- "Striae" is also a general term referring to thin, narrow grooves or channels, or a thin line or band especially if several of them are parallel or close together.
| Striae atrophicae |
| Classification and external resources |
|
| ICD-10 |
L90.6 |
| ICD-9 |
701.3 |
| DiseasesDB |
30027 |
| MedlinePlus |
003287 |
| eMedicine |
derm/406 |
Stretch marks in a female breast
Striae distensae in an obese white male
Striae gravidarum in a white female two weeks before delivery of a healthy infant
Stretch marks or striae (singular stria), as they are called in dermatology, are a form of scarring on the skin with an off-color hue. They are caused by tearing of the dermis, which over time may diminish, but will not disappear completely.
Stretch marks are often the result of the rapid stretching of the skin associated with rapid growth or rapid weight changes. Stretch marks may also be influenced by hormonal changes associated with puberty, pregnancy, bodybuilding, hormone replacement therapy, etc.[1] Medical terminology for these kinds of markings includes striae atrophicae, vergetures, stria distensae, striae cutis distensae, lineae atrophicae, linea albicante, or simply striae. Stretch marks formed during pregnancy, usually during the last trimester, and usually on the belly, but also commonly occurring on the breasts, thighs, hips, lower back and buttocks, are known as striae gravidarum.[2]
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Contents
- 1 Symptoms and signs
- 2 Causes
- 3 Prevention
- 4 Removal
- 5 References
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Symptoms and signs [edit]
They first appear as reddish or purple lines, but tend to gradually fade to a lighter range. The affected areas appear empty and are soft to the touch.[3] Stretch marks occur in the dermis, the resilient middle layer that helps the skin retain its shape. No stretch marks will form as long as there is support within the dermis. Stretching plays more of a role in where the marks occur and in what direction they run. Stretching alone is not the cause.
Stretch marks can appear anywhere on the body, but are most likely to appear in places where larger amounts of fat are stored. Most common places are the abdomen (especially near the navel), breasts, upper arms, underarms, back, thighs (both inner and outer), hips, and buttocks. They pose no health risk in and of themselves, and do not compromise the body's ability to function normally and repair itself.[4]
Causes [edit]
A number of causes promote the appearance of stretchmarks: one study of 324 women just after giving birth demonstrated that low maternal age, high body mass index, weight gain over 15 kg (33 pounds) and higher neonatal birth weight were independently associated with the occurrence of striae. Teenagers are at highest risk of developing severe striae.[5]
The glucocorticoid hormones responsible for the development of stretch marks affect the dermis by preventing the fibroblasts from forming collagen and elastin fibers, necessary to keep rapidly growing skin taut. This creates a lack of supportive material, as the skin is stretched and leads to dermal and epidermal tearing.
Skin subjected to more stretching force than it can handle will tear. Hormonal changes and genetics influence the skin's capacity to withstand stretching forces, as do diet and (possibly) exercise.[6]
Prevention [edit]
Between 75% and 90% of women develop stretch marks to some degree during pregnancy. The sustained hormonal levels as a result of pregnancy usually means stretch marks may appear during the sixth or seventh month, primarily during the 3rd trimester, as that is when skin tends to be subjected to higher levels of stretching forces. A German research team tested the application of massage and cream and found that only one-third of women with this treatment developed stretchmarks, while two-thirds of an untreated control group developed stretchmarks, but it is not clear if this was a double-blind study.[7]
A randomized controlled study tested whether oils or creams prevent the development of stretchmarks. This study found a daily application of a cream (Trofolastin) containing Gotu Kola extract and vitamin E was associated with fewer stretch marks during pregnancy.[8][9] Another study, though lacking a placebo control, examined a cream (Verum) containing vitamin E, panthenol, hyaluronic acid, elastin and menthol. It was associated with fewer stretch marks during pregnancy versus no treatment.[9] During pregnancy, the pregnant woman can prevent itchiness from the dry, stretched skin by keeping it moisturized.[10][unreliable source?]
Another randomized, placebo-controlled double-blind study of 300 women tested cocoa butter. The result was not found to be a statistically significant difference.[11]
Removal [edit]
Various treatments are available for the purpose of improving the appearance of existing stretch marks, including laser treatments, dermabrasion, and prescription retinoids.[citation needed] Some cream manufacturers claim the best results are achieved on recent stretch marks; however, few studies exist to support these claims.
