WordNet

any of various organs that synthesize substances needed by the body and release it through ducts or directly into the bloodstream (同)secretory organ, secretor, secreter
relating to or associated with the bronchi; "bronchial tubes"; "bronchial pneumonia"

PrepTutorEJDIC

(生物体内の)腺(せん)
気管支の
bronchusの複数形

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1. 唾液腺腫瘍：疫学、診断、評価、および病期分類 salivary gland tumors epidemiology diagnosis evaluation and staging
2. シェーグレン症候群の臨床症状：外分泌腺障害 clinical manifestations of sjogrens syndrome exocrine gland disease
3. 唾石症 salivary gland stones
4. 唾液腺腫瘍：限局性疾患の治療 salivary gland tumors treatment of locoregional disease
5. 気管支神経内分泌（カルチノイド）腫瘍：治療および予後 bronchial neuroendocrine carcinoid tumors treatment and prognosis

English Journal

Middle East Respiratory Syndrome Coronavirus (MERS-CoV) Infection Mediated by the Transmembrane Serine Protease TMPRSS2.

Shirato K, Kawase M, Matsuyama S.SourceDepartment of Virology III, National Institute of Infectious Diseases, Murayama Branch, 4-7-1 Gakuen, Musashi-Murayama, Tokyo 208-0011, Japan.
Journal of virology.J Virol.2013 Sep 11. [Epub ahead of print]
The Middle East respiratory syndrome coronavirus (MERS-CoV) utilizes host proteases for virus entry into lung cells. In the current study, Vero cells constitutively expressing type II transmembrane serine protease (Vero-TMPRSS2 cells) showed larger syncytia at 18 h after infection with MERS-CoV than
PMID 24027332

[Carcinoma of the Pararenal Gland Infiltrating the Surrounding Tissue and Involving the Inferior Vena cava].

Dillner J, Meyer F, Lippert H, Huth C, Klose S, Roessner A, Halloul Z.SourceKlinik für Allgemein-, Viszeral- und Gefäßchirurgie, Universitätsklinikum Magdeburg A. ö. R.
Aktuelle Urologie.Aktuelle Urol.2013 Sep;44(5):375-80. doi: 10.1055/s-0033-1351306. Epub 2013 Sep 16.
A 61-year-old woman presented with a 2-month-history of progressive deterioration, increasing exertional dyspnoea and pain in the right upper abdomen (past medical history: bronchial asthma and hypertension). The physical examination showed mild generalized weakness, tenderness in the right upper ab
PMID 24043537

Imre schneider, ferenc harangi, béla sebők: clinical and pathological aspects of atopic dermatitis.

