WordNet

UpToDate Contents

全文を閲覧するには購読必要です。 To read the full text you will need to subscribe.

Isolation of Synaptosomes, Synaptic Plasma Membranes, and Synaptic Junctional Complexes.

Michaelis ML1, Jiang L2, Michaelis EK2.
Methods in molecular biology (Clifton, N.J.).Methods Mol Biol.2017;1538:107-119.
Isolation of synaptic nerve terminals or synaptosomes provides an opportunity to study the process of neurotransmission at many levels and with a variety of approaches. For example, structural features of the synaptic terminals and the organelles within them, such as synaptic vesicles and mitochondr
PMID 27943187

Annexin A2 is involved in Ca2+-dependent plasma membrane repair in primary human endothelial cells.

Koerdt SN1, Gerke V2.
Biochimica et biophysica acta.Biochim Biophys Acta.2016 Dec 9. pii: S0167-4889(16)30332-9. doi: 10.1016/j.bbamcr.2016.12.007. [Epub ahead of print]
Many cells in an organism are exposed to constant and acute mechanical stress that can induce plasma membrane injuries. These plasma membrane wounds have to be resealed rapidly to guarantee cell survival. Plasma membrane resealing in response to mechanical strain has been studied in some detail in m
PMID 27956131

The effects of poloxamer 188 on the autophagy induced by traumatic brain injury.

Bao H1, Yang X1, Zhuang Y1, Huang Y1, Wang T2, Zhang M2, Dai D3, Wang S1, Xiao H1, Huang G1, Kuai J4, Tao L5.
Neuroscience letters.Neurosci Lett.2016 Nov 10;634:7-12. doi: 10.1016/j.neulet.2016.09.052. Epub 2016 Sep 29.
Poloxamer 188 (P188) has been reported to reseal plasma membranes and attenuate TBI-induced neuronal death by suppressing apoptosis. Recent studies also confirm increased autophagy after traumatic brain injury (TBI). The present study aimed to investigate the effects of plasmalemmal resealing by P18
PMID 27693566