A study in the journal Dermatologic Surgery showed that radiofrequency combined with 585-nm pulsed dye laser treatment gave "good and very good" subjective improvement in stretch marks in 33 of 37 patients, although further studies would be required to follow up on these results. In addition, the use of a pulsed dye laser was shown to increase pigmentation in darker skinned individuals with repeated treatments.[12]
A surgical procedure for removing lower abdominal stretch marks is the tummy tuck, which removes the skin below the navel where stretch marks frequently occur.
A new modality, fractional laser resurfacing, offers a novel approach to treating striae. Using scattered pulses of light only a fraction of the scar is zapped by the laser over the course of several treatments. This creates microscopic wounds. The body responds to each treatment by producing new collagen and epithelium. In a 2007 clinical trial, 5-6 treatments resulted in striae improving by as much as 75 percent.[13] A 2007 Brazilian clinical study showed that fractional laser resurfacing improved both texture and appearance of mature, white striae in skin types I-IV.[14]
A recent study of new modality for the treatment of stretch marks published in the Journal of Dermatological Treatment was done on seventeen females with stretch marks that underwent six weekly treatments. Results showed that at one week after the final (sixth) treatment 38.2% and 11.8% of the patients were assessed to have 25-50% and 51-75% improvement in the appearance of their stretch marks, respectively. The long term effect of the treatment was confirmed during the 6 weeks follow up which showed that a higher percentage of the patients were rated to have improvement of their stretch marks, including 26.5% and 5.9% showing 51-75% and >75% improvement, respectively. None of the participants was rated as having no improvement in their clinical appearance of stretch marks. Patient satisfaction was also measured and 65% of patients reported that they were very satisfied with the treatment, 23% were satisfied and 12% were slightly satisfied.[15]
References [edit]
- ^ Bernstein, Eric. What Causes Stretch Marks?. 15 December 2008. The Patient's Guide to Stretch Marks. 10 Feb 2009
- ^ "Are Pregnancy Stretch Marks Different?". American Pregnancy Association.
- ^ "Stretch Mark". Encyclopædia Britannica. Retrieved 2009-11-01.
- ^ "How to prevent and treat stretch marks". www.ivillage.co.uk. iVillage. Retrieved 2009-11-01.
- ^ Atwal, G.S.S.; Manku, L.K.; Griffiths, C.E.M.; Polson, D.W. (2006). "Striae gravidarum in primiparae". British Journal of Dermatology 155 (5): 965–9. doi:10.1111/j.1365-2133.2006.07427.x. PMID 17034526.
- ^ "Role of Hormones In the Development of Stretch Marks". Stretch Marks. nostrechmarks.com. Retrieved 2009-11-01. [unreliable medical source?]
- ^ Wierrani, F; Kozak, W; Schramm, W; Grünberger, W (1992). "Attempt of preventive treatment of striae gravidarum using preventive massage ointment administration". Wiener klinische Wochenschrift 104 (2): 42–4. PMID 1609525.
- ^ Mallol, J.; Belda, M.A.; Costa, D.; Noval, A.; Sola, M. (1991). "Prophylaxis of Striae gravidarum with a topical formulation. A double blind trial". International Journal of Cosmetic Science 13 (1): 51–57. doi:10.1111/j.1467-2494.1991.tb00547.x. PMID 19291041.
- ^ a b Young, Gavin; Jewell, David; Young, Gavin (1996). "Creams for preventing stretch marks in pregnancy". In Young, Gavin. Cochrane Database of Systematic Reviews. doi:10.1002/14651858.CD000066.
- ^ Stretch Marks During Pregnancy PregnancyBest. Retrieved on 2011-09-22.
- ^ Buchanan, Keisha; Fletcher, Horace M.; Reid, Marvin (2010). "Prevention of striae gravidarum with cocoa butter cream". International Journal of Gynecology & Obstetrics 108 (1): 65–8. doi:10.1016/j.ijgo.2009.08.008. PMID 19793585.
- ^ Suh, Dong-HYE; Chang, KA-Yeun; Son, HO-Chan; Ryu, JI-HO; Lee, Sang-JUN; Song, KYE-Yong (2007). "Radiofrequency and 585-nm Pulsed Dye Laser Treatment of Striae Distensae: A Report of 37 Asian Patients". Dermatologic Surgery 33 (1): 29–34. doi:10.1111/j.1524-4725.2007.33004.x. PMID 17214676.
- ^ Petrou I (February 2007). "Fractional photothermolysis tackles striae distensae". Dermatology Times 28 (2): 94–106. Retrieved 2007-05-23.
