WordNet
- a branch of the Indo-European family of languages (同)Slavic language, Slavonic, Slavonic language
- a family of Indo-European languages including the Slavic and Baltic languages (同)Balto-Slavic language, Balto-Slavonic
- a race of people speaking a Slavonic language (同)Slavic race
- speaking a Slavic language; "the Slav population of Georgia"
- any member of the people of eastern Europe or Asian Russia who speak a Slavonic language
PrepTutorEJDIC
- スラブ民族の,スラブ語族の / スラブ語族
- スラブ人;《the Slavs》スラブ民族(ロシア人・ポーランド人・チェコ人・ブルガリア人・スロバキア人など) / スラブ人の,スラブ民族の;スラブ語の
Wikipedia preview
出典(authority):フリー百科事典『ウィキペディア(Wikipedia)』「2016/12/23 19:34:35」(JST)
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Look up Slavic or Slavonic in Wiktionary, the free dictionary. |
Slav, Slavic or Slavonic may refer to:
- Slav (village), a former Israeli settlement in the Gaza Strip
- Slavic people, an ethno-linguistic group living in Europe and Asia
- Slavic languages, a group of closely related Indo-European languages
- Slavic mythology, the mythological aspect of the polytheistic religion that was practised by the Slavs before Christianisation
- Slavic names, names originating from the Slavic languages
- Church Slavonic language, Slavic liturgical language used by the Eastern Orthodox Church
- Old Church Slavonic language, 9th century Slavic literary language
- Slav Defense, a chess opening
See also
- Slavonia, a region in eastern Croatia
- Slavyansky (disambiguation)
UpToDate Contents
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English Journal
- Secondary acute monocytic leukemia positive for 11q23 rearrangement in Nijmegen breakage syndrome.
- Pastorczak A1, Szczepanski T, Trelinska J, Finalet Ferreiro J, Wlodarska I, Mycko K, Polucha A, Sedek L, Meyer C, Marschalek R, Młynarski W.Author information 1Department of Pediatrics, Hematology, Oncology & Diabetology, Medical University of Lodz, Lodz, Poland.AbstractNijmegen breakage syndrome (NBS) is an autosomal recessive chromosomal instability disorder characterized by a high incidence of pediatric hematologic malignancies. Majority of patients affected are of Slavic origin and share the same founder mutation of 657del5 within the NBN gene encoding protein involved in DNA double-strand breaks (DSB) repair. We report a case of a pediatric patient with NBS, who developed t(9;11)/AF9-MLL-positive AML as a second malignancy after successful treatment of T-NHL. The coexistence of NBN and MLL mutations suggests that the profound dysfunction of NBN may promote alterations of MLL that is mediated by error-prone non-homologous end joining pathway particularly in patients treated with DNA topoisomerase II inhibitors. Pediatr Blood Cancer © 2014 Wiley Periodicals, Inc.
- Pediatric blood & cancer.Pediatr Blood Cancer.2014 Mar 11. doi: 10.1002/pbc.24994. [Epub ahead of print]
- Nijmegen breakage syndrome (NBS) is an autosomal recessive chromosomal instability disorder characterized by a high incidence of pediatric hematologic malignancies. Majority of patients affected are of Slavic origin and share the same founder mutation of 657del5 within the NBN gene encoding protein
- PMID 24619942
- Staphylococcus delphini and Methicillin-Resistant S. pseudintermedius in Horses, Canada.
- Stull JW, Slavić D, Rousseau J, Weese JS.KEYWORDS: Canada, MecA protein, S. pseudintermedius, Staphylococcus delphini, antimicrobial resistance, bacteria, equid, equine, horses, infection control, mass spectrometry, matrix-assisted laser desorption-ionization, methicillin-resistant S. pseudintermedius, staphylococci, surveillance
- Emerging infectious diseases.Emerg Infect Dis.2014 Mar;20(3):485-7. doi: 10.3201/eid2003.130139.
