- 関
- descendant、offspring、progeny
WordNet
- something that comes into existence as a result; "industrialism prepared the way for acceptance of the French Revolutions various socialistic offspring"; "this skyscraper is the solid materialization of his efforts" (同)materialization, materialisation
- the immediate descendants of a person; "she was the mother of many offspring"; "he died without issue" (同)progeny, issue
- proceeding by descent from an ancestor; "descendent gene" (同)descendent
- a person considered as descended from some ancestor or race (同)descendent
- going or coming down (同)descendent
PrepTutorEJDIC
- =descendant
- 《単数・複数扱い》(人間や動物の)『子;子孫』 / (…から)生じたもの,(…の)結果,所産《+『of』+『名』》
- (…の)『子孫』《+『of』+『名』》 / 下りの,下降する / 祖先伝来の,世襲の
- (人・動植物の)子孫
Wikipedia preview
出典(authority):フリー百科事典『ウィキペディア(Wikipedia)』「2013/06/17 16:40:33」(JST)
[Wiki en表示]
Descendant or Descendent may refer to:
- Lineal descendant, a blood relative in the direct line of descent
- Collateral descendant, a relative descended from a brother or sister of an ancestor
- Descendant (astrology), the point directly opposite from the Ascendant, or Rising Sign
- Descendents, a punk rock band from southern California
- Descendant (film), a 2003 film thriller starring Katherine Heigl and Jeremy London
- Descendants (film), a 2008 short film
- "The Descendant" (short story), a short story by H. P. Lovecraft
- The Descendants (novel), a novel by Kaui Hart Hemmings
- The Descendants, a 2011 film by Alexander Payne
- A descendant node in a tree structure
- A subordinate in a hierarchy
See also[edit]
UpToDate Contents
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English Journal
- Triggering receptor expressed on myeloid cells 2 variant is rare in late-onset Alzheimer's disease in Han Chinese individuals.
- Yu JT1, Jiang T2, Wang YL3, Wang HF3, Zhang W3, Hu N3, Tan L3, Sun L3, Tan MS4, Zhu XC2, Tan L5.Author information 1Department of Neurology, Qingdao Municipal Hospital, Nanjing Medical University, Qingdao, China; Department of Neurology, Qingdao Municipal Hospital, School of Medicine, Qingdao University, Qingdao, China; Department of Neurology, Qingdao Municipal Hospital, College of Medicine and Pharmaceutics, Ocean University of China, Qingdao, China. Electronic address: yu-jintai@163.com.2Department of Neurology, Qingdao Municipal Hospital, Nanjing Medical University, Qingdao, China.3Department of Neurology, Qingdao Municipal Hospital, School of Medicine, Qingdao University, Qingdao, China.4Department of Neurology, Qingdao Municipal Hospital, College of Medicine and Pharmaceutics, Ocean University of China, Qingdao, China.5Department of Neurology, Qingdao Municipal Hospital, Nanjing Medical University, Qingdao, China; Department of Neurology, Qingdao Municipal Hospital, School of Medicine, Qingdao University, Qingdao, China; Department of Neurology, Qingdao Municipal Hospital, College of Medicine and Pharmaceutics, Ocean University of China, Qingdao, China. Electronic address: dr.tanlan@163.com.AbstractRecent studies have reported that a rare mutation of triggering receptor expressed on myeloid cells 2 gene (TREM2 [rs75932628-T]) has significantly increased the risk of late-onset Alzhemier's disease (LOAD) in European-descendent population. To date, no study has investigated the association between rare mutations of TREM2 and LOAD risk in non-European population. Here, we sequenced exon2 of TREM2 in the northern Han Chinese population consisting of 1133 patients with LOAD and 1159 control subjects. Although, 4 novel mutations (c.102G>A: Val34Val, c.330C>T: Cys110Cys, c.342T>C: His114His, and c.343G>A: Gly115Ser) were identified in patients with LOAD, none of them exhibited significant association with LOAD risk after Bonferroni correction. Most importantly, the previously reported rare variants in European-descendent population including rs75932628-T (predicted to cause an R47H substitution) were absent in our cohort. These findings suggest that mutations in exon2 of TREM2 were unlikely to play a key role in the susceptibility of LOAD in the northern Han Chinese population.
- Neurobiology of aging.Neurobiol Aging.2014 Apr;35(4):937.e1-3. doi: 10.1016/j.neurobiolaging.2013.10.075. Epub 2013 Oct 11.
