WordNet

commit a sin; violate a law of God or a moral law (同)transgress, trespass
violent and excited activity; "they began to fight like sin" (同)hell
estrangement from god (同)sinfulness, wickedness
an act that is regarded by theologians as a transgression of Gods will (同)sinning
the 21st letter of the Hebrew alphabet
any of various air-filled cavities especially in the bones of the skull

PrepTutorEJDIC

〈U〉(宗教・道徳上の)『罪』 / 〈C〉(宗教・道徳上の)『罪の行為』,罪悪 / 〈C〉《特におどけて》違反(過失)の行為 / (宗教・道徳上の)おきてを破る,罪を犯す / (…に)違反(過失)をする,背く《+『against』+『名』》
sine
洞(どう)(骨や組織の中の穴・くぼみなど);静脈洞
腎臓の

Wikipedia preview

出典(authority):フリー百科事典『ウィキペディア（Wikipedia）』「2014/10/15 18:31:06」(JST)

wiki en

The renal sinus is a cavity within the kidney which is occupied by the renal pelvis, renal calyces, blood vessels, nerves and fat.

Additional Images

Renal sinus
Renal sinus

External links

Renal+sinus at eMedicine Dictionary

This article related to the genitourinary system is a stub. You can help Wikipedia by expanding it.

UpToDate Contents

全文を閲覧するには購読必要です。 To read the full text you will need to subscribe.

1. 鼻症状の原因：概要 etiologies of nasal symptoms an overview
2. 先天性腎尿路異常（CAKUT）の概要 overview of congenital anomalies of the kidney and urinary tract cakut
3. 結節性硬化症の腎症状 renal manifestations of tuberous sclerosis complex
4. 副鼻腔癌 paranasal sinus cancer
5. 小児における腎移植の一般原則 general principles of renal transplantation in children

English Journal

Six1 and Eya1 are critical regulators of peri-cloacal mesenchymal progenitors during genitourinary tract development.

Wang C, Gargollo P, Guo C, Tang T, Mingin G, Sun Y, Li X.AbstractThe evolutionarily conserved Six1-Eya1 transcription complex is central to mammalian organogenesis, and deletion of these genes in mice results in developmental anomalies of multiple organs that recapitulate human branchio-oto-renal (BOR) and DiGeorge syndromes. Here, we report that both Six1 and Eya1 are strongly expressed in the peri-cloacal mesenchyme (PCM) surrounding the cloaca, the terminal end of hindgut dilation. Six1 and Eya1 are absent from the intra-cloacal mesenchyme (ICM), a cell mass that divides the cloaca into dorsal hindgut and ventral urogenital sinus. Deletion of either or both Six1 and Eya1 genes results in a spectrum of genitourinary tract defects including persistent cloaca - hypoplastic perineum tissue between external urogenital and anorectal tracts; hypospadias - ectopic ventral positioning of the urethral orifice; and hypoplastic genitalia. Analyses of critical signaling molecules indicate normal expression of Shh in the cloaca and cloaca-derived endodermal epithelia. Using a Cre/loxP genetic fate mapping strategy, we demonstrate that Six1-positive PCM progenitors give rise to the most caudal structures of the body plan including the urogenital and anorectal complex, and the perineum region. Thus, Six1 and Eya1 are key regulators of both upper and lower urinary tract morphogenesis. Results from this study uncover essential roles of the PCM progenitors during genitourinary tract formation.
Developmental biology.Dev Biol.2011 Dec 1;360(1):186-94. Epub 2011 Sep 24.
The evolutionarily conserved Six1-Eya1 transcription complex is central to mammalian organogenesis, and deletion of these genes in mice results in developmental anomalies of multiple organs that recapitulate human branchio-oto-renal (BOR) and DiGeorge syndromes. Here, we report that both Six1 and Ey
PMID 21968101