- PMID 24565044
- Angiotensin type 2 receptor stimulation ameliorates left ventricular fibrosis and dysfunction via regulation of tissue inhibitor of matrix metalloproteinase 1/matrix metalloproteinase 9 axis and transforming growth factor β1 in the rat heart.
- Lauer D1, Slavic S, Sommerfeld M, Thöne-Reineke C, Sharkovska Y, Hallberg A, Dahlöf B, Kintscher U, Unger T, Steckelings UM, Kaschina E.Author information 1Center for Cardiovascular Research and Institute of Pharmacology, Charité - Universitätsmedizin, Berlin, Hessische Strasse 3-4, D-10115 Berlin, Germany. elena.kaschina@charite.de.AbstractLeft ventricular (LV) remodeling is the main reason for the development of progressive cardiac dysfunction after myocardial infarction (MI). This study investigated whether stimulation of the angiotensin type 2 receptor is able to ameliorate post-MI cardiac remodeling and what the underlying mechanisms may be. MI was induced in Wistar rats by permanent ligation of the left coronary artery. Treatment with the angiotensin type 2 receptor agonist compound 21 (0.03 mg/kg) was started 6 hours post-MI and continued for 6 weeks. Hemodynamic parameters were measured by echocardiography and intracardiac catheter. Effects on proteolysis were studied in heart tissue and primary cardiac fibroblasts. Compound 21 significantly improved systolic and diastolic functions, resulting in improved ejection fraction (71.2±4.7% versus 53.4±7.0%; P<0.001), fractional shortening (P<0.05), LV internal dimension in systole (P<0.05), LV end-diastolic pressure (16.9±1.2 versus 22.1±1.4 mm Hg; P<0.05), ratio of early (E) to late (A) ventricular filling velocities, and maximum and minimum rate of LV pressure rise (P<0.05). Compound 21 improved arterial stiffness parameters and reduced collagen content in peri-infarct myocardium. Tissue inhibitor of matrix metalloproteinase 1 was strongly upregulated, whereas matrix metalloproteinases 2 and 9 and transforming growth factor β1 were diminished in LV of treated animals. In cardiac fibroblasts, compound 21 initially induced tissue inhibitor of matrix metalloproteinase 1 expression followed by attenuated matrix metalloproteinase 9 and transforming growth factor β1 secretion. In conclusion, angiotensin type 2 receptor stimulation improves cardiac function and prevents cardiac remodeling in the late stage after MI, suggesting that angiotensin type 2 receptor agonists may be considered a future pharmacological approach for the improvement of post-MI cardiac dysfunction.
- Hypertension.Hypertension.2014 Mar;63(3):e60-7. doi: 10.1161/HYPERTENSIONAHA.113.02522. Epub 2013 Dec 30.
- Left ventricular (LV) remodeling is the main reason for the development of progressive cardiac dysfunction after myocardial infarction (MI). This study investigated whether stimulation of the angiotensin type 2 receptor is able to ameliorate post-MI cardiac remodeling and what the underlying mechani
- PMID 24379181
Japanese Journal
- Хама, Юкико, Юрасиа сюги това наника (Что такое евразийство?), Токио, Сэйбунся, 2010, 300 с.
- НАБОКОВ Владимир, Тамамоно. (Дар), Сэкай бунгаку дзэнсю II-10. (Библиотека мировой литературы II-10), Пер. с рус.: Мицуёси Нумано, Токио: Кавадэ-сёбо-синся, 2010, 4, 612+10с.
Related Links
- Japan's national center for Slavic and Eurasian studies ... Kindly inform webmaster of it when you find the URL(s) are not available anymore. HPの閉鎖やアドレスの変更などでリンク先が表示されない場合があります。
- Debra G. Van Mele Director of Finance & Administration John's Island Property Owners Association "Our 401(k) plan with Slavic401k was a significant improvement over our former plan from both a cost and service standpoint. The ...
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