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1. 高マグネシウム血症の症状 symptoms of hypermagnesemia
2. 非同期性興奮と早期興奮の一般原則 general principles of asynchronous activation and preexcitation
3. 骨代謝の生理学および生化学的マーカー bone physiology and biochemical markers of bone turnover
4. 播種性ヒストプラスマ症の病因および臨床症状 pathogenesis and clinical manifestations of disseminated histoplasmosis
5. 異物肉芽腫 foreign body granulomatosis

αB-crystallin reduces ristocetin‑induced soluble CD40 ligand release in human platelets: Suppression of thromboxane A2 generation.

Tsujimoto M1, Doi T2, Kuroyanagi G3, Yamamoto N3, Matsushima-Nishiwaki R3, Iida Y3, Enomoto Y1, Iida H4, Ogura S2, Otsuka T5, Tokuda H3, Kozawa O3, Iwama T1.
Molecular medicine reports.Mol Med Rep.2015 Jul;12(1):357-62. doi: 10.3892/mmr.2015.3445. Epub 2015 Mar 6.
Our group has previously shown that αB‑crystallin (HSPB5), a small heat shock protein, inhibits human platelet aggregation by ristocetin, an activator of glycoprotein Ib/IX/V. In addition, it was demonstrated that glycoprotein Ib/IX/V activation induces soluble CD40 (sCD40) ligand release via thr
PMID 25760062

Purification of galectin-1 mutants using an immobilized Galactoseβ1-4Fucose affinity adsorbent.

Takeuchi T1, Tamura M2, Ishii N2, Ishikida H2, Sugimoto S2, Suzuki D2, Nishiyama K3, Takahashi H3, Natsugari H3, Arata Y2.
Protein expression and purification.Protein Expr Purif.2015 Jul;111:82-6. doi: 10.1016/j.pep.2015.04.001. Epub 2015 Apr 6.
Galectins are a family of lectins characterized by their carbohydrate recognition domains containing eight conserved amino acid residues, which allows the binding of galectin to β-galactoside sugars such as Galβ1-4GlcNAc. Since galectin-glycan interactions occur extracellularly, recombinant galect
PMID 25858314

Hypophosphatasia: Validation and expansion of the clinical nosology for children from 25years experience with 173 pediatric patients.

Whyte MP1, Zhang F2, Wenkert D3, McAlister WH4, Mack KE5, Benigno MC6, Coburn SP7, Wagy S8, Griffin DM9, Ericson KL10, Mumm S11.
Bone.Bone.2015 Jun;75:229-39. doi: 10.1016/j.bone.2015.02.022. Epub 2015 Feb 27.
Hypophosphatasia (HPP) is caused by loss-of-function mutation(s) within the gene TNSALP that encodes the "tissue-nonspecific" isoenzyme of alkaline phosphatase (TNSALP). In HPP, inorganic pyrophosphate, an inhibitor of mineralization and substrate for TNSALP, accumulates extracellularly often leadin
PMID 25731960

Characterization of glycerophosphoethanolamine ethanolaminephosphodiesterase from Streptomyces sanglieri(ENZYMOLOGY, PROTEIN ENGINEERING, AND ENZYME TECHNOLOGY)