WordNet

of or relating to neurons; "neural network" (同)neuronal, neuronic
of or relating to or used in or practicing neurology; "neurological evidence" (同)neurologic
arising in or stimulated by nerve tissues

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1. 急性痙攣発作に関連するMRIの変化 magnetic resonance imaging changes related to acute seizure activity
2. 肝性脳症の病因 pathogenesis of hepatic encephalopathy

Interleukin-6 Attenuates Serotonin 2A Receptor Signaling by Activating the JAK-STAT Pathway.

Donegan JJ1, Patton MS1, Chavera TS1, Berg KA1, Morilak DA1, Girotti M2.
Molecular pharmacology.Mol Pharmacol.2015 Mar;87(3):492-500. doi: 10.1124/mol.114.096289. Epub 2014 Dec 30.
The serotonin 2A (5-HT2A) receptor and the proinflammatory cytokine, interleukin-6 (IL-6), have both been implicated in psychiatric disorders. Previously, we demonstrated that these molecules both facilitate cognitive flexibility, a prefrontal cortex-mediated executive function impaired in multiple
PMID 25549668

Identification of Non-Viable Genes Affecting Touch Sensitivity in Caenorhabditis elegans Using Neuronally-Enhanced Feeding RNAi.

Chen X1, Diaz Cuadros M1, Chalfie M2.
G3 (Bethesda, Md.).G3 (Bethesda).2015 Jan 9. pii: g3.114.015776. doi: 10.1534/g3.114.015776. [Epub ahead of print]
Caenorhabditis elegans senses gentle touch along the body using six touch receptor neurons (TRNs). Although genetic screens and microarray analyses have identified several genes needed for touch sensitivity, these methods miss pleiotropic genes that are essential for the viability, movement, or fert
PMID 25575561

Independent role for presynaptic FMRP revealed by an FMR1 missense mutation associated with intellectual disability and seizures.

Myrick LK1, Deng PY2, Hashimoto H3, Oh YM4, Cho Y4, Poidevin MJ1, Suhl JA1, Visootsak J5, Cavalli V4, Jin P1, Cheng X3, Warren ST6, Klyachko VA7.
Proceedings of the National Academy of Sciences of the United States of America.Proc Natl Acad Sci U S A.2015 Jan 5. pii: 201423094. [Epub ahead of print]
Fragile X syndrome (FXS) results in intellectual disability (ID) most often caused by silencing of the fragile X mental retardation 1 (FMR1) gene. The resulting absence of fragile X mental retardation protein 1 (FMRP) leads to both pre- and postsynaptic defects, yet whether the pre- and postsynaptic
PMID 25561520