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1. 原発性硬化性胆管炎：疫学および病因 primary sclerosing cholangitis epidemiology and pathogenesis

English Journal

Gutiérrez-González LA1.
Current rheumatology reports.Curr Rheumatol Rep.2016 Jul;18(7):39. doi: 10.1007/s11926-016-0588-6.
The use of biologics has been associated with the paradoxical development of biologics-induced autoimmune diseases. The purpose of this review was to describe the key immunopathogenic mechanisms involved in the development of these conditions, and to discuss the clinical and laboratory characteristi
PMID 27165496

Further Understanding of the Immunopathology of Multiple Sclerosis: Impact on Future Treatments.

Bhise V1, Dhib-Jalbut S2.
Expert review of clinical immunology.Expert Rev Clin Immunol.2016 May 18. [Epub ahead of print]
INTRODUCTION: The understanding of the immunopathogenesis of multiple sclerosis (MS) has expanded with more research into T-cell subtypes, cytokine contributors, B-cell participation, mitochondrial dysfunction, and more. Treatment options have rapidly expanded with three relatively recent oral thera
PMID 27191526

Increased Expression of the NOD-like Receptor Family, Pyrin Domain Containing 3 Inflammasome in Dermatomyositis and Polymyositis is a Potential Contributor to Their Pathogenesis.

Yin X, Han GC, Jiang XW, Shi Q, Pu CQ1.
Chinese medical journal.Chin Med J (Engl).2016 5th May;129(9):1047-1052. doi: 10.4103/0366-6999.180528.
BACKGROUND: Dermatomyositis (DM) and polymyositis (PM) are common inflammatory myopathies whose immunopathogenic mechanisms remain poorly understood. The NOD-like receptor family, pyrin domain containing 3 (NLRP3) inflammasome is a type of cytoplasmic multiprotein inflammasome and is responsible for
PMID 27098789

西野佳以,斑目広郎,舟場正幸
麻布大学雑誌 13/14, 218-221, 2006-00-00
ボルナ病ウイルス(borna disease virus;BDV)は,馬や羊に髄膜炎あるいは脳脊髄炎をおこし,神経疾患や時に致死的な経過を引き起こすボルナ病の原因ウイルスである。ところが,ラットにおいて高病原性であるCRNP5株は,免疫応答の未熟な新生仔ラットに感染させると脳炎は認められないものの重篤なボルナ病を発症する。従って,CRNP5株は免疫応答非依存的に感染ラットを発症させること,すなわち …
NAID 110006391521

The identification and characterization of new immunogenic egg components : implications for evaluation and control of the immunopathogenic T cell response in schistosomiasis