Lipozenčić J.AbstractThe book Clinical and Pathological Aspects of Atopic Dermatitis brings the latest information on atopic dermatitis. The book has 586 pages and 3 relevant parts. The first one is Clinical Aspects with 10 subparts and 29 color pictures, Pathology with 13 color pictures and 13 subparts, and Therapy with 15 subparts and Recommendations for the Treatment of Atopic Dermatitis and recent literature data. The book includes Abbreviations (7 pages) and Subject Index (14 pages). References follow every subpart in basic chapter: Clinical Aspects with 771 references, Pathology with 276 references, and Therapy with 580 references. In Clinical Aspects, there are valuable International Epidemiological Data on AD (Ferenc Harangi) in Europe (Norway, Russia, Sweden, Germany, France, Great Britain, Spain, Italy, Turkey), as well as in America (USA, Mexico, Argentina), Africa (Tunisia, Nigeria, Ethiopia, West Africa), Asia (Israel, Iran, Korea, Japan, China, Taiwan, India, Singapore), and Australia. A Survey of the Prevalence of AD in Domestic, Living and Nourishment Conditions of Children in Large Hungarian Population Counties offer highly important data. Clinical Symptoms (with 29 color pictures) are described in detail, from Major Criteria (Pruritus, Lichenification, Chronic or Chronically Recurrent Dermatitis, Individual or Family Appearance of Atopy); Minor Criteria (Ichthyosis vulgaris, Keratosis pilaris, Eczema of the hand and foot (dyshidrotic; atopic winter feet), Mammillary and lip eczema, and many more clinical aspects to Periorbital pigmentation. The Course of AD (with 29 color pictures) is divided into Chronic or Chronically Recurring Dermatitis (Infancy, Childhood), Young Adult and Adult Age and other clinical symptoms as well as Assessment of the Clinical Picture and Severity of AD. The histopathology of AD (with 13 color pictures) includes Typical Atopic Dermatitis, Mycosis Fungoides as a Complication of AD, Immunohistologic examination, DNA extraction and Molecular examination, and Case Reviews of three patients with clonal gene rearrangement at 200 bp. The authors proved that some long-lasting therapy-resistant dermatoses like AD may be transformed into mycosis fungoides. Immunodeficiency and AD is described logically with the Eczema in primary immunodeficiency as well as Independent cutaneous markers in 13 different immunodeficiency diseases, which are presented in detail. Differential Diagnostics of AD is presented in 7 parts as 1) Immunodeficiencies, 2) Chronic Inflammatory Dermatoses, 3) Dermatoses due to Infections, 4) Congenital Diseases, 5) Immunological Disorders, 6) Metabolic Disorders, and 7) Malignant Processes. Especially valuable is Chapter 9, Dermatoses Possibly Concomitant with and Disorders Similar to AD (AD and Immunodeficiencies) Ectodermal Dysplasias; Respiratory Atopic Symptoms - Bronchial Asthma; Gastrointestinal Symptoms; Cardiovascular System; AD and Disorders of the Nervous System; AD and Ophthalmological Disorders. The Association of AD with Various Syndromes; AD and Endocrine Disorders (Endocrine Impacts, Diabetes Mellitus, Thyroid Gland, Pituitary Gland); AD and Irregularities of the Hair; AD and Intervertebral Disc Degeneration; The Association of AD and Bone Density; AD and Left-Handedness; AD and Olecranon; AD and Pretibial Bursitis; AD and Viral Infections (Pityriasis Rosea, Molluscum Contagiosum; Primary Herpes Simplex) AD and Autoimmune Diseases; AD and Actinic Reticuloid and Lichen Nitidus; AD and Hypersensitivity to Nonsteroidal Antiinflammatory Drugs; AD and Vitiligo; Atopy and Renal Transplant Rejection; Ichthyosis Vulgaris - Xerosis, and many other diseases in patients with AD. In the AD and Skin Reactivity chapter, the authors classify AD into Allergic (IgE and Non-IgE associated variant), Non allergic AD and Unclassified AD type. The following are described: Prick Test, Intracutan Test, The Significance of the Positive Test, Food Allergens, Atopy Patch Test, RAST, PRIST, and other Diagnostic Methods (total IgE, RAST, Epicutaneous test, Metal Hypersensitivity, Latex Sensitivity, Natural rubber latex, Corticosteroid and Bufexamac allergy, House-Dust Mite, Mite, and Insects, Sensitivity to the Cockroach allergens, Tree, Grass and Weed Pollen, Yeast and mold fungi, dermatophytes, Cosmetics, Fragrance Mixes, Human dander). Second Chapter, Pathology (subparts 11 to 23), is written up-to-date, containing the following contributions: Atopic dermatitis and its genetic background; Results of genomic screening related to AD; Candidate gene studies; Interleukin 13 (IL-13) gene; IL-4 receptor alpha (IL-4 Rα) gene; and Serine protease inhibitor gene (SPINKS5). The following are described in detail: Stratum corneum (SC) and barrier function in AD; Ceramides, the lipids of SC in AD. Important is subchapter 13: Pruritus and AD; Interrelationship between itch and pain; Peripheral sensitization; Mediators of itch; Opioids and cannabinoids; Interleukins; Neutrophils, Prostanoids, Substance P. Neuropeptides are described in subchapter 14; neuropeptides have significant effects and are produced, for example, in the skin nerve endings and in thin C fibers. These polypeptides play a role as neuromodulators, neurotransmitters and neurohormones, which are important in AD. Subchapter 15 (4 color pictures) provides detailed description of the role of various cells in AD, SAG secretin of Staphylococcus aureus, IL-13-stimulated keratinocytes. The role of bacteria, viruses, fungi and mites in AD is described in subchapter 16. IgE and other antibodies are presented in subchapter 17. Food allergy in the etiology of AD is rather controversial and is described in subchapter 18, along with relevant test methods, food intolerance and prevention. The role of inhalant allergens and AD is explained in subchapter 19. Interesting is subchapter 20 on Experimental AD (animal models) in dogs and mice. Immunologic and allergologic background of AD with clinical picture, adhesion, receptors, molecules, cytokines, LCs, enzymes, chemokines is described in detail in subchapter 21, as well as cyclic AMP, free radicals, prostanoids, heat-shock proteins, environmental damages-factors, and abnormalities in the cutaneous vascular regulation. Physiological and psychiatric characteristics of AD are presented in subchapter 22. Subchapter 23 is dedicated to the effects of childhood AD on the family and society and its costs (written by Ferenc Harangi). Subchapter 24, Therapy, written by Béla Sebök, deals with emollients, humectans, carbamide, glycerol, choice of the vehicle, corticosteroids topical and systemic, antihistamines, immunosuppressants (Cyclosporine A®), tacrolimus (Protopic®), pimecrolimus (Elidel®), azathioprine (Imuran®), mycophenolate mofetil (Cellcept®), leflunomide (Arava®), methotrexate; leukotriene antagonists, interferon-α and -γ, thymopentin, intravenous immunoglobulin, unsaturated fatty acids; vitamin E and B12; Chinese herbal therapy; heliotherapy and climatotherapy; phototherapy; photopheresis; drugs under investigation; recommendations for the treatment of AD; practical considerations of topical corticosteroid treatment and calcineurin inhibitors; and patients unresponsive to topical treatment. This book gives an overview of all important segments of atopic dermatitis and most recent knowledge on it. This book is useful not only for dermatologists and pediatricians, but also for gastroenterologists, ophthalmologists and family practitioners who must help patients with this ever increasing disease.
Acta dermatovenerologica Croatica : ADC.Acta Dermatovenerol Croat.2013 Aug;21(2):137-8.
The book Clinical and Pathological Aspects of Atopic Dermatitis brings the latest information on atopic dermatitis. The book has 586 pages and 3 relevant parts. The first one is Clinical Aspects with 10 subparts and 29 color pictures, Pathology with 13 color pictures and 13 subparts, and Therapy wit
PMID 24001425

