WordNet

a substance that retards or stops an activity

PrepTutorEJDIC

抑制する人(物) / 化学反応抑制剤

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1. ACE 阻害薬誘発性血管浮腫 ace inhibitor induced angioedema
2. 高血圧治療におけるレニン・アンギオテンシン系阻害 renin angiotensin system inhibition in the treatment of hypertension

English Journal

Modulation of RAAS-natriuretic peptides in the treatment of HF: Old guys and newcomers.

Mollace V1, Gliozzi M2, Capuano A3, Rossi F3.
International journal of cardiology.Int J Cardiol.2016 Mar 24. pii: S0167-5273(16)30507-1. doi: 10.1016/j.ijcard.2016.03.085. [Epub ahead of print]
The use of renin-angiotensin-aldosterone system (RAAS) inhibitors in the treatment of chronic heart failure (HF) and arterial hypertension is recommended by the European Society of Cardiology Guidelines on the basis of consolidated evidence supporting their efficacy in the development of such a dise
PMID 27075034

The natriuretic peptides system in the pathophysiology of heart failure: from molecular basis to treatment.

Volpe M1, Carnovali M2, Mastromarino V3.
Clinical science (London, England : 1979).Clin Sci (Lond).2016 Jan;130(2):57-77. doi: 10.1042/CS20150469.
After its discovery in the early 1980s, the natriuretic peptide (NP) system has been extensively characterized and its potential influence in the development and progression of heart failure (HF) has been investigated. HF is a syndrome characterized by the activation of different neurohormonal syste
PMID 26637405

Japanese Journal

Vasopeptidase inhibitors : a new therapeutic concept in cardiovascular disease?

Circulation 104, 1856-1862, 2001
NAID 30029738371

Vasopeptidase inhibitors

Lancet 358, 1525-1532, 2001
NAID 30005700323

Related Links

Vasopeptidase inhibitors. - National Center for Biotechnology ...

1. J Renin Angiotensin Aldosterone Syst. 2002 Jun;3(2):90-5. Vasopeptidase inhibitors. Sagnella GA(1). Author information: (1)Blood Pressure Unit, St Georges Hospital Medical School, London, UK. g.sagnella@sghms.ac ...

Vasopeptidase Inhibitors - Circulation

Vascular Effects of ACE Inhibitors ACE is located mainly on endothelial cells, where it transforms Ang I into Ang II and degrades bradykinin, a potent stimulator of the l-arginine and cyclooxygenase pathways 1 ().