- トリコスタチン、(省略して)リコスタチンA
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- 1. 免疫調節および抗線維化による全身性硬化症（強皮症）治療 immunomodulatory and antifibrotic approaches to the treatment of systemic sclerosis scleroderma
- 2. デュシェンヌ型およびベッカー型筋ジストロフィーの治療 treatment of duchenne and becker muscular dystrophy
- 3. 選択的エストロゲン受容体モジュレーターの作用機序 mechanisms of action of selective estrogen receptor modulators
- 4. βサラセミアの治療 treatment of beta thalassemia
English Journal
- Kinetic method for the large-scale analysis of the binding mechanism of histone deacetylase inhibitors.
- Meyners C1, Baud MG2, Fuchter MJ3, Meyer-Almes FJ4.
- Analytical biochemistry.Anal Biochem.2014 Sep 1;460:39-46. doi: 10.1016/j.ab.2014.05.014. Epub 2014 Jun 2.
- Performing kinetic studies on protein ligand interactions provides important information on complex formation and dissociation. Beside kinetic parameters such as association rates and residence times, kinetic experiments also reveal insights into reaction mechanisms. Exploiting intrinsic tryptophan
- PMID 24882269
- Epigenetic silencing of BTB and CNC homology 2 and concerted promoter CpG methylation in gastric cancer.
- Haam K1, Kim HJ1, Lee KT2, Kim JH2, Kim M1, Kim SY1, Noh SM3, Song KS3, Kim YS4.
- Cancer letters.Cancer Lett.2014 Sep 1;351(2):206-14. doi: 10.1016/j.canlet.2014.05.009. Epub 2014 May 23.
- BTB and CNC homology 2 (BACH2) is a lymphoid-specific transcription factor with a prominent role in B-cell development. Genetic polymorphisms within a single locus encoding BACH2 are associated with various autoimmune diseases and allergies. In this study, restriction landmark genomic scanning revea
- PMID 24858026
- Inhibitory activities of trichostatin a in u87 glioblastoma cells and tumorsphere-derived cells.
- Sassi Fde A1, Caesar L, Jaeger M, Nör C, Abujamra AL, Schwartsmann G, de Farias CB, Brunetto AL, Lopez PL, Roesler R.
- Journal of molecular neuroscience : MN.J Mol Neurosci.2014 Sep;54(1):27-40. doi: 10.1007/s12031-014-0241-7. Epub 2014 Jan 25.
- Epigenetic alterations have been increasingly implicated in glioblastoma (GBM) pathogenesis, and epigenetic modulators including histone deacetylase inhibitors (HDACis) have been investigated as candidate therapies. GBMs are proposed to contain a subpopulation of glioblastoma stem cells (GSCs) that
- PMID 24464841
Japanese Journal
- P2-41-1 ヒストン脱アセチル化酵素阻害剤Trichostatin A (TSA)によるレチノイン酸合成酵素(RALDH)発現について(Group 82 生殖医学4 中枢・他,一般演題,公益社団法人日本産科婦人科学会第67回学術講演会)
- 金崎 春彦,ミジドルジ ツェルメグ,スクバッタル ウヌルジャルガル,折出 亜希,石原 とも子,原 友美,京 哲
- 日本産科婦人科學會雜誌 67(2), 755, 2015-02-01
- NAID 110009941337
- IC50濃度のTrichostatin A (HDAC阻害薬)によるレチノイン酸誘導の神経芽腫細胞株の増殖停止とアポトーシスの増強
- 力石 健,今泉 益栄,新妻 秀剛 [他]
- 日本小児血液・がん学会雑誌 = Japanese journal of Pediatric Hematology/Oncology 52(1), 10-19, 2015
- NAID 40020456068
- Effects of Trichostatin A on In Vitro Development and DNA Methylation Level of the Satellite I Region of Swamp Buffalo (Bubalus bubalis) Cloned Embryos
- SRIRATTANA Kanokwan,KETUDAT-CAIRNS Mariena,NAGAI Takashi [他]
- The journal of reproduction and development 60(5), 336-341, 2014-10
- NAID 40020237660
Related Links
- Trichostatin A (TSA) is a histone deacetylase (HDAC) inhibitor with IC50 of ~1.8 nM. Find all the information about Trichostatin A (TSA) for cell signaling research. ... In vitro HDAC activity Total cellular extracts are prepared from each ...
- Trichostatin A (TSA) is a potent and specific inhibitor of histone deacetylase (HDAC). TSA suppresses the activity of HDAC leading to an increase in histone acetylation. TSA has been shown to induce apoptosis in many cancer cells ...
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