestinal tract is a mucosal surface constantly exposed to foreign antigens and microbes, and is protected by a vast array of immunologically active structures and cells. Epithelial cells directly participate in immunological surveillance and direction of host responses in the gut and can express numerous pattern recognition receptors, including Toll-like receptor 5 (TLR5), TLR1, TLR2, TLR3, TLR9, and nucleotide oligomerization domain 2, as well as produce chemotactic factors for both myeloid and lymphoid cells following inflammatory stimulation. Within the epithelium and in the underlying lamina propria resides a population of innate lymphoid cells that, following stimulation, can become activated and produce effector cytokines and exert both protective and pathogenic roles during inflammation. Lamina propria dendritic cells play a large role in determining whether the response to a particular antigen will be inflammatory or anti-inflammatory. It is becoming clear that the composition and metabolic activity of the intestinal microbiome, as a whole community, exerts a profound influence on mucosal immune regulation. The microbiome produces short-chain fatty acids, polysaccharide A, α-galactosylceramide and tryptophan metabolites, which can induce interleukin-22, Reg3γ, IgA and interleukin-17 responses. However, much of what is known about microbiome-host immune interactions has come from the study of single bacterial members of the gastrointestinal microbiome and their impact on intestinal mucosal immunity. Additionally, evidence continues to accumulate that alterations of the intestinal microbiome can impact not only gastrointestinal immunity but also immune regulation at distal mucosal sites.
- Immunology.Immunology.2014 May;142(1):24-31. doi: 10.1111/imm.12231.
- The gastrointestinal tract is a mucosal surface constantly exposed to foreign antigens and microbes, and is protected by a vast array of immunologically active structures and cells. Epithelial cells directly participate in immunological surveillance and direction of host responses in the gut and can
- PMID 24329495
- Effect of bone marrow-derived CD11b(+)F4/80 (+) immature dendritic cells on the balance between pro-inflammatory and anti-inflammatory cytokines in DBA/1 mice with collagen-induced arthritis.
- Fu J1, Zhang L, Song S, Sheng K, Li Y, Li P, Song S, Wang Q, Chu J, Wei W.Author information 1Institute of Clinical Pharmacology, Anhui Medical University, Key Laboratory of Antiinflammatory and Immune Medicine (Anhui Medical University), Ministry of Education, 230032, Hefei, China.AbstractOBJECTIVE: To explore the effect of bone marrow-derived CD11b(+)F4/80(+) immature dendritic cells (BM CD11b(+)F4/80(+)iDC) on the balance between pro-inflammatory and anti-inflammatory cytokines in DBA/1 mice with collagen-induced arthritis (CIA).
- Inflammation research : official journal of the European Histamine Research Society ... [et al.].Inflamm Res.2014 May;63(5):357-67. doi: 10.1007/s00011-014-0707-7. Epub 2014 Jan 24.
- OBJECTIVE: To explore the effect of bone marrow-derived CD11b(+)F4/80(+) immature dendritic cells (BM CD11b(+)F4/80(+)iDC) on the balance between pro-inflammatory and anti-inflammatory cytokines in DBA/1 mice with collagen-induced arthritis (CIA).METHODS: BM CD11b(+)F4/80(+)iDC were induced with rmG
- PMID 24458308
Japanese Journal
- HMGB1Modulates the Treg/Th17 Ratio in Atherosclerotic Patients
- Coordinated effect of IL-17A and IL-27 on osteoclast differentiation of RANKL-stimulated RAW264.7 cells
- 7 RANKL刺激によるRAW264.7細胞の破骨細胞分化におけるIL-17AとIL-27の協調効果(第545回大阪歯科学会例会)
Related Links
- Yao Z, Spriggs MK, Derry JM, Strockbine L, Park LS, VandenBos T, Zappone JD, Painter SL, Armitage RJ (1997). "Molecular characterization of the human interleukin (IL)-17 receptor". Cytokine 9 (11): 794–800. doi:10.1006/cyto. 1997.0240.
★リンクテーブル★
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- 英
- interleukin-17 receptor、IL-17 receptor
- 関
- インターロイキン17レセプター、IL-17レセプター、IL-17受容体
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- 関
- IL-17 receptor
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- 英
- IL-17 receptor
- 関
- インターロイキン17受容体、IL-17レセプター
[★]
- 英
- IL-17 receptor
- 関
- インターロイキン17受容体、IL-17受容体
[★]
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