反射性筋緊張

WordNet

an automatic instinctive unlearned reaction to a stimulus (同)reflex response, reflex action, instinctive reflex, innate reflex, inborn reflex, unconditioned_reflex, physiological reaction
(of leaves) bent downward and outward more than 90 degrees

PrepTutorEJDIC

反射(刺激に対する無意識の反応) / 《複数形で》反射的な動き / (光などの)反射,反射光;映像 / 反射性の / (カメラが)レフ鋼の,反射型の
(重量単位の)『トン』 / (容積単位の)トン(物によって異なり,木材では40立方フィート,小麦では20立方フィート) / 《話》大量(の…),多数(の…)《+of+名》

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1. 反射性失神 reflex syncope
2. 成人における昏迷および昏睡 stupor and coma in adults
3. 幼児および小児における視覚の発達および視力評価 visual development and vision assessment in infants and children
4. 成人における亜急性および慢性咳嗽の評価 evaluation of subacute and chronic cough in adults
5. 成人における詳細な神経学的検査 the detailed neurologic examination in adults

English Journal

Role of duodenal mucosal nerve endings in the acid-induced duodenogastric sensorimotor reflex: effect of benzocaine in healthy humans.

Vanuytsel T1, Karamanolis G, Vos R, Van Oudenhove L, Farré R, Tack J.Author information 1Translational Research Center for Gastrointestinal Disorders (TARGID), University of Leuven, Leuven, Belgium.AbstractBACKGROUND: Duodenal acid exposure induces a duodenogastric reflex resulting in gastric relaxation, inhibition of antral motility, and sensitization of the proximal stomach to distension. Duodenal hypersensitivity to acid has been identified as a potential pathogenic mechanism in functional dyspepsia. The nature and localization of the duodenal acid-sensitive receptors are still elusive. We hypothesize that acid directly activates superficial afferent nerve endings in the duodenal mucosa, triggering the duodenogastric reflex.
Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.Neurogastroenterol Motil.2013 May;25(5):e353-61. doi: 10.1111/nmo.12126. Epub 2013 Apr 9.
BACKGROUND: Duodenal acid exposure induces a duodenogastric reflex resulting in gastric relaxation, inhibition of antral motility, and sensitization of the proximal stomach to distension. Duodenal hypersensitivity to acid has been identified as a potential pathogenic mechanism in functional dyspepsi
PMID 23565580

Stretch reflex spatial threshold measure discriminates between spasticity and rigidity.

Mullick AA1, Musampa NK, Feldman AG, Levin MF.Author information 1School of Physical and Occupational Therapy, McGill University, Canada.AbstractOBJECTIVE: Muscle spasticity following stroke has been shown to result from limitations in the range of regulation of the tonic reflex spatial threshold (ST), i.e., the joint angle at which the stretch reflex begins to act due to descending and segmental influences on motoneurons. The purpose of this study was to determine whether spasticity due to stroke and rigidity due to parkinsonism can be discriminated based on the ST measure.
Clinical neurophysiology : official journal of the International Federation of Clinical Neurophysiology.Clin Neurophysiol.2013 Apr;124(4):740-51. doi: 10.1016/j.clinph.2012.10.008. Epub 2012 Nov 10.
OBJECTIVE: Muscle spasticity following stroke has been shown to result from limitations in the range of regulation of the tonic reflex spatial threshold (ST), i.e., the joint angle at which the stretch reflex begins to act due to descending and segmental influences on motoneurons. The purpose of thi
PMID 23146713

Pathophysiology of spasticity in stroke.

Burke D1, Wissel J, Donnan GA.Author information 1Department of Neurology, Royal Prince Alfred Hospital and University of Sydney, Australia. david.burke@sydney.edu.auAbstractSpasticity is defined clinically by increased muscle tone and tendon jerk hyperreflexia in patients who are at rest. However, the excitability of spinal circuits changes during movement, and this definition provides no insight into the extent to which spasticity and associated motor disturbances cause disability. Only a few spinal circuits have been shown to underlie the abnormalities of patients at rest. Movement can be restrained by pathologically enhanced muscle tone, and there is defective control of the feedback to active motoneurons through virtually all spinal reflex pathways. Spasticity does not necessarily require treatment: in fact, some patients rely on the increased muscle tone to help support otherwise weak muscle contractions for stance and locomotion. In addition, much of the increase in muscle tone arises from changes in muscle and motor units, independent of reflex mechanisms. Managing a patient with impairment after a stroke requires therapy tailored to that particular patient because the mechanisms contributing to the disability experienced by one patient may differ from those affecting another.
Neurology.Neurology.2013 Jan 15;80(3 Suppl 2):S20-6. doi: 10.1212/WNL.0b013e31827624a7.
Spasticity is defined clinically by increased muscle tone and tendon jerk hyperreflexia in patients who are at rest. However, the excitability of spinal circuits changes during movement, and this definition provides no insight into the extent to which spasticity and associated motor disturbances cau
PMID 23319482