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an inactive form of plasmin that occurs in plasma and is converted to plasmin by organic solvents

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1. 止血の概要 overview of hemostasis
2. 静脈血栓症の原因の概要 overview of the causes of venous thrombosis
3. 血栓溶解異常による血栓性および出血性疾患 thrombotic and hemorrhagic disorders due to abnormal fibrinolysis
4. 血管内皮機能および血栓溶解療法の基本的メカニズム vascular endothelial function and fundamental mechanisms of fibrinolysis thrombolysis
5. 線溶マーカーおよび心血管リスク fibrinolytic markers and cardiovascular risk

Candida albicans is a crafty microbe that deceives its host by using complement regulators and proteases.

Riesbeck K.
The Journal of infectious diseases.J Infect Dis.2013 Feb 15;207(4):550-2. doi: 10.1093/infdis/jis722. Epub 2012 Nov 29.
PMID 23204177

Glycerol-3-phosphate dehydrogenase 2 is a novel factor H-, factor H-like protein 1-, and plasminogen-binding surface protein of Candida albicans.

Luo S, Hoffmann R, Skerka C, Zipfel PF.SourceDepartment of Infection Biology, Leibniz Institute for Natural Product Research and Infection Biology-Hans Knöll Institute, Germany.
The Journal of infectious diseases.J Infect Dis.2013 Feb 15;207(4):594-603. doi: 10.1093/infdis/jis718. Epub 2012 Nov 29.
Candida albicans uses human complement regulators such as factor H and factor H-like protein 1 (FHL-1) for immune evasion. To define the whole panel of fungal complement-evasion molecules, C. albicans cell extract was absorbed to a factor H-coupled matrix. One 52-kDa protein was eluted and identifie
PMID 23204165

Dihydrolipoamide dehydrogenase of Pseudomonas aeruginosa is a surface-exposed immune evasion protein that binds three members of the factor H family and plasminogen.

Hallström T, Mörgelin M, Barthel D, Raguse M, Kunert A, Hoffmann R, Skerka C, Zipfel PF.SourceDepartment of Infection Biology, Leibniz Institute for Natural Product Research and Infection Biology, 07745 Jena, Germany.
Journal of immunology (Baltimore, Md. : 1950).J Immunol.2012 Nov 15;189(10):4939-50. doi: 10.4049/jimmunol.1200386. Epub 2012 Oct 15.
The opportunistic human pathogen Pseudomonas aeruginosa causes a wide range of diseases. To cross host innate immune barriers, P. aeruginosa has developed efficient strategies to escape host complement attack. In this study, we identify the 57-kDa dihydrolipoamide dehydrogenase (Lpd) as a surface-ex
PMID 23071278