Japanese Journal

6.喀血を契機に診断されたbronchial mucous gland adenomaの1例(第34回日本呼吸器内視鏡学会北海道支部会)

福家聡,小島哲弥,有賀伸,斎藤拓志,磯部宏,桑原博昭,鈴木昭,西浦洋一
気管支学 : 日本気管支研究会雑誌 34(6), 642-643, 2012-11-25
NAID 110009562619

気管支喘息発作治療中に広範な縦隔気腫とともに気管粘膜下気腫を合併した1症例

鳴海創大,玉田勉,阿部恭子,久田修,海老名雅仁
気管支学 : 日本気管支研究会雑誌 34(2), 184-188, 2012-03-25
背景.気管支喘息発作における合併症の一つとして縦隔気腫が知られている.一方で気管粘膜下気腫の合併が確認される症例は極めて稀であり,また縦隔気腫との関連性も不明である.症例.16歳女性.気管支喘息のため近医にて吸入ステロイドを含む治療中であったが,気管支喘息増悪に加えて広範な縦隔気腫・皮下気腫を合併したため当科紹介となった.入院時胸部CTでは気管内全周性に粘膜剥離像として矛盾しない薄壁の軟組織陰影を …
NAID 110009437403

肺・結膜および耳下腺に同時発生したMALTリンパ腫の1例

藤原俊哉,西川敏雄,片岡和彦,松浦求樹
日本呼吸器外科学会雑誌 = The journal of the Japanese Association for Chest Surgery 25(6), 615-620, 2011-09-15
症例は61歳，女性．検診の胸部X線で右肺野の異常陰影を指摘された．胸部CTで右肺に多発する腫瘤影を認めたため，当院へ紹介となった．初診時，眼瞼結膜に腫瘤を発見した．PET-CTでは右肺上葉の腫瘤に高集積とすりガラス陰影に低集積を認めた．その他，右耳下腺にも低集積を認めた．気管支鏡検査を施行し，右肺上葉の腫瘤の擦過細胞診で，悪性リンパ腫が疑われた．眼科紹介し，結膜腫瘤を生検したところ，MALTリンパ …
NAID 10029458599