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出典(authority):フリー百科事典『ウィキペディア（Wikipedia）』「2015/05/08 06:02:43」(JST)

Hexosyltransferases are a type of glycosyltransferase that catalyze the transfer of a hexose.

Examples include:

They are classified under EC number 2.4.1.

External links

Hexosyltransferases at the US National Library of Medicine Medical Subject Headings (MeSH)
CRISP Thesaurus 00003765

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Prevalence of enteroaggregative Escherichia coli and its virulence-related genes in a case-control study among children from north-eastern Brazil.

Lima IF, Boisen N, Quetz Jda S, Havt A, de Carvalho EB, Soares AM, Lima NL, Mota RM, Nataro JP, Guerrant RL, Lima AÂ.Source1Institute of Biomedicine for Brazilian Semi-Arid and Clinical Research Unit/Center for Global Health, School of Medicine, Federal University of Ceara, Fortaleza, CE, Brazil.
Journal of medical microbiology.J Med Microbiol.2013 May;62(Pt 5):683-93. doi: 10.1099/jmm.0.054262-0. Epub 2013 Feb 21.
Enteroaggregative Escherichia coli (EAEC) is an important agent that causes endemic and epidemic diarrhoeal diseases worldwide. Several EAEC virulence-related genes (VRGs) have been described but their role in the clinical outcome of infection is not completely defined. This study investigated the p
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Sonic hedgehog signaling directly targets Hyaluronic Acid Synthase 2, an essential regulator of phalangeal joint patterning.

Liu J, Li Q, Kuehn MR, Litingtung Y, Vokes SA, Chiang C.SourceDepartment of Cell and Developmental Biology, Vanderbilt University Medical Center, Nashville, TN 37232, USA.
Developmental biology.Dev Biol.2013 Mar 15;375(2):160-71. doi: 10.1016/j.ydbio.2012.12.018. Epub 2013 Jan 8.
Sonic hedgehog (Shh) signal, mediated by the Gli family of transcription factors, plays an essential role in the growth and patterning of the limb. Through analysis of the early limb bud transcriptome, we identified a posteriorly-enriched gene, Hyaluronic Acid Synthase 2 (Has2), which encodes a key
PMID 23313125

Hypomorphic MGAT5 polymorphisms promote multiple sclerosis cooperatively with MGAT1 and interleukin-2 and 7 receptor variants.

Li CF, Zhou RW, Mkhikian H, Newton BL, Yu Z, Demetriou M.SourceDepartment of Neurology, University of California, Irvine, CA 92869, USA.
Journal of neuroimmunology.J Neuroimmunol.2013 Mar 15;256(1-2):71-6. doi: 10.1016/j.jneuroim.2012.12.008. Epub 2013 Jan 22.
Deficiency of the Golgi N-glycan branching enzyme Mgat5 in mice promotes T cell hyperactivity, endocytosis of CTLA-4 and autoimmunity, including a spontaneous multiple sclerosis (MS)-like disease. Multiple genetic and environmental MS risk factors lower N-glycan branching in T cells. These include v
PMID 23351704