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出典(authority):フリー百科事典『ウィキペディア（Wikipedia）』「2014/09/29 06:58:03」(JST)

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1. 入院患者における糖尿病のマネージメント management of diabetes mellitus in hospitalized patients
2. 重症患者における血糖コントロールおよび集中的なインスリン療法 glycemic control and intensive insulin therapy in critical illness
3. 1型糖尿病を有する小児および思春期の患者における合併症およびスクリーニング complications and screening in children and adolescents with type 1 diabetes mellitus
4. 1型糖尿病の小児および青年における運動の管理 management of exercise for children and adolescents with type 1 diabetes mellitus
5. ST上昇型心筋梗塞後の有害転帰の危険因子 risk factors for adverse outcomes after st elevation myocardial infarction

Point-of-care capillary HbA1c measurement in the emergency department: a useful tool to detect unrecognized and uncontrolled diabetes.

Gomez-Peralta F1, Abreu C2, Andreu-Urioste L3, Antolí AC4, Rico-Fontsaré C5, Martín-Fernández D6, Resina-Rufes R7, Pérez-García JJ8, Negrete-Muñoz Á9, Muñoz-Álvarez D10, Umpierrez GE11.
International journal of emergency medicine.Int J Emerg Med.2016 Dec;9(1):7. doi: 10.1186/s12245-016-0107-6. Epub 2016 Feb 19.
BACKGROUND: Inpatient hyperglycaemia and diabetes mellitus (DM) are common and are associated with an increased risk of complications and mortality. The severity of hyperglycaemia determines the rate of complications in patients treated in the emergency department (ED). Our aim was to examine whethe
PMID 26894895

Effects of insulin therapy on porosity, non-enzymatic glycation and mechanical competence in the bone of rats with type 2 diabetes mellitus.

Campbell GM1, Tiwari S2, Picke AK3, Hofbauer C4, Rauner M3, Morlock MM5, Hofbauer LC6, Glüer CC2.
Bone.Bone.2016 Oct;91:186-93. doi: 10.1016/j.bone.2016.08.003. Epub 2016 Aug 4.
Type 2 diabetes mellitus increases skeletal fragility; however, the contributing mechanisms and optimal treatment strategies remain unclear. We studied the effects of diabetes and insulin therapy on non-enzymatic glycation (NEG), cortical porosity (Ct.Po) and biomechanics of the bone tissue in Zucke
PMID 27497735

Methylglyoxal-induced dicarbonyl stress in aging and disease: first steps towards glyoxalase 1-based treatments.

Rabbani N1, Xue M2, Thornalley PJ3.
Clinical science (London, England : 1979).Clin Sci (Lond).2016 Oct 1;130(19):1677-96. doi: 10.1042/CS20160025.
Dicarbonyl stress is the abnormal accumulation of dicarbonyl metabolites leading to increased protein and DNA modification contributing to cell and tissue dysfunction in aging and disease. It is produced by increased formation and/or decreased metabolism of dicarbonyl metabolites. MG (methylglyoxal)
PMID 27555612

Postprandial glucagon-like peptide-1 secretion is increased during the progression of glucose intolerance and obesity in high-fat/high-sucrose diet-fed rats