WordNet

any of a large group of nitrogenous organic compounds that are essential constituents of living cells; consist of polymers of amino acids; essential in the diet of animals for growth and for repair of tissues; can be obtained from meat and eggs and milk and legumes; "a diet high in protein"
causing motion or action or change (同)actuating

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1. 慢性骨髄性白血病の細胞生物学および分子生物学 cellular and molecular biology of chronic myeloid leukemia
2. 幼児の食物蛋白誘発性直腸炎/大腸炎、腸疾患、および腸炎 food protein induced proctitis colitis enteropathy and enterocolitis of infancy
3. 活性化プロテインＣ抵抗性および第V因子ライデン activated protein c resistance and factor v leiden
4. ペプチドホルモンのシグナル伝達および調節 peptide hormone signal transduction and regulation
5. 肥大型心筋症の遺伝学 genetics of hypertrophic cardiomyopathy

MicroRNAs 206 and 21 cooperate to promote RAS-extracellular signal-regulated kinase signaling by suppressing the translation of RASA1 and SPRED1.

Sharma SB1, Lin CC2, Farrugia MK1, McLaughlin SL3, Ellis EJ3, Brundage KM3, Salkeni MA4, Ruppert JM5.
Molecular and cellular biology.Mol Cell Biol.2014 Nov 15;34(22):4143-64. doi: 10.1128/MCB.00480-14. Epub 2014 Sep 8.
Despite the low prevalence of activating point mutation of RAS or RAF genes, the RAS-extracellular signal-regulated kinase (ERK) pathway is implicated in breast cancer pathogenesis. Indeed, in triple-negative breast cancer (TNBC), there is recurrent genetic alteration of pathway components. Using sh
PMID 25202123

Inhibition of cell migration and invasion mediated by the TAT-RasGAP317-326 peptide requires the DLC1 tumor suppressor.

Barras D1, Lorusso G2, Rüegg C2, Widmann C1.
Oncogene.Oncogene.2014 Oct 30;33(44):5163-72. doi: 10.1038/onc.2013.465. Epub 2013 Nov 11.
TAT-RasGAP(317-326), a peptide corresponding to the 317-326 sequence of p120 RasGAP coupled with a cell-permeable TAT-derived peptide, sensitizes the death response of various tumor cells to several anticancer treatments. We now report that this peptide is also able to increase cell adherence, preve
PMID 24213569

Activation of RhoA-ROCK-BMP signaling reprograms adult human corneal endothelial cells.

Zhu YT1, Li F2, Han B3, Tighe S1, Zhang S1, Chen SY1, Liu X3, Tseng SC4.
The Journal of cell biology.J Cell Biol.2014 Sep 15;206(6):799-811. doi: 10.1083/jcb.201404032. Epub 2014 Sep 8.
Currently there are limited treatment options for corneal blindness caused by dysfunctional corneal endothelial cells. The primary treatment involves transplantation of healthy donor human corneal endothelial cells, but a global shortage of donor corneas necessitates other options. Conventional tiss
PMID 25202030