- Recent studies have reported that a rare mutation of triggering receptor expressed on myeloid cells 2 gene (TREM2 [rs75932628-T]) has significantly increased the risk of late-onset Alzhemier's disease (LOAD) in European-descendent population. To date, no study has investigated the association betwee
- PMID 24184202
- Citicoline (CDP-choline) protects myocardium from ischemia/reperfusion injury via inhibiting mitochondrial permeability transition.
- Hernández-Esquivel L1, Pavón N2, Buelna-Chontal M3, González-Pacheco H4, Belmont J1, Chávez E5.Author information 1Departamento de Bioquímica, Instituto Nacional de Cardiología, Ignacio Chávez, Mexico, D. F. Mexico.2Departamento de Farmacología, Instituto Nacional de Cardiología, Ignacio Chávez, Mexico, D. F. Mexico.3Departamento de Biomedicina Cardiovascular, Instituto Nacional de Cardiología, Ignacio Chávez, Mexico, D. F. Mexico.4Unidad Coronaria, Instituto Nacional de Cardiología, Ignacio Chávez, Mexico, D. F. Mexico.5Departamento de Bioquímica, Instituto Nacional de Cardiología, Ignacio Chávez, Mexico, D. F. Mexico. Electronic address: echavez@salud.gob.mx.AbstractAIMS: Oxidative stress emerges after reperfusion of an organ following an ischemic period and results in tissue damage. In the heart, an amplified generation of reactive oxygen species and a significant Ca(2+) accumulation cause ventricular arrhythmias and mitochondrial dysfunction. This occurs in consequence of increased non-specific permeability. A number of works have shown that permeability transition is a common substrate that underlies the reperfusion-induced heart injury. The aim of this work was to explore the possibility that CDP-choline may circumvent heart damage and mitochondrial permeability transition.
- Life sciences.Life Sci.2014 Feb 6;96(1-2):53-8. doi: 10.1016/j.lfs.2013.12.026. Epub 2013 Dec 31.
- AIMS: Oxidative stress emerges after reperfusion of an organ following an ischemic period and results in tissue damage. In the heart, an amplified generation of reactive oxygen species and a significant Ca(2+) accumulation cause ventricular arrhythmias and mitochondrial dysfunction. This occurs in c
- PMID 24389400
- Association Study of DKK2 Polymorphisms with Alcohol Dependence and Alcohol-Related Harm.
- Kim JY, Bae JS, Park BL, Kim JH, Kim LH, Kim JW, Lee BC, Kang TC, Choi IG, Shin HD.Author information Department of Life Science, Sogang University, Seoul, Republic of Korea.AbstractBACKGROUND: Alcohol dependence (AD) is a common disorder with both environmental and genetic factors. Previous studies have shown that the genomic region from chromosome 4q22-q32 is closely associated with AD. Furthermore, a study with Irish subjects revealed that the polymorphisms of Dickkopf WNT signaling pathway inhibitor (DKK2), located at 4q25, showed a significant association with AD.
- Alcoholism, clinical and experimental research.Alcohol Clin Exp Res.2014 Feb;38(2):545-50. doi: 10.1111/acer.12265. Epub 2013 Oct 7.
- BACKGROUND: Alcohol dependence (AD) is a common disorder with both environmental and genetic factors. Previous studies have shown that the genomic region from chromosome 4q22-q32 is closely associated with AD. Furthermore, a study with Irish subjects revealed that the polymorphisms of Dickkopf WNT s
- PMID 24117450
Japanese Journal
- MoP-42 ALGORITHMIC PROCEDURE TO DETERMINE FEEDFORWARD TRAJECTORY OF OVERHEAD HOIST TRANSFER MACHINE FOR SUSPENSION OF RESIDUAL VIBRAION OF A LOAD BY USING VIBRATION MANIPUKATION FUNCTION
- Proceedings of ... JSME-IIP/ASME-ISPS Joint Conference on Micromechatronics for Information and Precision Equipment : IIP/ISPS joint MIPE 2015, "MoP-42-1"-"MoP-42-3", 2015-06-14
- NAID 110010044073
- 1P2-D02 天井搬送機構における懸垂体の高速制振を実現する搬送機のフィードフォワード関数
- 102 振動操作関数により導出した天井クレーンの制振軌道(OS4 機械の振動・騒音および制御に関する新技術(1),研究討論セッション)
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★リンクテーブル★
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- 関
- descendant、descendent、progeny
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- 英
- offspring、progeny、descendent、descendant
- 関
- 出生児
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- 関
- descendent、offspring、progeny
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- 関
- descendant、descendent、offspring