WordNet

malignant neoplasm of blood-forming tissues; characterized by abnormal proliferation of leukocytes; one of the four major types of cancer (同)leukaemia, leucaemia, cancer of the blood

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1. 急性骨髄性白血病の合併症の概要 overview of the complications of acute myeloid leukemia
2. 慢性リンパ性白血病に対する治療の概要 overview of the treatment of chronic lymphocytic leukemia
3. 小児および思春期における急性リンパ芽球性白血病の症状および診断の概要 overview of the presentation and diagnosis of acute lymphoblastic leukemia in children and adolescents
4. 小児および思春期における急性リンパ芽球性白血病の転帰に関する概要 overview of the outcome of acute lymphoblastic leukemia in children and adolescents
5. 小児および思春期における急性リンパ芽球性白血病の治療の概要 overview of the treatment of acute lymphoblastic leukemia in children and adolescents

Molecular characteristics of a pancreatic adenocarcinoma associated with Shwachman-Diamond syndrome.

Dhanraj S, Manji A, Pinto D, Scherer SW, Favre H, Loh ML, Chetty R, Wei AC, Dror Y.SourceCell Biology Program, Research Institute, The Hospital for Sick Children, University of Toronto, Toronto, Ontario, Canada.
Pediatric blood & cancer.Pediatr Blood Cancer.2013 May;60(5):754-60. doi: 10.1002/pbc.24453. Epub 2013 Jan 9.
BACKGROUND: Shwachman-Diamond syndrome (SDS) is characterized by hypoplasia of the bone marrow and exocrine pancreas and a high risk of leukemia. It is unknown whether solid tumors are part of the disease phenotype.PROCEDURE: We performed copy number alterations using Affymetrix human SNP 6.0 array.
PMID 23303473

Acute leukemia showing t(8;22)(p11;q11), myelodysplasia, CD13/CD33/CD19 expression and immunoglobulin heavy chain gene rearrangement.

Shimanuki M, Sonoki T, Hosoi H, Watanuki J, Murata S, Mushino T, Kuriyama K, Tamura S, Hatanaka K, Hanaoka N, Nakakuma H.SourceHematology/Oncology, Wakayama Medical University, Wakayama 641-8509, Japan.
Acta haematologica.Acta Haematol.2013;129(4):238-42. doi: 10.1159/000345727. Epub 2013 Jan 12.
t(8;22)(p11;q11) is a rare but recurrent chromosome translocation that has been reported in 11 cases of myeloproliferative neoplasm or B-acute lymphoblastic leukemia. This translocation results in an in-frame fusion of FGFR1 on 8p11 and BCR on 22q11, and causes constitutive activation of the tyrosin
PMID 23328683

Cytomegalovirus infection and ganciclovir resistance caused by UL97 mutations in pediatric transplant recipients.

Kim YJ, Boeckh M, Cook L, Stempel H, Jerome KR, Boucek R Jr, Burroughs L, Englund JA.SourceFred Hutchinson Cancer Research Center, Seattle, Washington, USA; Seattle Children's Hospital, Seattle, Washington, USA; Sungkyungkwan University, School of Medicine, Samsung Medical Center, Seoul, Korea.
Transplant infectious disease : an official journal of the Transplantation Society.Transpl Infect Dis.2012 Dec;14(6):611-7. doi: 10.1111/j.1399-3062.2012.00760.x.
BACKGROUND: Cytomegalovirus (CMV) infection may cause serious disease after hematopoietic cell transplantation (HCT) and solid organ transplantation (SOT), but few reports describe ganciclovir (GCV) resistance in pediatric patients.OBJECTIVES: This study was performed to describe the clinical impact
PMID 23198963

Acute promyelocytic leukemia presenting initially with pancytopenia and severe hypocellular marrow which was successfully treated with all-trans retinoic acid and chemotherapy

Suzukawa Munehiro,Sugiyama Akiko,Nakazora Tatsuki,Kawasaki Yasufumi,Tominaga Takayuki,Shinohara Kenji
The bulletin of the Yamaguchi Medical School 57(1-2), 9-15, 2010
… Cytogenetic analysis demonstrated t(15;17)(q22;q12) and promyleocytic leukemia/retinoic acid receptor-α (PML/BAR-α) fusion genes on employing interphase fluorescence in situ-hybridization (FISH) and nested PCR... …
NAID 120002314472