WordNet

sprinkle as if with salt; "the rebels had salted the fields with mines and traps"
a compound formed by replacing hydrogen in an acid by a metal (or a radical that acts like a metal)
preserve with salt; "people used to salt meats on ships"
the taste experience when common salt is taken into the mouth (同)saltiness, salinity
white crystalline form of especially sodium chloride used to season and preserve food (同)table_salt, common salt
(of speech) painful or bitter; "salt scorn"- Shakespeare; "a salt apology"
add salt to
add zest or liveliness to; "She salts her lectures with jokes"
(used especially of meats) preserved in salt (同)salt-cured, brine-cured
something lost (especially money lost at gambling) (同)losses

PrepTutorEJDIC

〈U〉『塩』食塩 / 〈U〉(化学で)塩 / 《複数形で》薬用塩類(Epsom salt[s],smelling salts) / 〈U〉生気(刺激,興趣)を与えるもの / 〈C〉《話》(特に老練な)水夫 / 塩の,塩を含んでいる;塩気のある,塩辛い / 塩漬けにした / (土地が)塩につかった / 塩信でできた,塩水の中で育つ / 〈食べ物〉‘を'塩で味をつける;〈肉・魚など〉‘を'塩漬けにして保存する《+『名』+『down』,+『down』+『名』》 / 《受動態で》(…で)〈言葉・話なで〉‘を'ぴりっとさせる,‘に'味をつける《+『名』+『with』+『名』》
塩で味をつけて,塩味の;塩漬けの
Strategic Arms Limitation Talks戦略兵器制限交渉
負ける,損をする,勝ち目がない

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Novel missense mutations of WNK1 in patients with hypokalemic salt-losing tubulopathies.

Zhang C, Zhu Y, Huang F, Jiang G, Chang J, Li R.SourceDepartment of Nephrology, Xinhua Hospital Chongming Branch, School of Medicine, Shanghai Jiao Tong University, Shanghai, China. zhangchongzc@gmail.com
Clinical genetics.Clin Genet.2013 Jun;83(6):545-52. doi: 10.1111/cge.12008. Epub 2012 Sep 27.
Clinical and genetic studies have suggested that a loss of function and gain of function mutation in the same gene can cause different diseases. The aim of this study was to test the hypothesis that inactivating mutations in WNK1 (with no K (lysine) protein kinase-1) or WNK4 could be a new candidate
PMID 22934535

Novel CLCNKB Mutations Causing Bartter Syndrome Affect Channel Surface Expression.

Keck M, Andrini O, Lahuna O, Burgos J, Cid LP, Sepúlveda FV, L'hoste S, Blanchard A, Vargas-Poussou R, Lourdel S, Teulon J.SourceUPMC Université Paris 06, UMR_S 872, Laboratoire de génomique, physiologie et physiopathologie rénales, Paris, France; INSERM, UMR_S 872, Laboratoire de génomique, physiologie et physiopathologie rénales, Paris, France.
Human mutation.Hum Mutat.2013 May 22. doi: 10.1002/humu.22361. [Epub ahead of print]
Mutations in the CLCNKB gene encoding the ClC-Kb Cl- channel cause Bartter syndrome, which is a salt-losing renal tubulopathy. Here, we investigate the functional consequences of seven mutations. When expressed in Xenopus laevis oocytes, four mutants carried no current (c.736G>C, p.Gly246Arg; c.1
PMID 23703872

Generation and validation of a zebrafish model of EAST (epilepsy, ataxia, sensorineural deafness and tubulopathy) syndrome.

Mahmood F, Mozere M, Zdebik AA, Stanescu HC, Tobin J, Beales PL, Kleta R, Bockenhauer D, Russell C.SourceDepartment of Comparative Biomedical Sciences, Royal Veterinary College, London, UK.
Disease models & mechanisms.Dis Model Mech.2013 May;6(3):652-60. doi: 10.1242/dmm.009480. Epub 2013 Feb 14.
Recessive mutations in KCNJ10, which encodes an inwardly rectifying potassium channel, were recently identified as the cause of EAST syndrome, a severe and disabling multi-organ disorder consisting of epilepsy, ataxia, sensorineural deafness and tubulopathy that becomes clinically apparent with seiz
PMID 23471908

TERANISHI Junya,NAGATOYA Katsuyuki,KAKITA Tomoko,YAMAUCHI Yoko,MATSUDA Hirohisa,MORI Tatsuhiko,INOUE Toru
Clinical and experimental nephrology 14(4), 377-380, 2010-08-01
NAID 10027706890

Siblings of 21-Hydroxylase Deficiency (Non-Salt-Losing) with Aldosterone Hypersecretion.

MORIMOTO Isao,YAMAMOTO Shigeki,TATEISHI Hajime,UEDA Singi,TODA Youko,ZEKI Kazuya,WATANABE Kenichi,NAKANO Yoichiro,KASHIMURA Masamichi,ETO Sumiya
Internal Medicine 31(5), 690-694, 1992
… We describe siblings with the non-salt-losing form of 21-hydroxylase deficiency who had hypersecretion of aldosterone and plasma renin activity (PRA). … Furthermore, aldosterone hypersecretion and high PRA may serve to compensate for the sodium loss which results in turn from the overproduction of the sodium-losing steroids, such as progesterone and 17α-hydroxyprogesterone which are aldosterone antagonists.(Internal Medicine 31 : 690-694, 1992) …
NAID 130000770073