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1. 緊急避妊剤 emergency contraception
2. 月経抑制のためのホルモン避妊法 hormonal contraception for suppression of menstruation
3. エストロゲン・プロゲスチン避妊剤の使用に関する概要 overview of the use of estrogen progestin contraceptives
4. エストロゲン関連片頭痛 estrogen associated migraine
5. 経皮吸収避妊パッチ transdermal contraceptive patch

Phosphorylation of Human Metapneumovirus M2-1 Protein Upregulates Viral Replication and Pathogenesis.

Cai H1, Zhang Y1, Lu M2, Liang X1, Jennings R1, Niewiesk S1, Li J3.
Journal of virology.J Virol.2016 Jul 27;90(16):7323-38. doi: 10.1128/JVI.00755-16. Print 2016 Aug 15.
Human metapneumovirus (hMPV) is a major causative agent of upper- and lower-respiratory-tract infections in infants, the elderly, and immunocompromised individuals worldwide. Like all pneumoviruses, hMPV encodes the zinc binding protein M2-1, which plays important regulatory roles in RNA synthesis.
PMID 27252537

Zinc binding activity of human metapneumovirus M2-1 protein is indispensable for viral replication and pathogenesis in vivo.

Cai H1, Zhang Y1, Ma Y1, Sun J1, Liang X1, Li J2.
Journal of virology.J Virol.2015 Jun;89(12):6391-405. doi: 10.1128/JVI.03488-14. Epub 2015 Apr 8.
Human metapneumovirus (hMPV) is a member of the Pneumovirinae subfamily in the Paramyxoviridae family that causes respiratory tract infections in humans. Unlike members of the Paramyxovirinae subfamily, the polymerase complex of pneumoviruses requires an additional cofactor, the M2-1 protein, which
PMID 25855728

Deletion of the SH gene from avian metapneumovirus has a greater impact on virus production and immunogenicity in turkeys than deletion of the G gene or M2-2 open reading frame.

Ling R1, Sinkovic S, Toquin D, Guionie O, Eterradossi N, Easton AJ.
The Journal of general virology.J Gen Virol.2008 Feb;89(Pt 2):525-33. doi: 10.1099/vir.0.83309-0.
Subgroup A avian metapneumoviruses lacking either the SH or G gene or the M2-2 open reading frame were generated by using a reverse-genetics approach. The growth properties of these viruses were studied in vitro and in vivo in their natural host. Deletion of the SH gene alone resulted in the generat
PMID 18198384