- 関
- vasomotion
WordNet
- relating to the nerves and muscles that cause the blood vessels to constrict or dilate
Wikipedia preview
出典(authority):フリー百科事典『ウィキペディア(Wikipedia)』「2017/05/28 05:37:53」(JST)
[Wiki en表示]
Vasomotor refers to actions upon a blood vessel which alter its diameter.[1][2] More specifically, it can refer to vasodilator action and vasoconstrictor action.
Contents
- 1 Control
- 1.1 Sympathetic innervation
- 1.2 Endothelium derived chemicals
- 2 Pathology
- 3 See also
- 4 References
Control
Sympathetic innervation
Sympathetic nerve fibers travel around the tunica media of the artery, secrete neurotransmitters such as norepinephrine into the extracellular fluid surrounding the smooth muscle (tunica media) from the terminal knob of the axon. The smooth muscle cell membranes have α and β-adrenergic receptors for these neurotransmitters. Activation of α-adrenergic receptors promotes vasoconstriction, while the activation of β-adrenergic receptors mediates the relaxation of muscle cells, resulting in vasodilation. Normally, α-adrenergic receptors predominates in smooth muscle of resistance vessels.[3]
Endothelium derived chemicals
Endothelin, and angiotensin are the vasocontrictors of smooth muscles while nitric oxide and prostacyclin are vasodilators of the smooth muscles.[3]
Pathology
Some vasoactive chemicals such as vasodilator acetylcholine are known for causing reduced/increased blood flow in the tumours by vasomotor changes. Inadequate blood supply to the tumour cells can cause the cells to be radio-resistant and resulted in reduced accessibility to chemotherapeutic agents.[4]
Injuries to nerves of the lower trunk of the brachial plexus (Klumpke's paralysis) and compression of median nerve at the flexor retinaculum of the hand (Carpal Tunnel Syndrome) can cause vasomotor changes at the areas innervated by the nerves. This area of the skin will become warmer because of vasodilation (loss of vasoconstriction).[5]
Depression of the vasomotor center of the brain can cause the loss of vasomotor tone of blood vessels, resulting in massive dilatation of veins. This will result in a condition called as neurogenic shock.[6]
See also
- Vasoconstriction
- Vasodilation
References
- ^ "Vasomotor" at Dorland's Medical Dictionary
- ^ WebMD (2009). "vasomotor". Webster's New World Medical Dictionary (3rd ed.). Houghton Mifflin Harcourt. p. 447. ISBN 978-0-544-18897-6.
- ^ a b Robert C. Ward et al. (2002). In Foundations for osteopathic medicine. Lippincott Williams & Wilkins. 2nd edition. p. 98. ISBN 978-0-7817-3497-4. Google Book Search. Retrieved on 5 December 2010.
- ^ Cater, D B; Adair, H M; Grove, C A (1966). "Effects of vasomotor drugs and 'mediators' of the inflammatory reaction upon the oxygen tension of tumours and tumour blood-flow". British Journal of Cancer. 20 (3): 504–16. doi:10.1038/bjc.1966.62. PMC 2007997 . PMID 4288476.
- ^ B.D Cahurasia (2010). Human Anatomy: Regional & Applied (Dissection & Clinical) 5e (in 3 Vols.) Vol. 1: Upper Limb & Thorax With CD. New Delhi: CBS Publishers & Distributors. pp. 59, 133, 296. ISBN 978-81-239-1863-1.
- ^ Guyton, Arthur C.; Hall, John Edward (2006). Textbook of Medical Physiology. Pennsylvania, United States: Elsevier Saunders. pp. 285, 1116. ISBN 0-7216-0240-1.
UpToDate Contents
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English Journal
- Commissural nucleus of the solitary tract regulates the antihypertensive effects elicited by moxonidine.
