WordNet

to a very great extent or degree; "the idea is so obvious"; "never been so happy"; "I love you so"; "my head aches so!"
(usually followed by `that'
in a manner that facilitates; "he observed the snakes so he could describe their behavior"; "he stooped down so he could pick up his hat"
in such a condition or manner, especially as expressed or implied; "Theyre happy and I hope they will remain so"; "so live your life that old age will bring no regrets"
in the same way; also; "I was offended and so was he"; "worked hard and so did she"
to a certain unspecified extent or degree; "I can only go so far with this student"; "can do only so much in a day"
any of a large group of nitrogenous organic compounds that are essential constituents of living cells; consist of polymers of amino acids; essential in the diet of animals for growth and for repair of tissues; can be obtained from meat and eggs and milk and legumes; "a diet high in protein"
a male human offspring; "their son became a famous judge"; "his boy is taller than he is" (同)boy
small fruit fly used by Thomas Hunt Morgan in studying basic mechanisms of inheritance (同)Drosophila_melanogaster

PrepTutorEJDIC

《様態の指示》『そういうふうに』,『そのように』・《補語に用いて》『そのようで』,そうで・《程度》『それほど』,そんなに・《強意として》《話》『非常に』,『とても』,たいへん・《前に述べたことの内容に対する同意を表して;「so+主語+[助]動詞」》『その通りで』,『本当に』,確かに,おっしゃるとおり・《肯定文を受けて;「so+[助]動詞+主語」の語順で》…『もまた』,『も同様に』・《as~, so…で》~と同じように…,(~のように)そのように・《目的を表して》…『するように』・《理由・結果を表して》『その結果』,それで・《文・節の頭に用いて》『それで』,だから・《驚き・賛成・嫌悪・疑いなどを表して》そうか,そうなんだ,どうかな,まさか・それでよし
=sol
《所有・所属》…『の』,…のものである,…に属する・《材料・要素》…『でできた』,から成る・《部分》…『の』[『中の』] ・《数量・単位・種類を表す名詞に付いて》…の・《原因・動機》…『で』,のために(because of) ・《主格関係》…『の』,による,によって・《目的格関係》…『を』,の・《同格関係》…『という』・《関係・関連》…『についての』[『の』],の点で・《抽象名詞などと共に》…の[性質をもつ] ・《『It is』+『形』+『of』+『名』+『to』 doの形で,ofの後の名詞を意味上の主語として》・《分離》…『から』・《起原・出所》…『から』[『の』](out of) ・《『名』+『of』+『a』(『an』)+『名』の形で》…のような・《『名』+『of』+『mine』(『yours, his』など独立所有格)の形で》…の…・《時》(1)《副詞句を作って》…に《形容詞句を作って》…の・《時刻》《米》…前(to,《米》before)
蛋白(たんばく)質
『息子』・《複数形で》(男子の)子孫・《文》《比喩(ひゆ)的に》(場所・主義などの)子,後継者・《年長者が年少者に呼び掛けて》・お前さん・《the S-》イエスキリスト

UpToDate Contents

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1. プロテインＳ欠乏症 protein s deficiency
2. プロテインＣ欠乏症 protein c deficiency
3. Factor V Leiden and activated protein C resistance
4. Hypertrophic cardiomyopathy: Gene mutations and clinical genetic testing
5. 慢性骨髄性白血病の細胞生物学および分子生物学 cellular and molecular biology of chronic myeloid leukemia

English Journal

Rit subfamily small GTPases: regulators in neuronal differentiation and survival.

Shi GX, Cai W, Andres DA.SourceDepartment of Molecular and Cellular Biochemistry, University of Kentucky College of Medicine, BBSRB, 741S. Limestone St., Lexington, KY 40536-0509, USA.
Cellular signalling.Cell Signal.2013 Oct;25(10):2060-8. doi: 10.1016/j.cellsig.2013.06.002. Epub 2013 Jun 11.
Ras family small GTPases serve as binary molecular switches to regulate a broad array of cellular signaling cascades, playing essential roles in a vast range of normal physiological processes, with dysregulation of numerous Ras-superfamily G-protein-dependent regulatory cascades underlying the devel
PMID 23770287

Complexity reduction preserving dynamical behavior of biochemical networks.

Apri M, de Gee M, Molenaar J.SourceBiometris, Wageningen University and Research Center, 6708 PB, Wageningen, The Netherlands. mochamad.apri@wur.nl
Journal of theoretical biology.J Theor Biol.2012 Jul 7;304:16-26. doi: 10.1016/j.jtbi.2012.03.019. Epub 2012 Mar 23.
The complexity of biochemical systems, stemming from both the large number of components and the intricate interactions between these components, may hinder us in understanding the behavior of these systems. Therefore, effective methods are required to capture their key components and interactions.
PMID 22465110

Regulation in the targeting of TRAIL receptor 1 to cell surface via GODZ for TRAIL sensitivity in tumor cells.

Oh Y, Jeon YJ, Hong GS, Kim I, Woo HN, Jung YK.SourceGlobal Research Laboratory, School of Biological Science/Bio-Max Institute, Seoul National University, Korea.
Cell death and differentiation.Cell Death Differ.2012 Jul;19(7):1196-207. doi: 10.1038/cdd.2011.209. Epub 2012 Jan 13.
Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) and its receptors, TRAIL-R1 (DR4) and TRAIL-R2 (DR5), promote the selective clearing of various malignancies by inducing apoptosis, holding the promise as a potent therapeutic agent for anticancer. Though DR4 and DR5 have high sequence
PMID 22240897