- ^ "Fractional photothermolysis for the treatment of striae distensae". Journal of the American Academy of Dermatology 56 (2, Supplement 2): AB204–AB204. 2007. doi:10.1016/j.jaad.2006.10.931.
- ^ Manuskiatti, Woraphong; Boonthaweeyuwat, Einapak; Varothai, Supenya (2009). "Treatment of striae distensae with a TriPollar radiofrequency device: A pilot study". Journal of Dermatological Treatment 20 (6): 359–64. doi:10.1080/09546630903085278. PMID 19954393.
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Cutaneous keratosis, ulcer, atrophy, and necrobiosis (L82–L94, 700–701.5)
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| Epidermal thickening |
- keratoderma: Keratoderma climactericum
- Paraneoplastic keratoderma
- Acrokeratosis paraneoplastica of Bazex
- Aquagenic keratoderma
- Drug-induced keratoderma
- Paraneoplastic keratoderma
- psoriasis
- Keratoderma blennorrhagicum
- keratosis: Seborrheic keratosis
- Clonal seborrheic keratosis
- Common seborrheic keratosis
- Irritated seborrheic keratosis
- Seborrheic keratosis with squamous atypia
- Reticulated seborrheic keratosis
- Dermatosis papulosa nigra
- Keratosis punctata of the palmar creases
- other hyperkeratosis: Acanthosis nigricans
- Confluent and reticulated papillomatosis
- Callus
- Ichthyosis acquisita
- Arsenical keratosis
- Chronic scar keratosis
- Hyperkeratosis lenticularis perstans
- Hydrocarbon keratosis
- Hyperkeratosis of the nipple and areola
- Inverted follicular keratosis
- Lichenoid keratosis
- Multiple minute digitate hyperkeratosis
- PUVA keratosis
- Reactional keratosis
- Stucco keratosis
- Thermal keratosis
- Viral keratosis
- Warty dyskeratoma
- Waxy keratosis of childhood
- other hypertrophy: Keloid
- Hypertrophic scar
- Cutis verticis gyrata
|
|
| Necrobiosis/granuloma |
| Necrobiotic/palisading |
- Granuloma annulare
- Perforating
- Generalized
- Subcutaneous
- Granuloma annulare in HIV disease
- Localized granuloma annulare
- Patch-type granuloma annulare
- Necrobiosis lipoidica
- Annular elastolytic giant cell granuloma
- Granuloma multiforme
- Necrobiotic xanthogranuloma
- Palisaded neutrophilic and granulomatous dermatitis
- Rheumatoid nodulosis
- Interstitial granulomatous dermatitis/Interstitial granulomatous drug reaction
|
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| Foreign body granuloma |
- Beryllium granuloma
- Mercury granuloma
- Silica granuloma
- Silicone granuloma
- Zirconium granuloma
- Soot tattoo
- Tattoo
- Carbon stain
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| Other/ungrouped |
|
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|
Dermis/
localized CTD |
Cutaneous lupus
erythematosus |
- chronic: Discoid
- Panniculitis
- ungrouped: Chilblain
- Lupus erythematosus–lichen planus overlap syndrome
- Tumid
- Verrucous
- Rowell's syndrome
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Scleroderma/
Morphea |
- Localized scleroderma
- Localized morphea
- Morphea–lichen sclerosus et atrophicus overlap
- Generalized morphea
- Atrophoderma of Pasini and Pierini
- Pansclerotic morphea
- Morphea profunda
- Linear scleroderma
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|
Atrophic/
atrophoderma |
- Lichen sclerosus
- Anetoderma
- Schweninger–Buzzi anetoderma
- Jadassohn–Pellizzari anetoderma
- Atrophoderma of Pasini and Pierini
- Acrodermatitis chronica atrophicans
- Semicircular lipoatrophy
- Follicular atrophoderma
- Linear atrophoderma of Moulin
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| Perforating |
- Kyrle disease
- Reactive perforating collagenosis
- Elastosis perforans serpiginosa
- Perforating folliculitis
- Acquired perforating dermatosis
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| Skin ulcer |
|
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| Other |
- Calcinosis cutis
- Sclerodactyly
- Poikiloderma vasculare atrophicans
- Ainhum/Pseudo-ainhum
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noco (i/b/d/q/u/r/p/m/k/v/f)/cong/tumr (n/e/d), sysi/epon
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proc, drug (D2/3/4/5/8/11)
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