- Totola LT, Alves TB, Takakura AC, Ferreira-Neto HC, Antunes VR, Menani JV, Colombari E, Moreira TS.SourceDepartment of Physiology and Biophysics, Institute of Biomedical Science, University of São Paulo, São Paulo, SP, Brazil.
- Neuroscience.Neuroscience.2013 Oct 10;250:80-91. doi: 10.1016/j.neuroscience.2013.06.065. Epub 2013 Jul 10.
- The rostral ventrolateral medulla (RVLM) contains the presympathetic neurons involved in cardiovascular regulation that has been implicated as one of the most important central sites for the antihypertensive action of moxonidine (an α2-adrenergic and imidazoline agonist). Here, we sought to evaluat
- PMID 23850502
- Menopausal Hormone Therapy and Health Outcomes During the Intervention and Extended Poststopping Phases of the Women's Health Initiative Randomized Trials.
- Manson JE, Chlebowski RT, Stefanick ML, Aragaki AK, Rossouw JE, Prentice RL, Anderson G, Howard BV, Thomson CA, Lacroix AZ, Wactawski-Wende J, Jackson RD, Limacher M, Margolis KL, Wassertheil-Smoller S, Beresford SA, Cauley JA, Eaton CB, Gass M, Hsia J, Johnson KC, Kooperberg C, Kuller LH, Lewis CE, Liu S, Martin LW, Ockene JK, O'Sullivan MJ, Powell LH, Simon MS, Van Horn L, Vitolins MZ, Wallace RB.SourceBrigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts.
- JAMA : the journal of the American Medical Association.JAMA.2013 Oct 2;310(13):1353-1368.
- IMPORTANCE Menopausal hormone therapy continues in clinical use but questions remain regarding its risks and benefits for chronic disease prevention. OBJECTIVE To report a comprehensive, integrated overview of findings from the 2 Women's Health Initiative (WHI) hormone therapy trials with extended p
- PMID 24084921
- Short-term administration of progesterone and estradiol independently alter carotid-vasomotor, but not carotid-cardiac, baroreflex function in young women.
- Brunt VE, Miner JA, Kaplan PF, Halliwill JR, Strycker LA, Minson CT.SourceDepartment of Human Physiology, University of Oregon, Eugene, Oregon;
- American journal of physiology. Heart and circulatory physiology.Am J Physiol Heart Circ Physiol.2013 Oct;305(7):H1041-H1049. Epub 2013 Jul 19.
- The individual effects of estrogen and progesterone on baroreflex function remain poorly understood. We sought to determine how estradiol (E2) and progesterone (P4) independently alter the carotid-cardiac and carotid-vasomotor baroreflexes in young women by using a hormone suppression and exogenous
- PMID 23873800
Japanese Journal
- Peripheral circulation indicators in veteran trail runners
- 急性の電気刺激が血流依存性の血管調節機能に与える影響
- 複合性局所疼痛症候群Ⅰ型に対し 交代浴の効果が認められた症例
Related Pictures
★リンクテーブル★
[★]
- 英
- vasomotor rhinitis
- ラ
- rhinitis vasomotorica
- 同
- 本態性鼻炎
- 関
- 血管運動神経性鼻炎、アレルギー性鼻炎、好酸球増多性鼻炎
[show details]
概念
- 鼻炎の一種。
- くしゃみ、水様性鼻漏、鼻閉を主徴候とするが、皮内テスト、鼻粘膜誘発テストで特定の発症抗原が同定できず、鼻汁の好酸球増多はみられず、また血液検査でも特異的IgEの証明ができないもの。
- 自律神経の異常や鼻粘膜過敏性が原因と考えられている。
症状
治療
- 薬物療法:抗コリン作用のある抗ヒスタミン薬(第一世代抗ヒスタミン薬)、局所抗コリン薬
[★]
- 英
- vasomotor
- 関
- 血管運動
[★]
- 関
- vasomotor
[★]
血管運動神経不全麻痺
[★]
血管運動神経性充血
[★]
血管運動神経性失神