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1. 若年性特発性関節炎：疫学と免疫病態学 juvenile idiopathic arthritis epidemiology and immunopathogenesis
2. ヘリコバクターピロリ感染の病態生理およびそれへの免疫応答 pathophysiology of and immune response to helicobacter pylori infection
3. HIV感染患者におけるC型肝炎の疫学、自然経過、および診断 epidemiology natural history and diagnosis of hepatitis c in the hiv infected patient
4. デング熱ウイルス感染症の病因 pathogenesis of dengue virus infection
5. HIV感染患者の心障害 cardiac involvement in hiv infected patients

English Journal

Hajarizadeh B1, Grebely J, Applegate T, Matthews GV, Amin J, Petoumenos K, Hellard M, Rawlinson W, Lloyd A, Kaldor J, Dore GJ; ATAHC study group.
Journal of medical virology.J Med Virol.2014 Oct;86(10):1722-9. doi: 10.1002/jmv.24010. Epub 2014 Jul 8.
Understanding viral dynamics during acute hepatitis C virus (HCV) infection can provide important insights into immunopathogenesis and guide early treatment. The aim of this study was to investigate the dynamics of HCV RNA and alanine transaminase (ALT) levels during recent HCV infection in the Aust
PMID 25042465

HIV-1 gp120 Activates the STAT3/Interleukin-6 Axis in Primary Human Monocyte-Derived Dendritic Cells.

Del Cornò M1, Donninelli G1, Varano B1, Da Sacco L2, Masotti A2, Gessani S3.
Journal of virology.J Virol.2014 Oct 1;88(19):11045-55. doi: 10.1128/JVI.00307-14. Epub 2014 Jul 9.
Dendritic cells (DCs) are fundamental for the initiation of immune responses and are important players in AIDS immunopathogenesis. The modulation of DC functional activities represents a strategic mechanism for HIV-1 to evade immune surveillance. Impairment of DC function may result from bystander e
PMID 25008924

Heme Oxygenase-1 Dysregulates Macrophage Polarization and the Immune Response to Helicobacter pylori.

Gobert AP1, Verriere T2, Asim M2, Barry DP2, Piazuelo MB2, de Sablet T2, Delgado AG2, Bravo LE3, Correa P2, Peek RM Jr4, Chaturvedi R2, Wilson KT5.
Journal of immunology (Baltimore, Md. : 1950).J Immunol.2014 Sep 15;193(6):3013-22. doi: 10.4049/jimmunol.1401075. Epub 2014 Aug 8.
Helicobacter pylori incites a futile inflammatory response, which is the key feature of its immunopathogenesis. This leads to the ability of this bacterial pathogen to survive in the stomach and cause peptic ulcers and gastric cancer. Myeloid cells recruited to the gastric mucosa during H. pylori in
PMID 25108023