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1. 糸球体足細胞のバイオロジー biology of glomerular podocytes
2. 僧帽弁逸脱症候群 mitral valve prolapse syndrome
3. ハーブ含有アリストロキン酸（AA）誘発性腎症 nephropathy induced by aristolochic acid aa containing herbs
4. 大腸癌の分子遺伝学 molecular genetics of colorectal cancer
5. 非淡明細胞型腎癌に対する治療アプローチ the treatment approach to non clear cell renal carcinoma

Wiest DL1.
Methods in molecular biology (Clifton, N.J.).Methods Mol Biol.2016;1323:23-32. doi: 10.1007/978-1-4939-2809-5_2.
While the functions of αβ T cells in host resistance to pathogen infection are understood in far more detail than those of γδ lineage T cells, γδ T cells perform critical, essential functions during immune responses that cannot be compensated by αβ T cells. Accordingly, it is essential to un
PMID 26294395

Alzheimer's as a Systems-Level Disease Involving the Interplay of Multiple Cellular Networks.

Castrillo JI1, Oliver SG.
Methods in molecular biology (Clifton, N.J.).Methods Mol Biol.2016;1303:3-48. doi: 10.1007/978-1-4939-2627-5_1.
Alzheimer's disease (AD), and many neurodegenerative disorders, are multifactorial in nature. They involve a combination of genomic, epigenomic, interactomic and environmental factors. Progress is being made, and these complex diseases are beginning to be understood as having their origin in altered
PMID 26235058

Subchronic phencyclidine treatment in adult mice increases GABAergic transmission and LTP threshold in the hippocampus.

Nomura T1, Oyamada Y2, Fernandes HB1, Remmers CL1, Xu J1, Meltzer HY3, Contractor A4.
Neuropharmacology.Neuropharmacology.2016 Jan;100:90-7. doi: 10.1016/j.neuropharm.2015.04.012. Epub 2015 Apr 30.
Repeated administration of non-competitive N-methyl-d-aspartate (NMDA) receptor antagonists such as phencyclidine (PCP) to rodents causes long-lasting deficits in cognition and memory, and has effects on behaviors that have been suggested to be models of the cognitive impairment associated with schi
PMID 25937215

Mice heterozygous for cathepsin D deficiency exhibit mania-related behavior and stress-induced depression.

Zhou R1, Lu Y1, Han Y1, Li X1, Lou H1, Zhu L1, Zhen X2, Duan S3.
Progress in neuro-psychopharmacology & biological psychiatry.Prog Neuropsychopharmacol Biol Psychiatry.2015 Dec 3;63:110-8. doi: 10.1016/j.pnpbp.2015.06.007. Epub 2015 Jun 16.
Mutations in cathepsin D (CTSD), an aspartic protease in the endosomal-lysosomal system, underlie congenital neuronal ceroid-lipofuscinosis (cNCL, also known as CLN10), a devastating neurodegenerative disease. CLN10 patients die within the first few days of life, and in the few patients who live int
PMID 26092248