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- 1. 皮膚リーシュマニア症の治療 treatment of cutaneous leishmaniasis
English Journal
- Leukocyte extravasation and vascular permeability are each controlled in vivo by different tyrosine residues of VE-cadherin.
- Wessel F1, Winderlich M1, Holm M1, Frye M1, Rivera-Galdos R1, Vockel M1, Linnepe R1, Ipe U1, Stadtmann A2, Zarbock A3, Nottebaum AF1, Vestweber D1.
- Nature immunology.Nat Immunol.2014 Mar;15(3):223-30. doi: 10.1038/ni.2824. Epub 2014 Feb 9.
- Tyrosine phosphorylation of the adhesion molecule VE-cadherin is assumed to affect endothelial junction integrity. However, it remains unclear whether tyrosine residues of VE-cadherin are required for the induction of vascular permeability and the regulation of leukocyte extravasation in vivo. We fo
- PMID 24487320
- Cutaneous leishmaniasis in Switzerland: first experience with species-specific treatment.
- Mosimann V1, Neumayr A, Hatz C, Blum JA.
- Infection.Infection.2013 Dec;41(6):1177-82. doi: 10.1007/s15010-013-0500-5. Epub 2013 Jul 9.
- BACKGROUND: Different species of the genus Leishmania can cause cutaneous (CL) and mucosal leishmaniasis (ML). PCR-based tests allow a rapid diagnosis and determination of the species, thereby enabling species-oriented treatment. Such treatment procedures have not been evaluated to date.METHODS: Pat
- PMID 23835701
- High-throughput screening of a collection of known pharmacologically active small compounds for identification of Candida albicans biofilm inhibitors.
- Siles SA1, Srinivasan A, Pierce CG, Lopez-Ribot JL, Ramasubramanian AK.
- Antimicrobial agents and chemotherapy.Antimicrob Agents Chemother.2013 Aug;57(8):3681-7. doi: 10.1128/AAC.00680-13. Epub 2013 May 20.
- Candida albicans is the most common etiologic agent of systemic fungal infections with unacceptably high mortality rates. The existing arsenal of antifungal drugs is very limited and is particularly ineffective against C. albicans biofilms. To address the unmet need for novel antifungals, particular
- PMID 23689719
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