WordNet

the organic process in which the substance of some differentiated structure that has been produced by the body undergoes lysis and assimilation (同)reabsorption
undergo resorption (同)resorb

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出典(authority):フリー百科事典『ウィキペディア（Wikipedia）』「2015/10/01 23:10:06」(JST)

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In physiology, reabsorption or tubular reabsorption is themethod of [contraception from the male tubule of the nephron into the peritubular capillariesऑठोफृपेटठृपेऋपैठपृपृडफेफृएडफृडेठृऑअःठपःपृऋडृफृठृअःr from the urine into the blood. It is termed "reabsorption" because the fluid and other substances that are reabsorbed from the bloodstream through the glomerulus into Bowman's capsule, which leads to the peritubular capillaries. This happens as a result of sodium transport from the lumen into the blood by the Na+/K+ ATPase in the basolateral membrane of the epithelial cells. Thus, the glomerular filtrate becomes more concentrated, which is one of the steps in forming urine. In this way, many useful solutes (primarily glucose and amino acids), salts and water that have passed in the proximal convoluted tubule through Bowman's capsule, return in the circulation. These solutes are reabsorbed isotonically, in that the osmotic potential of the fluid leaving the proximal convoluted tubule is the same as that of the initial glomerular filtrate. However, glucose, amino acids, inorganic phosphate, and some other solutes are reabsorbed via secondary active transport through cotransport channels driven by the sodium gradient out of the nephron.

Renin Angiotensin System:

1. The kidneys sense low blood pressure.

2. Release renin into the blood.

3. Renin causes production of Angiotensin I.

4. Angiotensin Converting Enzyme (ACE) converts Angiotensin I to Angiotensin II.

5. Angiotensin II stimulates the release of Aldosterone, ADH, and thirst.

6. Aldosterone causes kidneys to reabsorb sodium. ADH increases the uptake of water.

7. Water follows sodium.

8. As blood volume increases, pressure also increases.

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English Journal

Role of Carbon Monoxide in Kidney Function: Is a little Carbon Monoxide Good for the Kidney?

Csongradi E, Juncos LA, Drummond HA, Vera T, Stec DE.SourceDepartment of Physiology & Biophysics, University of Mississippi Medical Center Jackson, MS 39216. dstec@physiology.umsmed.edu.
Current pharmaceutical biotechnology.Curr Pharm Biotechnol.2012 Sep 1. [Epub ahead of print]
Carbon monoxide (CO) is an endogenously produced gas resulting from the degradation of heme by heme oxygense or from fatty acid oxidation. Heme oxygenase (HO) enzymes are constitutively expressed in the kidney (HO-2) and HO-1 is induced in the kidney in response to several physiological and patholog
PMID 22201605

Renal tubular dysfunction during long-term adefovir or tenofovir therapy in chronic hepatitis B.

Gara N, Zhao X, Collins MT, Chong WH, Kleiner DE, Jake Liang T, Ghany MG, Hoofnagle JH.SourceLiver Diseases Branch, National Institute of Diabetes and Digestive and Kidney Diseases, Bethesda, MD, USA.
Alimentary pharmacology & therapeutics.Aliment Pharmacol Ther.2012 Jun;35(11):1317-1325. doi: 10.1111/j.1365-2036.2012.05093.x. Epub 2012 Apr 16.
BACKGROUND: Adefovir and tenofovir are nucleotide analogues used as long-term therapy of chronic hepatitis B. Side effects are few, but prolonged and high-dose therapy has been associated with proximal renal tubular dysfunction (RTD).AIM: To assess the incidence of RTD during long-term nucleotide th
PMID 22506503